Quantitative network signal combinations downstream of TCR activation can predict IL-2 production response.
about
Metabolomics of oxidative stress in recent studies of endogenous and exogenously administered intermediate metabolitesMultiscale models of cell signaling.TNF-insulin crosstalk at the transcription factor GATA6 is revealed by a model that links signaling and transcriptomic data tensors.Models of signalling networks - what cell biologists can gain from them and give to themModeling the latent dimensions of multivariate signaling datasets.Comparative analysis of transcriptional profiling of CD3+, CD4+ and CD8+ T cells identifies novel immune response players in T-cell activation.Transforming Boolean models to continuous models: methodology and application to T-cell receptor signaling.RAS mutations affect tumor necrosis factor-induced apoptosis in colon carcinoma cells via ERK-modulatory negative and positive feedback circuits along with non-ERK pathway effectsPredicting cytotoxic T-cell age from multivariate analysis of static and dynamic biomarkers.Signaling network state predicts twist-mediated effects on breast cell migration across diverse growth factor contexts.Capturing cell-fate decisions from the molecular signatures of a receptor-dependent signaling response.Epi-allelic Erk1 and Erk2 knockdown series for quantitative analysis of T cell Erk regulation and IL-2 productionMathematical and computational approaches can complement experimental studies of host-pathogen interactions.PRMT5-Selective Inhibitors Suppress Inflammatory T Cell Responses and Experimental Autoimmune Encephalomyelitis.T cells translate individual, quantal activation into collective, analog cytokine responses via time-integrated feedbacks.Partial Least Squares Regression Models for the Analysis of Kinase Signaling.Effect of multiple genetic polymorphisms on antigen presentation and susceptibility to Mycobacterium tuberculosis infection.Three-kinase inhibitor combination recreates multipathway effects of a geldanamycin analogue on hepatocellular carcinoma cell death.Integration of signals from the B-cell antigen receptor and the IL-4 receptor leads to a cooperative shift in the cellular response axis.Multipathway model enables prediction of kinase inhibitor cross-talk effects on migration of Her2-overexpressing mammary epithelial cells.Parallel multi-time point cell stimulation and lysis on-chip for studying early signaling events in T cell activation.Single-cell quantification of IL-2 response by effector and regulatory T cells reveals critical plasticity in immune response.Pairwise agonist scanning predicts cellular signaling responses to combinatorial stimuli.Multipathway kinase signatures of multipotent stromal cells are predictive for osteogenic differentiation: tissue-specific stem cells.Cytokine-associated drug toxicity in human hepatocytes is associated with signaling network dysregulation.Correlated receptor transport processes buffer single-cell heterogeneity.Diverse continuum of CD4+ T-cell states is determined by hierarchical additive integration of cytokine signals.
P2860
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P2860
Quantitative network signal combinations downstream of TCR activation can predict IL-2 production response.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Quantitative network signal co ...... dict IL-2 production response.
@en
type
label
Quantitative network signal co ...... dict IL-2 production response.
@en
prefLabel
Quantitative network signal co ...... dict IL-2 production response.
@en
P2093
P1476
Quantitative network signal co ...... dict IL-2 production response.
@en
P2093
Christina L Lewis
Lindsay B Nicholson
Lucia Wille
Melissa L Kemp
P304
P356
10.4049/JIMMUNOL.178.8.4984
P407
P577
2007-04-01T00:00:00Z