SMAC/Diablo-dependent apoptosis induced by nonsteroidal antiinflammatory drugs (NSAIDs) in colon cancer cells.
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P1343
PUMA Dissociates Bax and Bcl-X(L) to induce apoptosis in colon cancer cellsPUMA regulates intestinal progenitor cell radiosensitivity and gastrointestinal syndromeSulindac induces apoptosis and inhibits tumor growth in vivo in head and neck squamous cell carcinomap53 independent induction of PUMA mediates intestinal apoptosis in response to ischaemia-reperfusionSMAC/Diablo mediates the proapoptotic function of PUMA by regulating PUMA-induced mitochondrial eventsSmac modulates chemosensitivity in head and neck cancer cells through the mitochondrial apoptotic pathwayBH3-based fusion artificial peptide induces apoptosis and targets human colon cancer.PUMA induction by FoxO3a mediates the anticancer activities of the broad-range kinase inhibitor UCN-01.Chemoprevention by nonsteroidal anti-inflammatory drugs eliminates oncogenic intestinal stem cells via SMAC-dependent apoptosis.BID mediates selective killing of APC-deficient cells in intestinal tumor suppression by nonsteroidal antiinflammatory drugs.Growth factors protect intestinal stem cells from radiation-induced apoptosis by suppressing PUMA through the PI3K/AKT/p53 axis.SapC-DOPS nanovesicles induce Smac- and Bax-dependent apoptosis through mitochondrial activation in neuroblastomas.The nuclear function of p53 is required for PUMA-mediated apoptosis induced by DNA damage.Inhibiting oncogenic signaling by sorafenib activates PUMA via GSK3β and NF-κB to suppress tumor cell growthThe role of mitochondria in apoptosis*.Cytotoxicity and cell cycle arrest induced by andrographolide lead to programmed cell death of MDA-MB-231 breast cancer cell lineRole of apoptosis in colon cancer biology, therapy, and preventionRole of Bcl-2 family proteins and caspases in the regulation of apoptosis.Chemoprevention in experimental animals.PINCH-1 regulates the ERK-Bim pathway and contributes to apoptosis resistance in cancer cells.Consumption of Red/Processed Meat and Colorectal Carcinoma: Possible Mechanisms Underlying the Significant Association.Targeting Bax interaction sites reveals that only homo-oligomerization sites are essential for its activation.Promotion of Caspase Activation by Caspase-9-mediated Feedback Amplification of Mitochondrial Damage.Smac deficiency affects endoplasmic reticulum stress-induced apoptosis in human colon cancer cells.Sulindac Sulfide Differentially Induces Apoptosis in Smac-Proficient and -Deficient Human Colon Cancer Cells.TAp73 promotes cell survival upon genotoxic stress by inhibiting p53 activity.An apoptosis-independent role of SMAC in tumor suppressionDownregulation of Dkk3 activates beta-catenin/TCF-4 signaling in lung cancer.
P2860
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P2860
SMAC/Diablo-dependent apoptosis induced by nonsteroidal antiinflammatory drugs (NSAIDs) in colon cancer cells.
description
2004 nî lūn-bûn
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2004年の論文
@ja
2004年学术文章
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2004年学术文章
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2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
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2004年學術文章
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name
SMAC/Diablo-dependent apoptosi ...... NSAIDs) in colon cancer cells.
@en
SMAC/Diablo-dependent apoptosis induced by nonsteroidal antiinflammatory drugs
@nl
type
label
SMAC/Diablo-dependent apoptosi ...... NSAIDs) in colon cancer cells.
@en
SMAC/Diablo-dependent apoptosis induced by nonsteroidal antiinflammatory drugs
@nl
prefLabel
SMAC/Diablo-dependent apoptosi ...... NSAIDs) in colon cancer cells.
@en
SMAC/Diablo-dependent apoptosis induced by nonsteroidal antiinflammatory drugs
@nl
P2093
P2860
P50
P356
P1476
SMAC/Diablo-dependent apoptosi ...... NSAIDs) in colon cancer cells.
@en
P2093
Christoph Lengauer
Craig Seaman
Manu Kohli
P2860
P304
16897-16902
P356
10.1073/PNAS.0403405101
P407
P4510
P577
2004-11-19T00:00:00Z