Neuronal overexpression of mutant amyloid precursor protein results in prominent deposition of cerebrovascular amyloid.
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Induction of tau pathology by intracerebral infusion of amyloid-beta -containing brain extract and by amyloid-beta deposition in APP x Tau transgenic miceCerebrovascular disease in ageing and Alzheimer's diseaseEndogenous murine Aβ increases amyloid deposition in APP23 but not in APPPS1 transgenic mice.Dense-core senile plaques in the Flemish variant of Alzheimer's disease are vasocentricAbeta-induced inflammatory processes in microglia cells of APP23 transgenic miceRegional distribution of amyloid-Bri deposition and its association with neurofibrillary degeneration in familial British dementiaStructural and functional disruption of vascular smooth muscle cells in a transgenic mouse model of amyloid angiopathyAPP transgenic mice for modelling behavioural and psychological symptoms of dementia (BPSD).Alzheimer disease models and human neuropathology: similarities and differencesAnalysis of cerebral amyloid angiopathy in a transgenic mouse model of Alzheimer disease using in vivo multiphoton microscopy.Mitochondrial superoxide contributes to blood flow and axonal transport deficits in the Tg2576 mouse model of Alzheimer's disease.Age-related vascular pathology in transgenic mice expressing presenilin 1-associated familial Alzheimer's disease mutations.Vascular pathology in the aged human brainGenetics and molecular pathogenesis of sporadic and hereditary cerebral amyloid angiopathiesModeling familial Danish dementia in mice supports the concept of the amyloid hypothesis of Alzheimer's disease.Chronic delivery of antibody fragments using immunoisolated cell implants as a passive vaccination toolApolipoprotein E, smooth muscle cells and the pathogenesis of cerebral amyloid angiopathy: the potential role of impaired cerebrovascular A beta clearance.Mechanisms of cerebrovascular amyloid deposition. Lessons from mouse models.Clearance of Alzheimer's amyloid-ss(1-40) peptide from brain by LDL receptor-related protein-1 at the blood-brain barrierRole of vascular risk factors and vascular dysfunction in Alzheimer's disease.Animal models of amyloid-beta-related pathologies in Alzheimer's disease.Cerebral amyloid angiopathy in the elderlyVascular dementia: different forms of vessel disorders contribute to the development of dementia in the elderly brainAbeta is targeted to the vasculature in a mouse model of hereditary cerebral hemorrhage with amyloidosis.Genetic ablation of apolipoprotein A-IV accelerates Alzheimer's disease pathogenesis in a mouse modelEstrogen anti-inflammatory activity in brain: a therapeutic opportunity for menopause and neurodegenerative diseases.Observations in APP bitransgenic mice suggest that diffuse and compact plaques form via independent processes in Alzheimer's disease.Transgenic animal models of neurodegeneration based on human genetic studies.Amyloid deposition in the hippocampus and entorhinal cortex: quantitative analysis of a transgenic mouse model.Neuronal or glial expression of human apolipoprotein e4 affects parenchymal and vascular amyloid pathology differentially in different brain regions of double- and triple-transgenic miceDense-core plaques in Tg2576 and PSAPP mouse models of Alzheimer's disease are centered on vessel wallsCerebral microvascular amyloid beta protein deposition induces vascular degeneration and neuroinflammation in transgenic mice expressing human vasculotropic mutant amyloid beta precursor protein.Joint assessment of structural, perfusion, and diffusion MRI in Alzheimer's disease and frontotemporal dementia.beta-site amyloid precursor protein cleaving enzyme 1 increases amyloid deposition in brain parenchyma but reduces cerebrovascular amyloid angiopathy in aging BACE x APP[V717I] double-transgenic miceImmunotherapy of cerebrovascular amyloidosis in a transgenic mouse modelDoes my mouse have Alzheimer's disease?Association of neprilysin polymorphism with cerebral amyloid angiopathy.The contribution of activated astrocytes to Aβ production: implications for Alzheimer's disease pathogenesisSoluble Aβ seeds are potent inducers of cerebral β-amyloid depositionPeripherally applied Abeta-containing inoculates induce cerebral beta-amyloidosis.
P2860
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P2860
Neuronal overexpression of mutant amyloid precursor protein results in prominent deposition of cerebrovascular amyloid.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Neuronal overexpression of mut ...... on of cerebrovascular amyloid.
@ast
Neuronal overexpression of mut ...... on of cerebrovascular amyloid.
@en
type
label
Neuronal overexpression of mut ...... on of cerebrovascular amyloid.
@ast
Neuronal overexpression of mut ...... on of cerebrovascular amyloid.
@en
prefLabel
Neuronal overexpression of mut ...... on of cerebrovascular amyloid.
@ast
Neuronal overexpression of mut ...... on of cerebrovascular amyloid.
@en
P2093
P2860
P356
P1476
Neuronal overexpression of mut ...... on of cerebrovascular amyloid.
@en
P2093
A L Phinney
C Sturchler-Pierrat
D Abramowski
K H Wiederhold
M E Calhoun
M Staufenbiel
P2860
P304
14088-14093
P356
10.1073/PNAS.96.24.14088
P407
P577
1999-11-01T00:00:00Z