Adaptation to ER stress as a driver of malignancy and resistance to therapy in human melanoma.
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St John's Wort (Hypericum perforatum L.) photomedicine: hypericin-photodynamic therapy induces metastatic melanoma cell deathThe endoplasmic reticulum stress response in aging and age-related diseasesEndoplasmic reticulum stress-induced resistance to doxorubicin is reversed by paeonol treatment in human hepatocellular carcinoma cellsC/EBP-beta regulates endoplasmic reticulum stress-triggered cell death in mouse and human modelsEndoplasmic Reticulum Stress, Unfolded Protein Response, and Cancer Cell FateFateful music from a talented orchestra with a wicked conductor: Connection between oncogenic BRAF, ER stress, and autophagy in human melanoma.Enhanced lysosomal activity is involved in Bax inhibitor-1-induced regulation of the endoplasmic reticulum (ER) stress response and cell death against ER stress: involvement of vacuolar H+-ATPase (V-ATPase).Novel role of VMP1 as modifier of the pancreatic tumor cell response to chemotherapeutic drugs.Targeted activation of innate immunity for therapeutic induction of autophagy and apoptosis in melanoma cellsAutophagy induction and CHOP under-expression promotes survival of fibroblasts from rheumatoid arthritis patients under endoplasmic reticulum stressAutophagy protects against oxaliplatin-induced cell death via ER stress and ROS in Caco-2 cellsEts-1 mediates upregulation of Mcl-1 downstream of XBP-1 in human melanoma cells upon ER stress.MicroRNA-149*, a p53-responsive microRNA, functions as an oncogenic regulator in human melanomaCell intrinsic and extrinsic activators of the unfolded protein response in cancer: Mechanisms and targets for therapySuppression of PP2A is critical for protection of melanoma cells upon endoplasmic reticulum stress.RIPK1 regulates survival of human melanoma cells upon endoplasmic reticulum stress through autophagy.miR-663 overexpression induced by endoplasmic reticulum stress modulates hepatocellular carcinoma cell apoptosis via transforming growth factor beta 1.Vemurafenib potently induces endoplasmic reticulum stress-mediated apoptosis in BRAFV600E melanoma cells.Human melanoma cells under endoplasmic reticulum stress acquire resistance to microtubule-targeting drugs through XBP-1-mediated activation of Akt.Human melanoma cells under endoplasmic reticulum stress are more susceptible to apoptosis induced by the BH3 mimetic obatoclax.IRE1α-XBP1 pathway promotes melanoma progression by regulating IL-6/STAT3 signaling.The protein disulfide isomerases PDIA4 and PDIA6 mediate resistance to cisplatin-induced cell death in lung adenocarcinoma.Endoplasmic reticulum stress response in cancer: molecular mechanism and therapeutic potential.Molecular chaperones as therapeutic targets to counteract proteostasis defects.The gluttonous side of malignant melanoma: basic and clinical implications of macroautophagy.ER stress-induced autophagy in melanoma.Emerging targets for combination therapy in melanomas.BRAF inhibitors amplify the pro-apoptotic activity of MEK inhibitors by inducing ER stress in NRAS-mutant melanoma.Downregulation of pyrroline-5-carboxylate reductase-2 induces the autophagy of melanoma cells via AMPK/mTOR pathway.Cytotoxic mechanisms of panduratin A on A375 melanoma cells: A quantitative and temporal proteomics analysis.Oncogenic BRAF induces chronic ER stress condition resulting in increased basal autophagy and apoptotic resistance of cutaneous melanoma.Cell-type variation in stress responses as a consequence of manipulating GRP78 expression in neuroectodermal cells.Involvement of ER stress and activation of apoptotic pathways in fisetin induced cytotoxicity in human melanoma.Loss of Oca2 disrupts the unfolded protein response and increases resistance to endoplasmic reticulum stress in melanocytes.T-type calcium channel blockers inhibit autophagy and promote apoptosis of malignant melanoma cells.Melatonin sensitizes human hepatoma cells to endoplasmic reticulum stress-induced apoptosis.Targeting GRP78 to enhance melanoma cell death.Glucose-regulated protein 78 antagonizes cisplatin and adriamycin in human melanoma cells.The unfolded protein response in melanocytes: activation in response to chemical stressors of the endoplasmic reticulum and tyrosinase misfolding.Lactate dehydrogenase 5 expression in melanoma increases with disease progression and is associated with expression of Bcl-XL and Mcl-1, but not Bcl-2 proteins.
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Adaptation to ER stress as a driver of malignancy and resistance to therapy in human melanoma.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Adaptation to ER stress as a d ...... to therapy in human melanoma.
@en
Adaptation to ER stress as a d ...... to therapy in human melanoma.
@nl
type
label
Adaptation to ER stress as a d ...... to therapy in human melanoma.
@en
Adaptation to ER stress as a d ...... to therapy in human melanoma.
@nl
prefLabel
Adaptation to ER stress as a d ...... to therapy in human melanoma.
@en
Adaptation to ER stress as a d ...... to therapy in human melanoma.
@nl
P2860
P1476
Adaptation to ER stress as a d ...... to therapy in human melanoma.
@en
P2093
Peter Hersey
Xu Dong Zhang
P2860
P304
P356
10.1111/J.1755-148X.2008.00467.X
P577
2008-06-01T00:00:00Z