T lymphocytes potentiate endogenous neuroprotective inflammation in a mouse model of ALS.
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Progressive changes in microglia and macrophages in spinal cord and peripheral nerve in the transgenic rat model of amyotrophic lateral sclerosisTumor necrosis factor alpha: a link between neuroinflammation and excitotoxicityMajor Histocompatibility Complex I Expression by Motor Neurons and Its Implication in Amyotrophic Lateral SclerosisAre Microglial Cells the Regulators of Lymphocyte Responses in the CNS?Neuroinflammation in motor neuron diseaseEvidence from Human and Animal Studies: Pathological Roles of CD8(+) T Cells in Autoimmune Peripheral NeuropathiesMicroglia centered pathogenesis in ALS: insights in cell interconnectivityTherapeutic neuroprotective agents for amyotrophic lateral sclerosisAblation of proliferating cells in the CNS exacerbates motor neuron disease caused by mutant superoxide dismutaseMonitoring CSF proteome alterations in amyotrophic lateral sclerosis: obstacles and perspectives in translating a novel marker panel to the clinicExcess circulating alternatively activated myeloid (M2) cells accelerate ALS progression while inhibiting experimental autoimmune encephalomyelitisMechanisms underlying inflammation in neurodegenerationMajor histocompatibility complex class I molecules protect motor neurons from astrocyte-induced toxicity in amyotrophic lateral sclerosis.A conceptual revolution in the relationships between the brain and immunity.Loss-of-function mutations in the C9ORF72 mouse ortholog cause fatal autoimmune diseaseBcl11b: A New Piece to the Complex Puzzle of Amyotrophic Lateral Sclerosis Neuropathogenesis?Immune dysregulation may contribute to disease pathogenesis in spinal muscular atrophy miceThe role of oxidative stress in degeneration of the neuromuscular junction in amyotrophic lateral sclerosisDownregulation of MicroRNA-193b-3p Promotes Autophagy and Cell Survival by Targeting TSC1/mTOR Signaling in NSC-34 Cells.Expanding Role of T Cells in Human Autoimmune Diseases of the Central Nervous System.Abnormal changes in NKT cells, the IGF-1 axis, and liver pathology in an animal model of ALS.The role of microglia in human disease: therapeutic tool or target?SOD1(G93A) transgenic mouse CD4(+) T cells mediate neuroprotection after facial nerve axotomy when removed from a suppressive peripheral microenvironment.Vitamin D3 deficiency differentially affects functional and disease outcomes in the G93A mouse model of amyotrophic lateral sclerosis.Levels of soluble platelet endothelial cell adhesion molecule-1 and P-selectin are decreased in children with autism spectrum disorder.Possible association between expression of chemokine receptor-2 (CCR2) and amyotrophic lateral sclerosis (ALS) patients of North India.Deleterious effects of lymphocytes at the early stage of neurodegeneration in an animal model of amyotrophic lateral sclerosis.Myelosuppressive conditioning using busulfan enables bone marrow cell accumulation in the spinal cord of a mouse model of amyotrophic lateral sclerosis.Vector-free and transgene-free human iPS cells differentiate into functional neurons and enhance functional recovery after ischemic stroke in mice.A neurodegeneration-specific gene-expression signature of acutely isolated microglia from an amyotrophic lateral sclerosis mouse modelBAFF controls neural cell survival through BAFF receptor.Neuroinflammation modulates distinct regional and temporal clinical responses in ALS mice.Endogenous regulatory T lymphocytes ameliorate amyotrophic lateral sclerosis in mice and correlate with disease progression in patients with amyotrophic lateral sclerosis.Early-stage treatment with Withaferin A reduces levels of misfolded superoxide dismutase 1 and extends lifespan in a mouse model of amyotrophic lateral sclerosis.Dual destructive and protective roles of adaptive immunity in neurodegenerative disorders.Transcription factor p53 influences microglial activation phenotypeEffect of thymic stimulation of CD4+ T cell expansion on disease onset and progression in mutant SOD1 mice.The Role of immune and inflammatory mechanisms in ALS.Macrophage migration inhibitory factor as a chaperone inhibiting accumulation of misfolded SOD1.Protective and Toxic Neuroinflammation in Amyotrophic Lateral Sclerosis.
P2860
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P2860
T lymphocytes potentiate endogenous neuroprotective inflammation in a mouse model of ALS.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
T lymphocytes potentiate endogenous neuroprotective inflammation in a mouse model of ALS.
@en
type
label
T lymphocytes potentiate endogenous neuroprotective inflammation in a mouse model of ALS.
@en
prefLabel
T lymphocytes potentiate endogenous neuroprotective inflammation in a mouse model of ALS.
@en
P2093
P2860
P356
P1476
T lymphocytes potentiate endogenous neuroprotective inflammation in a mouse model of ALS
@en
P2093
Bela Kosaras
Michael C Carroll
Stefanos A Tsiftsoglou
Timothy K Vartanian
P2860
P304
17913-17918
P356
10.1073/PNAS.0804610105
P407
P577
2008-11-07T00:00:00Z