Oxidative inactivation of key mitochondrial proteins leads to dysfunction and injury in hepatic ischemia reperfusion. - Wikidata - wikimedia - TriplyDB

Oxidative inactivation of key mitochondrial proteins leads to dysfunction and injury in hepatic ischemia reperfusion.

Oxidative inactivation of key mitochondrial proteins leads to dysfunction and injury in hepatic ischemia reperfusion.

http://www.wikidata.org/entity/Q37002521

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Mitochondrial dysfunction and tissue injury by alcohol, high fat, nonalcoholic substances and pathological conditions through post-translational protein modifications Functional roles of protein nitration in acute and chronic liver diseases Dysfunctional mitochondrial bioenergetics and the pathogenesis of hepatic disorders Phenotype of transgenic mice overexpressed with inducible nitric oxide synthase in the retina Pathogenic role of oxidative and nitrosative stress in primary biliary cirrhosis.Inhibition of hepatic mitochondrial aldehyde dehydrogenase by carbon tetrachloride through JNK-mediated phosphorylation.Cannabinoid-2 receptor limits inflammation, oxidative/nitrosative stress, and cell death in nephropathy.A simple method to systematically study oxidatively modified proteins in biological samples and its applications Posttranslational modification of differentially expressed mitochondrial proteins in the retina during early experimental autoimmune uveitis.Mechanisms of MDMA (ecstasy)-induced oxidative stress, mitochondrial dysfunction, and organ damage.Superoxide dismutase mimics: chemistry, pharmacology, and therapeutic potential Divergent roles of superoxide and nitric oxide in liver ischemia and reperfusion injury.Lack of PPARα exacerbates lipopolysaccharide-induced liver toxicity through STAT1 inflammatory signaling and increased oxidative/nitrosative stress Cannabidiol protects against hepatic ischemia/reperfusion injury by attenuating inflammatory signaling and response, oxidative/nitrative stress, and cell death.Zidovudine (AZT) and hepatic lipid accumulation: implication of inflammation, oxidative and endoplasmic reticulum stress mediators.The effects of low-intensity laser therapy on hepatic ischemia-reperfusion injury in a rat model.Post-translational modifications of mitochondrial aldehyde dehydrogenase and biomedical implications.Diverse functions of cationic Mn(III) N-substituted pyridylporphyrins, recognized as SOD mimics Sulforaphane, a natural constituent of broccoli, prevents cell death and inflammation in nephropathy Bioenergetic programming of macrophages by the apolipoprotein A-I mimetic peptide 4F Increased Sensitivity to Binge Alcohol-Induced Gut Leakiness and Inflammatory Liver Disease in HIV Transgenic Rats.Increased oxidative-modifications of cytosolic proteins in 3,4-methylenedioxymethamphetamine (MDMA, ecstasy)-exposed rat liver.Nitric oxide in liver diseases.Matrix metalloproteinase-9 contributes to parenchymal hemorrhage and necrosis in the remnant liver after extended hepatectomy in mice Fatty liver is associated with reduced SIRT3 activity and mitochondrial protein hyperacetylation Δ8-Tetrahydrocannabivarin prevents hepatic ischaemia/reperfusion injury by decreasing oxidative stress and inflammatory responses through cannabinoid CB2 receptors.A new cannabinoid CB2 receptor agonist HU-910 attenuates oxidative stress, inflammation and cell death associated with hepatic ischaemia/reperfusion injury Mitochondrial reactive oxygen species generation triggers inflammatory response and tissue injury associated with hepatic ischemia-reperfusion: therapeutic potential of mitochondrially targeted antioxidants.Critical role of cytochrome P450 2E1 (CYP2E1) in the development of high fat-induced non-alcoholic steatohepatitis.Fibronectin-α4β1 interactions in hepatic cold ischemia and reperfusion injury: regulation of MMP-9 and MT1-MMP via the p38 MAPK pathway.Mouse liver protein sulfhydryl depletion after acetaminophen exposure.Role of CYP2E1 in mitochondrial dysfunction and hepatic tissue injury in alcoholic and non-alcoholic diseases.Cytochrome P450-2E1 promotes aging-related hepatic steatosis, apoptosis and fibrosis through increased nitroxidative stress.Protective mechanisms of hypothermia in liver surgery and transplantation.Mitochondrial dysfunction and cell death in neurodegenerative diseases through nitroxidative stress Increased nitroxidative stress promotes mitochondrial dysfunction in alcoholic and nonalcoholic fatty liver disease.A silybin-phospholipids complex counteracts rat fatty liver degeneration and mitochondrial oxidative changes.Robust protein nitration contributes to acetaminophen-induced mitochondrial dysfunction and acute liver injury Molecular mechanisms of alcoholic fatty liver Role of superoxide, nitric oxide, and peroxynitrite in doxorubicin-induced cell death in vivo and in vitro

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Oxidative inactivation of key mitochondrial proteins leads to dysfunction and injury in hepatic ischemia reperfusion.

http://www.wikidata.org/entity/Q37002521

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 25 June 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Oxidative inactivation of key ...... hepatic ischemia reperfusion.

@en

Oxidative inactivation of key ...... hepatic ischemia reperfusion.

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type

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Oxidative inactivation of key ...... hepatic ischemia reperfusion.

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Oxidative inactivation of key ...... hepatic ischemia reperfusion.

@nl

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Oxidative inactivation of key ...... hepatic ischemia reperfusion.

@en

Oxidative inactivation of key ...... hepatic ischemia reperfusion.

@nl

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J.GASTRO.2008.06.048

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Gastroenterology

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Oxidative inactivation of key ...... hepatic ischemia reperfusion.

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Brian L Hood

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Byoung-Joon Song

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Kwan-Hoon Moon

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Mohamed A Abdelmegeed

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Mohanraj Rajesh

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Thomas P Conrads

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Timothy D Veenstra

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Yong-Il Kwon

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P2860

Nitric oxide and peroxynitrite in health and disease

Therapeutic effects of xanthine oxidase inhibitors: renaissance half a century after the discovery of allopurinol

Inactivation of cytosolic aldehyde dehydrogenase via S-nitrosylation in ethanol-exposed rat liver

Peroxynitrite: biochemistry, pathophysiology and development of therapeutics

Protective effect of allopurinol on hepatic energy metabolism in ischemic and reperfused rat liver.

Mitochondrial dysfunction in cardiac ischemia-reperfusion injury: ROS from complex I, without inhibition.

Potential biomarkers for ischemic heart damage identified in mitochondrial proteins by comparative proteomics.

Increased oxidation and degradation of cytosolic proteins in alcohol-exposed mouse liver and hepatoma cells.

Proteomic analysis of liver tissues subjected to early ischemia/reperfusion injury during human orthotopic liver transplantation.

Inhibition of mitochondrial aldehyde dehydrogenase by nitric oxide-mediated S-nitrosylation.

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1344-1357

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scholarly article

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10.1053/J.GASTRO.2008.06.048

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4

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Partha Mukhopadhyay

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2008-06-25T00:00:00Z

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23244486

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