How vaccinia virus has evolved to subvert the host immune response.
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Structural Conservation and Functional Diversity of the Poxvirus Immune Evasion (PIE) Domain SuperfamilyCytokine production associated with smallpox vaccine responsesSubversion of cytokine networks by virally encoded decoy receptorsCharacterization and Structure of the Vaccinia Virus NF-κB Antagonist A46Poxviruses deploy genomic accordions to adapt rapidly against host antiviral defensesStructure of vaccinia virus A46, an inhibitor of TLR4 signaling pathway, shows the conformation of VIPER motif.Systems kinomics demonstrates Congo Basin monkeypox virus infection selectively modulates host cell signaling responses as compared to West African monkeypox virus.Poxvirus targeting of E3 ligase β-TrCP by molecular mimicry: a mechanism to inhibit NF-κB activation and promote immune evasion and virulence.Host factor SAMHD1 restricts DNA viruses in non-dividing myeloid cellsMyxoma virus protein M029 is a dual function immunomodulator that inhibits PKR and also conscripts RHA/DHX9 to promote expanded host tropism and viral replicationIn vitro inhibition of monkeypox virus production and spread by Interferon-β.Vaccinia Virus Immunomodulator A46: A Lipid and Protein-Binding Scaffold for Sequestering Host TIR-Domain ProteinsVaccinia virus F1L protein promotes virulence by inhibiting inflammasome activationVaccinia virus protein K7 is a virulence factor that alters the acute immune response to infectionBoth CD8+ and CD4+ T Cells Contribute to Corneal Clouding and Viral Clearance following Vaccinia Virus Infection in C57BL/6 MiceVaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulenceGenomic variability of monkeypox virus among humans, Democratic Republic of the CongoCells under siege: viral glycoprotein interactions at the cell surface.Viral modulation of programmed necrosis.Vaccination and the TAP-independent antigen processing pathways.Vaccinia virus inhibits NF-κB-dependent gene expression downstream of p65 translocation.Negative regulation of the inflammasome: keeping inflammation under control.Advances in Antiviral Therapies Targeting Toll-like Receptors.Lister strain vaccinia virus with thymidine kinase gene deletion is a tractable platform for development of a new generation of oncolytic virus.Virus-encoded ectopic CD74 enhances poxvirus vaccine efficacy.Protein kinase C overexpression does not enhance immune-stimulatory surface markers of vaccinia-infected dendritic cells and DC cell lines.The vaccinia virus-encoded Bcl-2 homologues do not act as direct Bax inhibitors.Predictive and Prognostic Clinical Variables in Cancer Patients Treated With Adenoviral Oncolytic Immunotherapy.IL-1R Type 1-Deficient Mice Demonstrate an Impaired Host Immune Response against Cutaneous Vaccinia Virus Infection.Mapping the IkappaB kinase beta (IKKbeta)-binding interface of the B14 protein, a vaccinia virus inhibitor of IKKbeta-mediated activation of nuclear factor kappaB.Innate Immune Recognition of Double-stranded RNA Triggers Increased Expression of NKG2D Ligands After Virus Infection.Evolution of Synonymous Codon Usage Bias in West African and Central African Strains of Monkeypox Virus.The RNA-binding protein of a double-stranded RNA virus acts like a scaffold protein
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How vaccinia virus has evolved to subvert the host immune response.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 17 March 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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How vaccinia virus has evolved to subvert the host immune response.
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How vaccinia virus has evolved to subvert the host immune response.
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How vaccinia virus has evolved to subvert the host immune response.
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How vaccinia virus has evolved to subvert the host immune response.
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How vaccinia virus has evolved to subvert the host immune response.
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How vaccinia virus has evolved to subvert the host immune response.
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How vaccinia virus has evolved to subvert the host immune response
@en
P2093
Geoffrey L Smith
Jonathan M Grimes
Samantha Cooray
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P304
P356
10.1016/J.JSB.2011.03.010
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2011-03-17T00:00:00Z