Impairment of insulin-stimulated glucose transport and ERK activation by adipocyte-specific knockout of PKC-λ produces a phenotype characterized by diminished adiposity and enhanced insulin suppression of hepatic gluconeogenesis.
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Hyperglycemic Stress and Carbon Stress in Diabetic GlucotoxicityPKCλ haploinsufficiency prevents diabetes by a mechanism involving alterations in hepatic enzymes.Optogenetic activation reveals distinct roles of PIP3 and Akt in adipocyte insulin action.Protein Kinase C as Regulator of Vascular Smooth Muscle Function and Potential Target in Vascular Disorders.
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Impairment of insulin-stimulated glucose transport and ERK activation by adipocyte-specific knockout of PKC-λ produces a phenotype characterized by diminished adiposity and enhanced insulin suppression of hepatic gluconeogenesis.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 10 September 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Impairment of insulin-stimulat ...... on of hepatic gluconeogenesis.
@en
Impairment of insulin-stimulat ...... on of hepatic gluconeogenesis.
@nl
type
label
Impairment of insulin-stimulat ...... on of hepatic gluconeogenesis.
@en
Impairment of insulin-stimulat ...... on of hepatic gluconeogenesis.
@nl
prefLabel
Impairment of insulin-stimulat ...... on of hepatic gluconeogenesis.
@en
Impairment of insulin-stimulat ...... on of hepatic gluconeogenesis.
@nl
P2093
P2860
P356
P1433
P1476
Impairment of insulin-stimulat ...... on of hepatic gluconeogenesis.
@en
P2093
Michael J Jurzak
Michael Leitges
Mini P Sajan
Robert V Farese
Ursula Braun
Varman T Samuels
P2860
P356
10.4161/ADIP.26305
P577
2013-09-10T00:00:00Z