Organization of the Epstein-Barr virus DNA molecule. III. Location of the P3HR-1 deletion junction and characterization of the NotI repeat units that form part of the template for an abundant 12-O-tetradecanoylphorbol-13-acetate-induced mRNA transcr
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A gammaherpesviral internal repeat contributes to latency amplification.Complex transcription of the Epstein-Barr virus BamHI fragment H rightward open reading frame 1 (BHRF1) in latently and lytically infected B lymphocytesAn Epstein-Barr virus isolated from a lymphoblastoid cell line has a 16-kilobase-pair deletion which includes gp350 and the Epstein-Barr virus nuclear antigen 3ABHRF1 of Epstein-Barr virus, which is homologous to human proto-oncogene bcl2, is not essential for transformation of B cells or for virus replication in vitro.Spliced RNA from the IR1-U2 region of Epstein-Barr virus: presence of an open reading frame for a repetitive polypeptide.C-terminal region of EBNA-2 determines the superior transforming ability of type 1 Epstein-Barr virus by enhanced gene regulation of LMP-1 and CXCR7.A wide extent of inter-strain diversity in virulent and vaccine strains of alphaherpesviruses.Molecular virology of Epstein-Barr virus.The Epstein-Barr virus nuclear antigen 2 interacts with an EBNA2 responsive cis-element of the terminal protein 1 gene promoterHigh-throughput RNA sequencing-based virome analysis of 50 lymphoma cell lines from the Cancer Cell Line Encyclopedia project.Viral noncoding RNAs: more surprises.trans-Repression of protein expression dependent on the Epstein-Barr virus promoter Wp during latency.Nucleotide sequences of mRNAs encoding Epstein-Barr virus nuclear proteins: a probable transcriptional initiation siteAutoantigenic proteins that bind recombinogenic sequences in Epstein-Barr virus and cellular DNAReducing the complexity of the transforming Epstein-Barr virus genome to 64 kilobase pairs.oriP is essential for EBNA gene promoter activity in Epstein-Barr virus-immortalized lymphoblastoid cell lines.Compilation of DNA strand exchange sites for non-homologous recombination in somatic cells.Isolation and characterization of cDNA clones corresponding to transcripts from the BamHI H and F regions of the Epstein-Barr virus genome.An Epstein-Barr virus with a 58-kilobase-pair deletion that includes BARF0 transforms B lymphocytes in vitroEpstein-Barr virus intrastrain recombination in oral hairy leukoplakia.Epstein-Barr virus recombinants from overlapping cosmid fragments.The only domain which distinguishes Epstein-Barr virus latent membrane protein 2A (LMP2A) from LMP2B is dispensable for lymphocyte infection and growth transformation in vitro; LMP2A is therefore nonessential.Epstein-Barr virus nuclear proteins EBNA-3A and EBNA-3C are essential for B-lymphocyte growth transformation.Identification of critical cis elements involved in mediating Epstein-Barr virus nuclear antigen 2-dependent activity of an enhancer located upstream of the viral BamHI C promoter.Use of second-site homologous recombination to demonstrate that Epstein-Barr virus nuclear protein 3B is not important for lymphocyte infection or growth transformation in vitroA selectable marker allows investigation of a nontransforming Epstein-Barr virus mutantThe Epstein-Barr virus (EBV) DR enhancer contains two functionally different domains: domain A is constitutive and cell specific, domain B is transactivated by the EBV early protein R.Epstein-Barr virus types 1 and 2 differ in their EBNA-3A, EBNA-3B, and EBNA-3C genes.The zta transactivator involved in induction of lytic cycle gene expression in Epstein-Barr virus-infected lymphocytes binds to both AP-1 and ZRE sites in target promoter and enhancer regions.Transcription of the Epstein-Barr virus genome during latency in growth-transformed lymphocytes.In vitro transcriptional activation, dimerization, and DNA-binding specificity of the Epstein-Barr virus Zta protein.The Epstein-Barr virus (EBV) ORI1yt enhancer is not B-cell specific and does not respond synergistically to the EBV transcription factors R and Z.BHRF1, the Epstein-Barr virus gene with homology to Bc12, is dispensable for B-lymphocyte transformation and virus replicationActivation of the human T-cell leukemia virus type I long terminal repeat by 12-O-tetradecanoylphorbol-13-acetate and by tax (p40x) occurs through similar but functionally distinct target sequencesResponsiveness of the Epstein-Barr virus NotI repeat promoter to the Z transactivator is mediated in a cell-type-specific manner by two independent signal regions.Relative rates of RNA synthesis across the genome of Epstein-Barr virus are highest near oriP and oriLyt.Expression of the Epstein-Barr virus nuclear protein 2 in rodent cells.A bicistronic Epstein-Barr virus mRNA encodes two nuclear proteins in latently infected, growth-transformed lymphocytesMechanisms of nonhomologous recombination in mammalian cells.Construction and use of cDNA clones for the mapping and identification of Epstein-Barr virus early P3HR-1 mRNAs.
P2860
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P2860
Organization of the Epstein-Barr virus DNA molecule. III. Location of the P3HR-1 deletion junction and characterization of the NotI repeat units that form part of the template for an abundant 12-O-tetradecanoylphorbol-13-acetate-induced mRNA transcr
description
1983 nî lūn-bûn
@nan
1983年の論文
@ja
1983年論文
@yue
1983年論文
@zh-hant
1983年論文
@zh-hk
1983年論文
@zh-mo
1983年論文
@zh-tw
1983年论文
@wuu
1983年论文
@zh
1983年论文
@zh-cn
name
Organization of the Epstein-Ba ...... 3-acetate-induced mRNA transcr
@en
type
label
Organization of the Epstein-Ba ...... 3-acetate-induced mRNA transcr
@en
prefLabel
Organization of the Epstein-Ba ...... 3-acetate-induced mRNA transcr
@en
P2860
P1433
P1476
Organization of the Epstein-Ba ...... 3-acetate-induced mRNA transcr
@en
P2093
S D Hayward
P2860
P304
P407
P577
1983-10-01T00:00:00Z