Genotoxic stress causes the accumulation of the splicing regulator Sam68 in nuclear foci of transcriptionally active chromatin.
about
The RNA recognition motif protein RBM11 is a novel tissue-specific splicing regulatorThe RNA Splicing Response to DNA DamageSplicing Regulation: A Molecular Device to Enhance Cancer Cell AdaptationHerpes simplex virus 2 infection impacts stress granule accumulationThe RNA-binding protein Sam68 regulates expression and transcription function of the androgen receptor splice variant AR-V7SpliceAid 2: a database of human splicing factors expression data and RNA target motifs.The role of nuclear bodies in gene expression and disease.Regulation of HuR by DNA Damage Response Kinases.The RNA binding protein SAM68 transiently localizes in the chromatoid body of male germ cells and influences expression of select microRNAs.To be or not to be: the regulation of mRNA fate as a survival strategy during mammalian hibernation.Soluble programmed cell death receptor-1 (sPD-1): a potential biomarker with anti-inflammatory properties in human and experimental acute respiratory distress syndrome (ARDS).Role of Sam68 in post-transcriptional gene regulation.The centrosomal kinase NEK2 is a novel splicing factor kinase involved in cell survival.Pre-mRNA processing factors meet the DNA damage response.On the origins of the androgen receptor low molecular weight species.RNA splicing: a new player in the DNA damage responseModulation of PKM alternative splicing by PTBP1 promotes gemcitabine resistance in pancreatic cancer cells.Mitoxantrone, More than Just Another Topoisomerase II Poison.Integrinβ1 modulates tumour resistance to gemcitabine and serves as an independent prognostic factor in pancreatic adenocarcinomas.Alternative splicing and cell survival: from tissue homeostasis to disease.The transcriptional co-activator SND1 is a novel regulator of alternative splicing in prostate cancer cells.Time course changes of anti- and pro-apoptotic proteins in apigenin-induced genotoxicity.SRSF10 Connects DNA Damage to the Alternative Splicing of Transcripts Encoding Apoptosis, Cell-Cycle Control, and DNA Repair Factors.T cell receptor (TCR)-induced tyrosine phosphorylation dynamics identifies THEMIS as a new TCR signalosome component.miR‑30c suppresses prostate cancer survival by targeting the ASF/SF2 splicing factor oncoprotein.An emerging role for nuclear RNA-mediated responses to genotoxic stress.Alternative polyadenylation of ZEB1 promotes its translation during genotoxic stress in pancreatic cancer cells.Clinical significance and effect of Sam68 expression in gastric cancer.A recently evolved class of alternative 3'-terminal exons involved in cell cycle regulation by topoisomerase inhibitors.Apoptotic Cell-Derived Extracellular Vesicles Promote Malignancy of Glioblastoma Via Intercellular Transfer of Splicing Factors.
P2860
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P2860
Genotoxic stress causes the accumulation of the splicing regulator Sam68 in nuclear foci of transcriptionally active chromatin.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
Genotoxic stress causes the ac ...... criptionally active chromatin.
@en
Genotoxic stress causes the ac ...... criptionally active chromatin.
@nl
type
label
Genotoxic stress causes the ac ...... criptionally active chromatin.
@en
Genotoxic stress causes the ac ...... criptionally active chromatin.
@nl
prefLabel
Genotoxic stress causes the ac ...... criptionally active chromatin.
@en
Genotoxic stress causes the ac ...... criptionally active chromatin.
@nl
P2860
P356
P1476
Genotoxic stress causes the ac ...... scriptionally active chromatin
@en
P2093
Roberta Busà
P2860
P304
P356
10.1093/NAR/GKQ004
P407
P577
2010-01-27T00:00:00Z