Differential response of human cells to deletions and stop codons in the gamma(1)34.5 gene of herpes simplex virus.
about
Complete sequence and comparative analysis of the genome of herpes B virus (Cercopithecine herpesvirus 1) from a rhesus monkeyEvasion of cellular antiviral responses by human cytomegalovirus TRS1 and IRS1A herpesvirus ribosome-associated, RNA-binding protein confers a growth advantage upon mutants deficient in a GADD34-related function.B-myb promoter retargeting of herpes simplex virus gamma34.5 gene-mediated virulence toward tumor and cycling cellsAssociation of a M(r) 90,000 phosphoprotein with protein kinase PKR in cells exhibiting enhanced phosphorylation of translation initiation factor eIF-2 alpha and premature shutoff of protein synthesis after infection with gamma 134.5- mutants of herActivated MEK suppresses activation of PKR and enables efficient replication and in vivo oncolysis by Deltagamma(1)34.5 mutants of herpes simplex virus 1Comparison of genetically engineered herpes simplex viruses for the treatment of brain tumors in a scid mouse model of human malignant glioma.Functional genomic analysis of herpes simplex virus type 1 counteraction of the host innate response.Separation of receptor-binding and profusogenic domains of glycoprotein D of herpes simplex virus 1 into distinct interacting proteins.The carboxyl terminus of the murine MyD116 gene substitutes for the corresponding domain of the gamma(1)34.5 gene of herpes simplex virus to preclude the premature shutoff of total protein synthesis in infected human cells.Phenotypic properties of herpes simplex virus 1 containing a derepressed open reading frame P gene.The range and distribution of murine central nervous system cells infected with the gamma(1)34.5- mutant of herpes simplex virus 1.Suppression of the phenotype of gamma(1)34.5- herpes simplex virus 1: failure of activated RNA-dependent protein kinase to shut off protein synthesis is associated with a deletion in the domain of the alpha47 gene.Transcription of the derepressed open reading frame P of herpes simplex virus 1 precludes the expression of the antisense gamma(1)34.5 gene and may account for the attenuation of the mutant virusHerpes simplex virus 1 gene products occlude the interferon signaling pathway at multiple sites.The application of genetically engineered herpes simplex viruses to the treatment of experimental brain tumorsHerpes simplex virus type 1 U(L)34 gene product is required for viral envelopment.Herpes simplex virus ICP0 and ICP34.5 counteract distinct interferon-induced barriers to virus replication.Suppression of apoptotic DNA fragmentation in herpes simplex virus type 1-infected cells.
P2860
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P2860
Differential response of human cells to deletions and stop codons in the gamma(1)34.5 gene of herpes simplex virus.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Differential response of human ...... gene of herpes simplex virus.
@ast
Differential response of human ...... gene of herpes simplex virus.
@en
type
label
Differential response of human ...... gene of herpes simplex virus.
@ast
Differential response of human ...... gene of herpes simplex virus.
@en
prefLabel
Differential response of human ...... gene of herpes simplex virus.
@ast
Differential response of human ...... gene of herpes simplex virus.
@en
P2093
P2860
P1433
P1476
Differential response of human ...... 5 gene of herpes simplex virus
@en
P2093
P2860
P304
P407
P577
1994-12-01T00:00:00Z