The oncogenic forms of N-ras or H-ras prevent skeletal myoblast differentiation.
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Human carcinoembryonic antigen, an intercellular adhesion molecule, blocks fusion and differentiation of rat myoblastsAlteration of homeobox gene expression by N-ras transformation of PA-1 human teratocarcinoma cellsHistone deacetylase 4 associates with extracellular signal-regulated kinases 1 and 2, and its cellular localization is regulated by oncogenic RasEpigenetic deregulation of microRNAs in rhabdomyosarcoma and neuroblastoma and translational perspectivesPositive regulation of skeletal myogenesis by R-RasThe pro-apoptotic Ras effector Nore1 may serve as a Ras-regulated tumor suppressor in the lungDifferential expression of myogenic determination genes in muscle cells: possible autoactivation by the Myf gene productsRetinoic acid induces myogenin synthesis and myogenic differentiation in the rat rhabdomyosarcoma cell line BA-Han-1CAmphoterin stimulates myogenesis and counteracts the antimyogenic factors basic fibroblast growth factor and S100B via RAGE bindingMicroRNA-214 promotes myogenic differentiation by facilitating exit from mitosis via down-regulation of proto-oncogene N-rasParticipation of multiple factors, including proliferin, in the inhibition of myogenic differentiationCDO, a robo-related cell surface protein that mediates myogenic differentiationPerspective: embedded molecular switches, anticancer selection, and effects on ontogenetic rates: a hypothesis of developmental constraint on morphogenesis and evolution.Transcriptional profiling of mesenchymal stromal cells from young and old rats in response to Dexamethasone.Neoplastic transformation inactivates specific trans-acting factor(s) required for the expression of the thyroglobulin gene.TC21 and Ras share indistinguishable transforming and differentiating activities.NF-kappaB controls cell growth and differentiation through transcriptional regulation of cyclin D1.The retinoblastoma protein is linked to the activation of Ras.Peptide growth factors can provoke "fetal" contractile protein gene expression in rat cardiac myocytes.Dihydropyridine receptor gene expression is regulated by inhibitors of myogenesis and is relatively insensitive to denervation.Activation of Ras and the mitogen-activated protein kinase pathway promotes protein degradation in muscle cells of Caenorhabditis elegans.Atypical protein kinase Cs are the Ras effectors that mediate repression of myogenic satellite cell differentiation.An activated c-Ha-ras allele blocks the induction of muscle-specific genes whose expression is contingent on mitogen withdrawal.Control of differentiation in BC3H1 muscle cellsA Drosophila model of the rhabdomyosarcoma initiator PAX7-FKHR.Rb and N-ras function together to control differentiation in the mouse.Metaplastic change in mesenchymal stem cells induced by activated ras oncogeneProbing for a deeper understanding of rhabdomyosarcoma: insights from complementary model systems.Dexamethasone-dependent inhibition of differentiation of C2 myoblasts bearing steroid-inducible N-ras oncogenes.Growth factors, signaling pathways, and the regulation of proliferation and differentiation in BC3H1 muscle cells. II. Two signaling pathways distinguished by pertussis toxin and a potential role for the ras oncogene.Growth factors, signaling pathways, and the regulation of proliferation and differentiation in BC3H1 muscle cells. I. A pertussis toxin-sensitive pathway is involved.Aberrant regulation of MyoD1 contributes to the partially defective myogenic phenotype of BC3H1 cells.Carcinoembryonic antigen, a human tumor marker, cooperates with Myc and Bcl-2 in cellular transformationOncogenic Ras-induced proliferation requires autocrine fibroblast growth factor 2 signaling in skeletal muscle cells.Serum response factor p67SRF is expressed and required during myogenic differentiation of both mouse C2 and rat L6 muscle cell lines.Inhibition of myogenesis by the H-ras oncogene: implication of a role for protein kinase C.Ras p21Val inhibits myogenesis without altering the DNA binding or transcriptional activities of the myogenic basic helix-loop-helix factors.Cyclin-mediated inhibition of muscle gene expression via a mechanism that is independent of pRB hyperphosphorylation.Signaling through mitogen-activated protein kinase and Rac/Rho does not duplicate the effects of activated Ras on skeletal myogenesisfos/jun repression of cardiac-specific transcription in quiescent and growth-stimulated myocytes is targeted at a tissue-specific cis element.
P2860
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P2860
The oncogenic forms of N-ras or H-ras prevent skeletal myoblast differentiation.
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
The oncogenic forms of N-ras or H-ras prevent skeletal myoblast differentiation.
@en
type
label
The oncogenic forms of N-ras or H-ras prevent skeletal myoblast differentiation.
@en
prefLabel
The oncogenic forms of N-ras or H-ras prevent skeletal myoblast differentiation.
@en
P2093
P2860
P356
P1476
The oncogenic forms of N-ras or H-ras prevent skeletal myoblast differentiation.
@en
P2093
P2860
P304
P356
10.1128/MCB.7.6.2104
P407
P577
1987-06-01T00:00:00Z