Loss of heterozygosity on the short arm of chromosome 17 is associated with high proliferative capacity and DNA aneuploidy in primary human breast cancer.
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Flow cytometric analysis of neoplastic nodules and hepatocellular carcinomas induced by ciprofibrate in the ratMutation in Rpa1 results in defective DNA double-strand break repair, chromosomal instability and cancer in miceTumor-suppressor genes: cardinal factors in inherited predisposition to human cancersRegulatory functions of the N-terminal domain of the 70-kDa subunit of replication protein A (RPA)Establishment and characterization of 6 novel patient-derived primary pancreatic ductal adenocarcinoma cell lines from Korean pancreatic cancer patients.Molecular biology in cytopathology: current applications and future directions.Repair-specific functions of replication protein A.Glucose-dependent insulinotropic peptide receptor overexpression in adrenocortical hyperplasia in MEN1 syndrome without loss of heterozygosity at the 11q13 locus.The polymerase chain reaction: its use in the molecular characterization and diagnosis of cancers.Detection of numerical and structural alterations and fusion of chromosomes 16 and 1 in low-grade papillary breast carcinoma by fluorescence in situ hybridization.Analysis of chromosome 17p13 (p53 locus) alterations in gastric carcinoma cells by dual-color fluorescence in situ hybridization.TP53 mutation status and gene expression profiles are powerful prognostic markers of breast cancer.Metastatic EML4-ALK fusion detected by circulating DNA genotyping in an EGFR-mutated NSCLC patient and successful management by adding ALK inhibitors: a case report.The TP53 tumour suppressor gene in colorectal carcinomas. I. Genetic alterations on chromosome 17Allelic loss on distal chromosome 17p is associated with poor prognosis in a group of Brazilian breast cancer patientsAllelic imbalance at chromosome 17p13.3 (YNZ22) in breast cancer is independent of p53 mutation or p53 overexpression and is associated with poor prognosis at medium-term follow-upGenetic analysis using genomic representations.Different mutant/wild-type p53 combinations cause a spectrum of increased invasive potential in nonmalignant immortalized human mammary epithelial cellsFunctional evidence for a second tumor suppressor gene on human chromosome 17.Cause and consequences of genetic and epigenetic alterations in human cancer.Mutations in p53 as potential molecular markers for human breast cancer.Physical deletion of the p53 gene in bladder cancer. Detection by fluorescence in situ hybridizationDetailed deletion mapping in sporadic breast cancer at chromosomal region 17p13 distal to the TP53 gene: association with clinicopathological parameters.Heterozygous deletion of chromosome 17p renders prostate cancer vulnerable to inhibition of RNA polymerase II
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Loss of heterozygosity on the short arm of chromosome 17 is associated with high proliferative capacity and DNA aneuploidy in primary human breast cancer.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on May 1991
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Loss of heterozygosity on the ...... n primary human breast cancer.
@en
Loss of heterozygosity on the ...... n primary human breast cancer.
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type
label
Loss of heterozygosity on the ...... n primary human breast cancer.
@en
Loss of heterozygosity on the ...... n primary human breast cancer.
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prefLabel
Loss of heterozygosity on the ...... n primary human breast cancer.
@en
Loss of heterozygosity on the ...... n primary human breast cancer.
@nl
P2093
P2860
P356
P1476
Loss of heterozygosity on the ...... n primary human breast cancer.
@en
P2093
A Neubauer
E S Goldman
F M Waldman
P2860
P304
P356
10.1073/PNAS.88.9.3847
P407
P577
1991-05-01T00:00:00Z