Tumor induction by a transformation-defective polyoma virus mutant blocked in signaling through Shc
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A Transformation-Defective Polyomavirus Middle T Antigen with a Novel Defect in PI3 Kinase Signaling.Receptor-binding and oncogenic properties of polyoma viruses isolated from feral micePolyoma virus-induced osteosarcomas in inbred strains of mice: host determinants of metastasis.Serine 257 phosphorylation regulates association of polyomavirus middle T antigen with 14-3-3 proteinsEvidence of a role for phosphatidylinositol 3-kinase activation in the blocking of apoptosis by polyomavirus middle T antigen.Wild-derived inbred mice have a novel basis of susceptibility to polyomavirus-induced tumorsNatural biology of polyomavirus middle T antigen.A tumor host range selection procedure identifies p150(sal2) as a target of polyoma virus large T antigen.Requirement for both Shc and phosphatidylinositol 3' kinase signaling pathways in polyomavirus middle T-mediated mammary tumorigenesis.Shp2 suppresses PyMT-induced transformation in mouse fibroblasts by inhibiting Stat3 activity.Cellular transformation by Simian Virus 40 and Murine Polyoma Virus T antigens.Lessons in signaling and tumorigenesis from polyomavirus middle T antigen.Lessons from polyoma middle T antigen on signaling and transformation: A DNA tumor virus contribution to the war on cancer.Signaling from polyomavirus middle T and small T defines different roles for protein phosphatase 2A.Polyomavirus middle T antigen induces the transcription of osteopontin, a gene important for the migration of transformed cells.The p110alpha isoform of phosphatidylinositol 3-kinase is essential for polyomavirus middle T antigen-mediated transformation.Polyomavirus small T antigen controls viral chromatin modifications through effects on kinetics of virus growth and cell cycle progression.Polyomavirus-induced pilomatricomas in mice: from viral inoculation to tumour development.Retinoic acid inhibits transformation by preventing phosphatidylinositol 3-kinase dependent activation of the c-fos promoter.Sustained ex vivo and in vivo transfer of a reporter gene using polyoma virus pseudocapsids.Formation of transformed endothelial cells in the absence of VEGFR-2/Flk-1 by Polyoma middle T oncogene.
P2860
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P2860
Tumor induction by a transformation-defective polyoma virus mutant blocked in signaling through Shc
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1997 nî lūn-bûn
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1997年の論文
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1997年学术文章
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1997年学术文章
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name
Tumor induction by a transform ...... ocked in signaling through Shc
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Tumor induction by a transform ...... ocked in signaling through Shc
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type
label
Tumor induction by a transform ...... ocked in signaling through Shc
@ast
Tumor induction by a transform ...... ocked in signaling through Shc
@en
prefLabel
Tumor induction by a transform ...... ocked in signaling through Shc
@ast
Tumor induction by a transform ...... ocked in signaling through Shc
@en
P2093
P2860
P356
P1476
Tumor induction by a transform ...... ocked in signaling through Shc
@en
P2093
P2860
P304
P356
10.1073/PNAS.94.15.7954
P407
P577
1997-07-01T00:00:00Z