cAMP signaling selectively influences Ras effectors pathways.
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Molecular mechanisms mediating the G protein-coupled receptor regulation of cell cycle progressionMitochondrial cAMP signalingNeonatal exposure to estradiol/bisphenol A alters promoter methylation and expression of Nsbp1 and Hpcal1 genes and transcriptional programs of Dnmt3a/b and Mbd2/4 in the rat prostate gland throughout lifeProtein kinase A and B-Raf mediate extracellular signal-regulated kinase activation by thyrotropinA Signaling Network of Thyroid-Stimulating HormoneDifferential regulation of vascular smooth muscle and endothelial cell proliferation in vitro and in vivo by cAMP/PKA-activated p85alphaPI3K.TSH signalling and cancer.Prostaglandin E2 induces interleukin-6 expression in human chondrocytes via cAMP/protein kinase A- and phosphatidylinositol 3-kinase-dependent NF-kappaB activation.Shear-induced interleukin-6 synthesis in chondrocytes: roles of E prostanoid (EP) 2 and EP3 in cAMP/protein kinase A- and PI3-K/Akt-dependent NF-kappaB activation.Neutral antibodies to the TSH receptor are present in Graves' disease and regulate selective signaling cascadesThe expression of the thyroid-stimulating hormone (TSH) receptor and the cAMP-dependent protein kinase RII beta regulatory subunit confers TSH-cAMP-dependent growth to mouse fibroblasts.Cellular N-Ras promotes cell survival by downregulation of Jun N-terminal protein kinase and p38Orchestration of multiple arrays of signal cross-talk and combinatorial interactions for maturation and cell death: another vision of t(15;17) preleukemic blast and APL-cell maturation.Antibody-dependent enhancement infection facilitates dengue virus-regulated signaling of IL-10 production in monocytes.By activating matrix metalloproteinase-7, shear stress promotes chondrosarcoma cell motility, invasion and lung colonization.Clinical and molecular genetics of primary pigmented nodular adrenocortical disease.PKA and GAB2 play central roles in the FSH signaling pathway to PI3K and AKT in ovarian granulosa cells.Insulin Receptor Substrate 1, the Hub Linking Follicle-stimulating Hormone to Phosphatidylinositol 3-Kinase ActivationG protein-coupled receptor-induced Akt activity in cellular proliferation and apoptosis.Modulation of sodium iodide symporter expression and function by LY294002, Akti-1/2 and Rapamycin in thyroid cellsNitric oxide and ERK/MAPK mediation of estrous behavior induced by GnRH, PGE2 and db-cAMP in ratsSomatostatin activates Ras and ERK1/2 via a G protein βγ-subunit-initiated pathway in thyroid cells.Multifaceted Breast Cancer: The Molecular Connection With Obesity.Activation of STAT3 by G alpha(s) distinctively requires protein kinase A, JNK, and phosphatidylinositol 3-kinase.PKA and Apicomplexan Parasite Diseases.G protein-coupled receptors (GPCRs) That Signal via Protein Kinase A (PKA) Cross-talk at Insulin Receptor Substrate 1 (IRS1) to Activate the phosphatidylinositol 3-kinase (PI3K)/AKT Pathway.Differential involvement of the Ras and Rap1 small GTPases in vasoactive intestinal and pituitary adenylyl cyclase activating polypeptides control of the prolactin gene.A-Kinase Anchoring Protein 4 (AKAP4) is an ERK1/2 substrate and a switch molecule between cAMP/PKA and PKC/ERK1/2 in human spermatozoa.Cell penetrating peptides to dissect host-pathogen protein-protein interactions in Theileria-transformed leukocytes.Growth hormone and insulin-like growth factor 1 affect the severity of Graves' disease.The cAMP-activated GTP exchange factor, Epac1 upregulates plasma membrane and nuclear Akt kinase activities in 8-CPT-2-O-Me-cAMP-stimulated macrophages: Gene silencing of the cAMP-activated GTP exchange Epac1 prevents 8-CPT-2-O-Me-cAMP activation ofInvolvement of cAMP response element-binding protein in the regulation of cell proliferation and the prolactin promoter of lactotrophs in primary culture.p85 regulatory subunit of PI3K mediates cAMP-PKA and estrogens biological effects on growth and survival.Activation of PI3K-Akt pathway mediates antiapoptotic effects of beta-adrenergic agonist in airway eosinophils.Coordinate regulation of forskolin-induced cellular proliferation in macrophages by protein kinase A/cAMP-response element-binding protein (CREB) and Epac1-Rap1 signaling: effects of silencing CREB gene expression on Akt activation.KRAS(G12D)-mediated oncogenic transformation of thyroid follicular cells requires long-term TSH stimulation and is regulated by SPRY1.The p85 regulatory subunit of PI3K mediates cAMP-PKA and insulin biological effects on MCF-7 cell growth and motility.
P2860
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P2860
cAMP signaling selectively influences Ras effectors pathways.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
cAMP signaling selectively influences Ras effectors pathways.
@en
type
label
cAMP signaling selectively influences Ras effectors pathways.
@en
prefLabel
cAMP signaling selectively influences Ras effectors pathways.
@en
P2093
P2860
P356
P1433
P1476
cAMP signaling selectively influences Ras effectors pathways.
@en
P2093
Avvedimento EV
Diez-Roux G
P2860
P2888
P304
P356
10.1038/SJ.ONC.1204219
P407
P577
2001-03-01T00:00:00Z
P5875
P6179
1011931204