Selective transformation of primitive lymphoid cells by the BCR/ABL oncogene expressed in long-term lymphoid or myeloid cultures.
about
CRKL binding to BCR-ABL and BCR-ABL transformationThe expression of the non-receptor tyrosine kinases Arg and c-abl is differently modulated in B lymphoid cells at different stages of differentiationInteractions of CBL with BCR-ABL and CRKL in BCR-ABL-transformed myeloid cells.Novel mechanism of tumor suppression by polarity gene discs large 1 (DLG1) revealed in a murine model of pediatric B-ALL.Induction of a chronic myelogenous leukemia-like syndrome in mice with v-abl and BCR/ABL.bcr-abl, the hallmark of chronic myeloid leukaemia in man, induces multiple haemopoietic neoplasms in mice.Tyrosyl phosphorylation and DNA binding activity of signal transducers and activators of transcription (STAT) proteins in hematopoietic cell lines transformed by Bcr/Abl.Hyperexpression of interleukin-7 is not necessary or sufficient for transformation of a pre-B lymphoid cell line.Early pre-B-cell transformation induced by the v-fms oncogene in long-term mouse bone marrow cultures.A bcr-v-abl oncogene induces lymphomas in transgenic mice.Alternative forms of the BCR-ABL oncogene have quantitatively different potencies for stimulation of immature lymphoid cells.T-cell immunity to the joining region of p210BCR-ABL proteinMolecular analysis of the Philadelphia chromosome.Activation of tyrosinase kinase and microfilament-binding functions of c-abl by bcr sequences in bcr/abl fusion proteinsA 41-kilodalton protein is a potential substrate for the p210bcr-abl protein-tyrosine kinase in chronic myelogenous leukemia cells.Hematologic disease induced in BALB/c mice by a bcr-abl retrovirus is influenced by the infection conditions.Blast crisis in a murine model of chronic myelogenous leukemia.The Use of Genetic Markers in Studies of Myeloid Neoplasia.The human leukemia oncogene bcr-abl abrogates the anchorage requirement but not the growth factor requirement for proliferationSubcellular localization of Bcr, Abl, and Bcr-Abl proteins in normal and leukemic cells and correlation of expression with myeloid differentiation.Role of the C-terminal actin binding domain in BCR/ABL-mediated survival and drug resistance.Immunity to oncogenic proteins.T-cell immunity to oncogenic proteins including mutated ras and chimeric bcr-abl.CML: mechanisms of disease initiation and progression.A coiled-coil tetramerization domain of BCR-ABL is essential for the interactions of SH2-containing signal transduction molecules.Induction of erythroid differentiation by 5-fluorouracil in K562 leukemia cells.Coupling between p210bcr-abl and Shc and Grb2 adaptor proteins in hematopoietic cells permits growth factor receptor-independent link to ras activation pathway.A specific need for CRKL in p210BCR-ABL-induced transformation of mouse hematopoietic progenitors.Polarized distribution of Bcr-Abl in migrating myeloid cells and co-localization of Bcr-Abl and its target proteins.The role of alternative splicing patterns of BCR/ABL transcripts in the generation of the blast crisis of chronic myeloid leukaemia.Molecular Diagnosis of the Philadelphia Chromosome in Acute Lymphoblastic Leukemia.
P2860
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P2860
Selective transformation of primitive lymphoid cells by the BCR/ABL oncogene expressed in long-term lymphoid or myeloid cultures.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
1988年论文
@zh
1988年论文
@zh-cn
name
Selective transformation of pr ...... lymphoid or myeloid cultures.
@en
type
label
Selective transformation of pr ...... lymphoid or myeloid cultures.
@en
prefLabel
Selective transformation of pr ...... lymphoid or myeloid cultures.
@en
P2860
P356
P1476
Selective transformation of pr ...... lymphoid or myeloid cultures.
@en
P2093
P2860
P304
P356
10.1128/MCB.8.10.4079
P407
P577
1988-10-01T00:00:00Z