Innocuous IFNgamma induced by adjuvant-free antigen restores normoglycemia in NOD mice through inhibition of IL-17 production - Wikidata - wikimedia - TriplyDB

Innocuous IFNgamma induced by adjuvant-free antigen restores normoglycemia in NOD mice through inhibition of IL-17 production

Innocuous IFNgamma induced by adjuvant-free antigen restores normoglycemia in NOD mice through inhibition of IL-17 production

http://www.wikidata.org/entity/Q36446935

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Berberine differentially modulates the activities of ERK, p38 MAPK, and JNK to suppress Th17 and Th1 T cell differentiation in type 1 diabetic mice Th17 Cells in Type 1 Diabetes: Role in the Pathogenesis and Regulation by Gut Microbiome Antigen-specific tolerance in immunotherapy of Th2-associated allergic diseases.Naturally occurring immunoglobulin M (nIgM) autoantibodies prevent autoimmune diabetes and mitigate inflammation after transplantation Current status of immunomodulatory and cellular therapies in preclinical and clinical islet transplantation.Genetic-induced variations in the GAD65 T-cell repertoire governs efficacy of anti-CD3/GAD65 combination therapy in new-onset type 1 diabetes.MSC-derived Extracellular Vesicles Attenuate Immune Responses in Two Autoimmune Murine Models: Type 1 Diabetes and Uveoretinitis.Suppression of ongoing T cell-mediated autoimmunity by peptide-MHC class II dimer vaccination.Comparison of sensitivity of Th1, Th2, and Th17 cells to Fas-mediated apoptosis.Th 17 cells interplay with Foxp3+ Tregs in regulation of inflammation and autoimmunity.Subcutaneous insulin B:9-23/IFA immunisation induces Tregs that control late-stage prediabetes in NOD mice through IL-10 and IFNgamma.Inflammatory tendencies and overproduction of IL-17 in the colon of young NOD mice are counteracted with diet change.Suppression of autoimmune diabetes by soluble galectin-1 Impact of dietary gluten on regulatory T cells and Th17 cells in BALB/c mice.Regulatory T cell epitopes (Tregitopes) in IgG induce tolerance in vivo and lack immunogenicity per se.Virtual optimization of nasal insulin therapy predicts immunization frequency to be crucial for diabetes protection.The dual role of scavenger receptor class A in development of diabetes in autoimmune NOD mice Interleukin-1β produced in response to islet autoantigen presentation differentiates T-helper 17 cells at the expense of regulatory T-cells: Implications for the timing of tolerizing immunotherapy.Aberrant ROCK activation promotes the development of type I diabetes in NOD mice Therapy of experimental type 1 diabetes by isolated Sertoli cell xenografts alone.IL-21 signaling is critical for the development of type I diabetes in the NOD mouse.Naturally transmitted segmented filamentous bacteria segregate with diabetes protection in nonobese diabetic mice.Tissue-specific expression of B7x protects from CD4 T cell-mediated autoimmunity.Potential role of IL-17-producing iNKT cells in type 1 diabetes.Development of autoimmune diabetes in the absence of detectable IL-17A in a CD8-driven virally induced model.CD4 T cells play important roles in maintaining IL-17-producing γδ T-cell subsets in naive animals Genome-wide transcriptional analyses of islet-specific CD4+ T cells identify Idd9 genes controlling diabetogenic T cell function Expansion of Th17 cells and functional defects in T regulatory cells are key features of the pancreatic lymph nodes in patients with type 1 diabetes.Differential IL-21 signaling in APCs leads to disparate Th17 differentiation in diabetes-susceptible NOD and diabetes-resistant NOD.Idd3 mice.No evidence for activation of T(H)1 or T(H)17 pathways in unstimulated peripheral blood mononuclear cells from children with β-cell autoimmunity or T1D.Helminth protection against autoimmune diabetes in nonobese diabetic mice is independent of a type 2 immune shift and requires TGF-β.Anti-FcεR1 antibody injections activate basophils and mast cells and delay Type 1 diabetes onset in NOD mice.Antigen-specific therapeutic approaches in Type 1 diabetes Mechanisms underlying antigen-specific tolerance of stable and convertible Th17 cells during suppression of autoimmune diabetes.Immunogenicity of Isogenic IgG in Aggregates and Immune Complexes.Transforming growth factor-beta suppresses the activation of CD8+ T-cells when naive but promotes their survival and function once antigen experienced: a two-faced impact on autoimmunity.Antigen-specific effector CD4 T lymphocytes school lamina propria dendritic cells to transfer innate tolerance.Innate pro-B-cell progenitors protect against type 1 diabetes by regulating autoimmune effector T cells Primer: immunity and autoimmunity HLA-DQ6 (DQB1*0601)-restricted T cells protect against experimental autoimmune encephalomyelitis in HLA-DR3.DQ6 double-transgenic mice by generating anti-inflammatory IFN-gamma

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Innocuous IFNgamma induced by adjuvant-free antigen restores normoglycemia in NOD mice through inhibition of IL-17 production

http://www.wikidata.org/entity/Q36446935

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2008 nî lūn-bûn

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2008年学术文章

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2008年學術文章

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Christine M Hoeman

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Craig L Franklin

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Danielle M Tartar

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Habib Zaghouani

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Hyun-Hee Lee

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J Jeremiah Bell

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Jason S Ellis

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Ping Yu

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Randal K Gregg

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Renu Jain

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P2860

Structural basis of peptide binding and presentation by the type I diabetes-associated MHC class II molecule of NOD mice

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Transforming growth factor-beta induces development of the T(H)17 lineage

Interleukin-17 family members and inflammation

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

Adult pancreatic beta-cells are formed by self-duplication rather than stem-cell differentiation

A CD4+ T-cell subset inhibits antigen-specific T-cell responses and prevents colitis

Identification and modulation of a naturally processed T cell epitope from the diabetes-associated autoantigen human glutamic acid decarboxylase 65 (hGAD65).

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207-218

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10.1084/JEM.20071878

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1

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205

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5656615

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18195074

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2234380

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