Co-expression of MGMT(P140K) and alpha-L-iduronidase in primary hepatocytes from mucopolysaccharidosis type I mice enables efficient selection with metabolic correction.
about
Cell- and gene-based therapeutic approaches for neurological deficits in mucopolysaccharidosesNormalization and improvement of CNS deficits in mice with Hurler syndrome after long-term peripheral delivery of BBB-targeted iduronidase.Engineering a lysosomal enzyme with a derivative of receptor-binding domain of apoE enables delivery across the blood-brain barrierMultifaceted roles of alkyltransferase and related proteins in DNA repair, DNA damage, resistance to chemotherapy, and research tools.
P2860
Co-expression of MGMT(P140K) and alpha-L-iduronidase in primary hepatocytes from mucopolysaccharidosis type I mice enables efficient selection with metabolic correction.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Co-expression of MGMT(P140K) a ...... ion with metabolic correction.
@en
type
label
Co-expression of MGMT(P140K) a ...... ion with metabolic correction.
@en
prefLabel
Co-expression of MGMT(P140K) a ...... ion with metabolic correction.
@en
P2093
P2860
P356
P1476
Co-expression of MGMT(P140K) a ...... ion with metabolic correction.
@en
P2093
D Nicole Worsham
Daren Wang
P2860
P304
P356
10.1002/JGM.1141
P577
2008-03-01T00:00:00Z