High levels of circulating beta-amyloid peptide do not cause cerebral beta-amyloidosis in transgenic mice.
about
The multifaceted nature of amyloid precursor protein and its proteolytic fragments: friends and foesAPP transgenic mice for modelling behavioural and psychological symptoms of dementia (BPSD).Alzheimer disease models and human neuropathology: similarities and differencesAdvances in our Understanding of the Pathophysiology, Detection and Management of Cerebral Amyloid Angiopathy.Prominent cerebral amyloid angiopathy in transgenic mice overexpressing the london mutant of human APP in neurons.Amyloid-beta deposition in skeletal muscle of transgenic mice: possible model of inclusion body myopathy.Transgenic mice over-expressing the C-99 fragment of betaPP with an alpha-secretase site mutation develop a myopathy similar to human inclusion body myositis.Elevated abeta42 in skeletal muscle of Alzheimer disease patients suggests peripheral alterations of AbetaPP metabolism.Neurologic and motor dysfunctions in APP transgenic mice.Neuronal overexpression of mutant amyloid precursor protein results in prominent deposition of cerebrovascular amyloid.Sporadic Alzheimer's disease begins as episodes of brain ischemia and ischemically dysregulated Alzheimer's disease genes.Genetic animal models of cerebral vasculopathies.Accumulation of amyloid-beta protein in exocrine glands of transgenic mice overexpressing a carboxyl terminal portion of amyloid protein precursor.
P2860
Q27026919-5C7AB33F-25E1-4955-8A44-CA87CCA2A6ADQ30455082-0BF22668-7FCD-4977-AD79-CB4C774D96D9Q30499574-F31CBE9F-F159-42F9-A823-CC100D4A2C30Q34994148-F38AF015-815D-4087-AA9F-C1C50E1C6844Q35745836-69F6106F-8F7D-4B9A-90C3-3C4B5062410EQ35786025-A9CA8ECA-F99A-4CF8-8648-1BAB9DE27308Q35786031-BDC1278C-109B-48FC-9CEA-10C71AB63B57Q35809798-3C0578A4-A007-42C7-BB8C-C0F8A6DF758AQ36347389-30B61F6F-34A8-45B1-8B75-05DDB03EC1BEQ36707976-F4545CBA-DD9D-41D2-9A6D-6B2626E4BA76Q37301029-3ED14D83-BC76-458C-BF66-8E5E55706D8BQ37542635-997F4CB2-C67B-4131-9BC9-2D8AF517C98AQ53342773-63E58A65-1F78-4434-9317-B669B8124229
P2860
High levels of circulating beta-amyloid peptide do not cause cerebral beta-amyloidosis in transgenic mice.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
High levels of circulating bet ...... myloidosis in transgenic mice.
@ast
High levels of circulating bet ...... myloidosis in transgenic mice.
@en
type
label
High levels of circulating bet ...... myloidosis in transgenic mice.
@ast
High levels of circulating bet ...... myloidosis in transgenic mice.
@en
prefLabel
High levels of circulating bet ...... myloidosis in transgenic mice.
@ast
High levels of circulating bet ...... myloidosis in transgenic mice.
@en
P2093
P2860
P1476
High levels of circulating bet ...... myloidosis in transgenic mice.
@en
P2093
C E Furlong
C E Ogburn
D D Kunkel
G M Martin
H M Wisniewski
R C LeBoeuf
P2860
P304
P407
P577
1996-07-01T00:00:00Z