CML: mechanisms of disease initiation and progression.

about

Expression of BCR/ABL and BCL-2 in myeloid progenitors leads to myeloid leukemias.Presence of P210bcrabl is associated with decreased expression of a beta chemokine C10 gene in a P210bcrabl-positive myeloid leukemia cell line.The Retinoblastoma Gene in Myeloid Leukaemias.

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CML: mechanisms of disease initiation and progression.

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http://www.wikidata.org/entity/Q40785980

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1993 nî lūn-bûn

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1993年の論文

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1993年学术文章

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1993年学术文章

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1993年学术文章

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1993年学术文章

@zh-my

1993年学术文章

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1993年學術文章

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1993年學術文章

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1993年學術文章

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name

CML: mechanisms of disease initiation and progression.

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CML: mechanisms of disease initiation and progression.

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CML: mechanisms of disease initiation and progression.

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P1476

CML: mechanisms of disease initiation and progression.

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P2093

Estrov Z

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Kurzrock R

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Talpaz M

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Wetzler M

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Selective inhibition of leukemia cell proliferation by BCR-ABL antisense oligodeoxynucleotides

Transformation of an interleukin 3-dependent hematopoietic cell line by the chronic myelogenous leukemia-specific P210bcr/abl protein.

Induction of a chronic myelogenous leukemia-like syndrome in mice with v-abl and BCR/ABL.

Alterations in the p53 gene and the clonal evolution of the blast crisis of chronic myelocytic leukemia.

In vitro transformation of immature hematopoietic cells by the P210 BCR/ABL oncogene product of the Philadelphia chromosome.

A bcr-v-abl oncogene induces lymphomas in transgenic mice.

Selective transformation of primitive lymphoid cells by the BCR/ABL oncogene expressed in long-term lymphoid or myeloid cultures.

Activation of tyrosinase kinase and microfilament-binding functions of c-abl by bcr sequences in bcr/abl fusion proteins

Clonal lymphoid progenitor cell lines expressing the BCR/ABL oncogene retain full differentiative function.

The molecular genetics of Philadelphia chromosome-positive leukemias.

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47-50

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scholarly article

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10.3109/10428199309047863

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11 Suppl 1

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1993-01-01T00:00:00Z

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8251916

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CML: mechanisms of disease initiation and progression.

about

P2860

P2860

CML: mechanisms of disease initiation and progression.

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