Phenotypic properties of herpes simplex virus 1 containing a derepressed open reading frame P gene.
about
Herpes simplex virus 1 open reading frames O and P are not necessary for establishment of latent infection in mice.The second-site mutation in the herpes simplex virus recombinants lacking the gamma134.5 genes precludes shutoff of protein synthesis by blocking the phosphorylation of eIF-2alphaThe polyserine tract of herpes simplex virus ICP4 is required for normal viral gene expression and growth in murine trigeminal gangliaHerpes simplex virus 1-infected cell protein 0 contains two E3 ubiquitin ligase sites specific for different E2 ubiquitin-conjugating enzymesCharacterization of the novel E3 ubiquitin ligase encoded in exon 3 of herpes simplex virus-1-infected cell protein 0.Disruption of HDAC/CoREST/REST repressor by dnREST reduces genome silencing and increases virulence of herpes simplex virusThe checkpoints of viral gene expression in productive and latent infection: the role of the HDAC/CoREST/LSD1/REST repressor complexExperimental investigation of herpes simplex virus latencyCharacterization of herpes simplex virus 2 primary microRNA Transcript regulationHSV-1 gene expression from reactivated ganglia is disordered and concurrent with suppression of latency-associated transcript and miRNAsThe herpes simplex virus type 1 2.0-kilobase latency-associated transcript is a stable intron which branches at a guanosineTranscription of the derepressed open reading frame P of herpes simplex virus 1 precludes the expression of the antisense gamma(1)34.5 gene and may account for the attenuation of the mutant virusPreclinical evaluation of a genetically engineered herpes simplex virus expressing interleukin-12T cell receptor-gamma/delta cells protect mice from herpes simplex virus type 1-induced lethal encephalitis.Preclinical evaluation of oncolytic δγ(1)34.5 herpes simplex virus expressing interleukin-12 for therapy of breast cancer brain metastasesThe product of ORF O located within the domain of herpes simplex virus 1 genome transcribed during latent infection binds to and inhibits in vitro binding of infected cell protein 4 to its cognate DNA site.The infected cell protein 0 of herpes simplex virus 1 dynamically interacts with proteasomes, binds and activates the cdc34 E2 ubiquitin-conjugating enzyme, and possesses in vitro E3 ubiquitin ligase activity.The function of herpes simplex virus genes: a primer for genetic engineering of novel vectorsNovel less-abundant viral microRNAs encoded by herpes simplex virus 2 latency-associated transcript and their roles in regulating ICP34.5 and ICP0 mRNAs.Open reading frame P--a herpes simplex virus gene repressed during productive infection encodes a protein that binds a splicing factor and reduces synthesis of viral proteins made from spliced mRNASpread and replication of and immune response to gamma134.5-negative herpes simplex virus type 1 vectors in BALB/c mice.The genome sequence of herpes simplex virus type 2Differentiated Human SH-SY5Y Cells Provide a Reductionist Model of Herpes Simplex Virus 1 Neurotropism.
P2860
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P2860
Phenotypic properties of herpes simplex virus 1 containing a derepressed open reading frame P gene.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Phenotypic properties of herpe ...... sed open reading frame P gene.
@ast
Phenotypic properties of herpe ...... sed open reading frame P gene.
@en
type
label
Phenotypic properties of herpe ...... sed open reading frame P gene.
@ast
Phenotypic properties of herpe ...... sed open reading frame P gene.
@en
prefLabel
Phenotypic properties of herpe ...... sed open reading frame P gene.
@ast
Phenotypic properties of herpe ...... sed open reading frame P gene.
@en
P2860
P1433
P1476
Phenotypic properties of herpe ...... ssed open reading frame P gene
@en
P2093
P2860
P304
P407
P577
1996-03-01T00:00:00Z