Changes in frequency, morphology, and behavior of tumors induced in mice by a polyoma virus mutant with a specifically altered oncogene.
about
Genetic analysis of a phosphatidylinositol 3-kinase SH2 domain reveals determinants of specificityA Transformation-Defective Polyomavirus Middle T Antigen with a Novel Defect in PI3 Kinase Signaling.Polyoma virus-induced osteosarcomas in inbred strains of mice: host determinants of metastasis.Radiation-resistant and radiation-sensitive forms of host resistance to polyomavirus.Phosphatidylinositol 3-kinase binding to polyoma virus middle tumor antigen mediates elevation of glucose transport by increasing translocation of the GLUT1 transporter.Serine 257 phosphorylation regulates association of polyomavirus middle T antigen with 14-3-3 proteinsEvidence of a role for phosphatidylinositol 3-kinase activation in the blocking of apoptosis by polyomavirus middle T antigen.Wild-derived inbred mice have a novel basis of susceptibility to polyomavirus-induced tumorsRequirement for both Shc and phosphatidylinositol 3' kinase signaling pathways in polyomavirus middle T-mediated mammary tumorigenesis.The phosphatidylinositol 3-kinase (PI3K) isoform dependence of tumor formation is determined by the genetic mode of PI3K pathway activation rather than by tissue type.Transgenic expression of polyomavirus middle T antigen in the mouse prostate gives rise to carcinoma.Cellular transformation by Simian Virus 40 and Murine Polyoma Virus T antigens.Lessons in signaling and tumorigenesis from polyomavirus middle T antigen.Oncogenesis of mammary glands, skin, and bones by polyomavirus correlates with viral persistence and prolonged genome replication potentialTransformation and tumorigenic properties of a mutant polyomavirus containing a middle T antigen defective in Shc binding.Tumor induction by a transformation-defective polyoma virus mutant blocked in signaling through ShcSusceptibility to tumors induced by polyoma virus is conferred by an endogenous mouse mammary tumor virus superantigen.Studies of partially transforming polyomavirus mutants establish a role for phosphatidylinositol 3-kinase in activation of pp70 S6 kinaseLessons from polyoma middle T antigen on signaling and transformation: A DNA tumor virus contribution to the war on cancer.Signaling from polyomavirus middle T and small T defines different roles for protein phosphatase 2A.Genetic and structural analysis of a virulence determinant in polyomavirus VP1.Polyomavirus middle T antigen induces the transcription of osteopontin, a gene important for the migration of transformed cells.The p110alpha isoform of phosphatidylinositol 3-kinase is essential for polyomavirus middle T antigen-mediated transformation.
P2860
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P2860
Changes in frequency, morphology, and behavior of tumors induced in mice by a polyoma virus mutant with a specifically altered oncogene.
description
1992 nî lūn-bûn
@nan
1992年の論文
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1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
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1992年论文
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name
Changes in frequency, morpholo ...... specifically altered oncogene.
@ast
Changes in frequency, morpholo ...... specifically altered oncogene.
@en
type
label
Changes in frequency, morpholo ...... specifically altered oncogene.
@ast
Changes in frequency, morpholo ...... specifically altered oncogene.
@en
prefLabel
Changes in frequency, morpholo ...... specifically altered oncogene.
@ast
Changes in frequency, morpholo ...... specifically altered oncogene.
@en
P2093
P2860
P1476
Changes in frequency, morpholo ...... specifically altered oncogene.
@en
P2093
J P Carroll
T L Benjamin
P2860
P304
P407
P577
1992-12-01T00:00:00Z