Increased regional cerebral glucose uptake in an APP/PS1 model of Alzheimer's disease. - Wikidata - wikimedia - TriplyDB

Increased regional cerebral glucose uptake in an APP/PS1 model of Alzheimer's disease.

Increased regional cerebral glucose uptake in an APP/PS1 model of Alzheimer's disease.

http://www.wikidata.org/entity/Q36884880

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Chronic rapamycin restores brain vascular integrity and function through NO synthase activation and improves memory in symptomatic mice modeling Alzheimer's disease.Structural Magnetic Resonance Imaging Markers of Alzheimer's Disease and Its Retranslation to Rodent Models.Age- and Brain Region-Specific Changes of Glucose Metabolic Disorder, Learning, and Memory Dysfunction in Early Alzheimer's Disease Assessed in APP/PS1 Transgenic Mice Using 18F-FDG-PET.The Effects of Physiological and Methodological Determinants on 18F-FDG Mouse Brain Imaging Exemplified in a Double Transgenic Alzheimer Model.Brain and Brown Adipose Tissue Metabolism in Transgenic Tg2576 Mice Models of Alzheimer Disease Assessed Using 18F-FDG PET Imaging.Early detection of cerebral glucose uptake changes in the 5XFAD mouse.Decreased neuronal bursting and phase synchrony in the hippocampus of streptozotocin diabetic rats Overexpression of pyruvate dehydrogenase kinase 1 and lactate dehydrogenase A in nerve cells confers resistance to amyloid β and other toxins by decreasing mitochondrial respiration and reactive oxygen species production.Hypermetabolic state in the 7-month-old triple transgenic mouse model of Alzheimer's disease and the effect of lipoic acid: a 13C-NMR study.Construction and evaluation of quantitative small-animal PET probabilistic atlases for [¹⁸F]FDG and [¹⁸F]FECT functional mapping of the mouse brain Templated misfolding of Tau by prion-like seeding along neuronal connections impairs neuronal network function and associated behavioral outcomes in Tau transgenic mice.In vivo molecular neuroimaging of glucose utilization and its association with fibrillar amyloid-β load in aged APPPS1-21 mice Magnetic Resonance Q Mapping Reveals a Decrease in Microvessel Density in the arcAβ Mouse Model of Cerebral Amyloidosis.Aerobic Glycolysis in the Frontal Cortex Correlates with Memory Performance in Wild-Type Mice But Not the APP/PS1 Mouse Model of Cerebral Amyloidosis.Regional brain hypometabolism is unrelated to regional amyloid plaque burden.Effects of amyloid precursor protein 17 peptide on the protection of diabetic encephalopathy and improvement of glycol metabolism in the diabetic rat Assessment of neuroinflammation in a mouse model of obesity and β-amyloidosis using PET.Longitudinal Characterization of [18F]-FDG and [18F]-AV45 Uptake in the Double Transgenic TASTPM Mouse Model.Imaging of cerebrovascular pathology in animal models of Alzheimer's disease Mitochondrial function in ageing: coordination with signalling and transcriptional pathways.Impairments of neural circuit function in Alzheimer's disease.Amyloid β 1-42 induces hypometabolism in human stem cell-derived neuron and astrocyte networks.Astrocytes and synaptic plasticity in health and disease.A disease with a sweet tooth: exploring the Warburg effect in Alzheimer's disease.A dual tracer PET-MRI protocol for the quantitative measure of regional brain energy substrates uptake in the rat.Insulin Resistance and Alzheimer's Disease: Bioenergetic Linkages.Analysis of the vasculature by immunohistochemistry in paraffin-embedded brains.Intranasal Insulin Ameliorates Cerebral Hypometabolism, Neuronal Loss, and Astrogliosis in Streptozotocin-Induced Alzheimer's Rat Model.Mouse models of neurodegenerative disease: preclinical imaging and neurovascular component.Brain energy metabolism and neuroinflammation in ageing APP/PS1-21 mice using longitudinal 18F-FDG and 18F-DPA-714 PET imaging.Does posterior cingulate hypometabolism result from disconnection or local pathology across preclinical and clinical stages of Alzheimer's disease?Early Detection of Aβ Deposition in the 5xFAD Mouse by Amyloid PET.Cortical hypoperfusion and reduced cerebral metabolic rate of oxygen in the arcAβ mouse model of Alzheimer's disease.Chemical exchange saturation transfer MRI shows low cerebral 2-deoxy-D-glucose uptake in a model of Alzheimer's Disease.

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Increased regional cerebral glucose uptake in an APP/PS1 model of Alzheimer's disease.

http://www.wikidata.org/entity/Q36884880

description

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name

Increased regional cerebral glucose uptake in an APP/PS1 model of Alzheimer's disease.

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Increased regional cerebral glucose uptake in an APP/PS1 model of Alzheimer's disease.

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Increased regional cerebral glucose uptake in an APP/PS1 model of Alzheimer's disease.

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J.NEUROBIOLAGING.2011.09.026

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Neurobiology of Aging

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Increased regional cerebral glucose uptake in an APP/PS1 model of Alzheimer's disease

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Andreas Volk

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Emmanuel Bourrin

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Géraldine Poisnel

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Jésus Benavides

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Nadine El Tannir El Tayara

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Philippe Hantraye

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Thierry Delzescaux

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Thomas Debeir

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Thomas Rooney

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P2860

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Spatial correlation between brain aerobic glycolysis and amyloid-β (Aβ ) deposition

Soluble oligomers of the amyloid beta-protein impair synaptic plasticity and behavior

Picomolar amyloid-beta positively modulates synaptic plasticity and memory in hippocampus.

Intracellular amyloid-beta in Alzheimer's disease

Basal cerebral metabolism may modulate the cognitive effects of Abeta in mild cognitive impairment: an example of brain reserve.

Alzheimer disease models and human neuropathology: similarities and differences

Assessment of microPET performance in analyzing the rat brain under different types of anesthesia: comparison between quantitative data obtained with microPET and ex vivo autoradiography.

Perfusion MRI of U87 brain tumors in a mouse model.

Brain glucose metabolism in the early and specific diagnosis of Alzheimer's disease. FDG-PET studies in MCI and AD.

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