Excessive alcohol consumption is blocked by glial cell line-derived neurotrophic factor.
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Dopamine D2 receptor activation leads to an up-regulation of glial cell line-derived neurotrophic factor via Gβγ-Erk1/2-dependent induction of Zif268Translational behaviour-genetic studies of alcohol: are we there yet?Recent advances in the discovery and preclinical testing of novel compounds for the prevention and/or treatment of alcohol use disorders.Noribogaine, but not 18-MC, exhibits similar actions as ibogaine on GDNF expression and ethanol self-administrationLesions of the lateral habenula increase voluntary ethanol consumption and operant self-administration, block yohimbine-induced reinstatement of ethanol seeking, and attenuate ethanol-induced conditioned taste aversionVoluntary Alcohol Intake following Blast Exposure in a Rat Model of Mild Traumatic Brain InjuryThe glucagon-like peptide 1 receptor agonist liraglutide attenuates the reinforcing properties of alcohol in rodentsPeripherally circulating ghrelin does not mediate alcohol-induced reward and alcohol intake in rodentsTargeted delivery of GDNF through the blood-brain barrier by MRI-guided focused ultrasound.Long-lasting adaptations of the NR2B-containing NMDA receptors in the dorsomedial striatum play a crucial role in alcohol consumption and relapse.Intermittent ethanol access schedule in rats as a preclinical model of alcohol abuse.Role of BDNF and GDNF in drug reward and relapse: a reviewCabergoline decreases alcohol drinking and seeking behaviors via glial cell line-derived neurotrophic factor.An evolutionary conserved role for anaplastic lymphoma kinase in behavioral responses to ethanol.Role for mammalian target of rapamycin complex 1 signaling in neuroadaptations underlying alcohol-related disordersThe histone deacetylase inhibitor sodium butyrate decreases excessive ethanol intake in dependent animals.DlgS97/SAP97, a neuronal isoform of discs large, regulates ethanol toleranceDevelopment of the Neuroimmune Modulator Ibudilast for the Treatment of Alcoholism: A Randomized, Placebo-Controlled, Human Laboratory Trial.Regulation of operant oral ethanol self-administration: a dose-response curve study in rats.Endogenous GDNF in ventral tegmental area and nucleus accumbens does not play a role in the incubation of heroin cravingGhrelin receptor (GHS-R1A) antagonism suppresses both alcohol consumption and the alcohol deprivation effect in rats following long-term voluntary alcohol consumption.Glial cell line-derived neurotrophic factor reverses alcohol-induced allostasis of the mesolimbic dopaminergic system: implications for alcohol reward and seekingEthanol-induced increase in Fyn kinase activity in the dorsomedial striatum is associated with subcellular redistribution of protein tyrosine phosphatase αLow frequency electroacupuncture selectively decreases voluntarily ethanol intake in rats.Insulin attenuates the acquisition and expression of ethanol-induced locomotor sensitization in DBA/2J mice.AKT signaling pathway in the nucleus accumbens mediates excessive alcohol drinking behaviors.Positive autoregulation of GDNF levels in the ventral tegmental area mediates long-lasting inhibition of excessive alcohol consumptionStriatal involvement in human alcoholism and alcohol consumption, and withdrawal in animal modelsEthanol and nicotine interaction within the posterior ventral tegmental area in male and female alcohol-preferring rats: evidence of synergy and differential gene activation in the nucleus accumbens shellD-Serine and D-Cycloserine Reduce Compulsive Alcohol Intake in RatsThe Class I-Specific HDAC Inhibitor MS-275 Decreases Motivation to Consume Alcohol and Relapse in Heavy Drinking Rats.Repeated Binge Drinking Increases Perineuronal Nets in the Insular Cortex.Ethanol consumption in the Sprague-Dawley rat increases sensitivity of the dorsal raphe nucleus to 5,7-dihydroxytryptamine.The small G protein H-Ras in the mesolimbic system is a molecular gateway to alcohol-seeking and excessive drinking behaviorsGDNF is a novel ethanol-responsive gene in the VTA: implications for the development and persistence of excessive drinking.GDNF--a potential target to treat addiction.Disruption of alcohol-related memories by mTORC1 inhibition prevents relapse.Protein tyrosine phosphatase α in the dorsomedial striatum promotes excessive ethanol-drinking behaviors.The Neurotrophic Factor Receptor p75 in the Rat Dorsolateral Striatum Drives Excessive Alcohol DrinkingLesion of the rostromedial tegmental nucleus increases voluntary ethanol consumption and accelerates extinction of ethanol-induced conditioned taste aversion.
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P2860
Excessive alcohol consumption is blocked by glial cell line-derived neurotrophic factor.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on February 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Excessive alcohol consumption is blocked by glial cell line-derived neurotrophic factor.
@en
Excessive alcohol consumption is blocked by glial cell line-derived neurotrophic factor.
@nl
type
label
Excessive alcohol consumption is blocked by glial cell line-derived neurotrophic factor.
@en
Excessive alcohol consumption is blocked by glial cell line-derived neurotrophic factor.
@nl
prefLabel
Excessive alcohol consumption is blocked by glial cell line-derived neurotrophic factor.
@en
Excessive alcohol consumption is blocked by glial cell line-derived neurotrophic factor.
@nl
P2093
P2860
P1433
P1476
Excessive alcohol consumption is blocked by glial cell line-derived neurotrophic factor.
@en
P2093
Ryoji Amamoto
Sebastien Carnicella
P2860
P356
10.1016/J.ALCOHOL.2008.12.001
P577
2009-02-01T00:00:00Z