The Rheb-mTOR pathway is upregulated in reactive astrocytes of the injured spinal cord. - Wikidata - wikimedia - TriplyDB

The Rheb-mTOR pathway is upregulated in reactive astrocytes of the injured spinal cord.

The Rheb-mTOR pathway is upregulated in reactive astrocytes of the injured spinal cord.

http://www.wikidata.org/entity/Q41957358

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Molecular dissection of reactive astrogliosis and glial scar formation Shedding new light on neurodegenerative diseases through the mammalian target of rapamycin Systemic bisperoxovanadium activates Akt/mTOR, reduces autophagy, and enhances recovery following cervical spinal cord injury Spatiotemporal characterization of mTOR kinase activity following kainic acid induced status epilepticus and analysis of rat brain response to chronic rapamycin treatment FOXO3a/p27kip1 expression and essential role after acute spinal cord injury in adult rat.Mapping the spatio-temporal pattern of the mammalian target of rapamycin (mTOR) activation in temporal lobe epilepsy Inhibition of EGFR/MAPK signaling reduces microglial inflammatory response and the associated secondary damage in rats after spinal cord injury Upregulation of glutamate transporter GLT-1 by mTOR-Akt-NF-кB cascade in astrocytic oxygen-glucose deprivation Phosphatidylinositol 3-kinase/protein kinase Cdelta activation induces close homolog of adhesion molecule L1 (CHL1) expression in cultured astrocytes.Glioblastoma subclasses can be defined by activity among signal transduction pathways and associated genomic alterations.Phosphatidic acid is a leukocyte chemoattractant that acts through S6 kinase signaling mTOR signaling inhibition modulates macrophage/microglia-mediated neuroinflammation and secondary injury via regulatory T cells after focal ischemia.The effect of systemic PTEN antagonist peptides on axon growth and functional recovery after spinal cord injury.Suppression of stretch reflex activity after spinal or systemic treatment with AMPA receptor antagonist NGX424 in rats with developed baclofen tolerance.Mammalian target of rapamycin signaling in the spinal cord is required for neuronal plasticity and behavioral hypersensitivity associated with neuropathy in the rat.Spinal phosphinositide 3-kinase-Akt-mammalian target of rapamycin signaling cascades in inflammation-induced hyperalgesia Frontier of epilepsy research - mTOR signaling pathway.Spatial and cellular characterization of mTORC1 activation after spinal cord injury reveals biphasic increase mainly attributed to microglia/macrophages.Mammalian target of rapamycin: hitting the bull's-eye for neurological disorders.Neurofibromatosis-1 regulates mTOR-mediated astrocyte growth and glioma formation in a TSC/Rheb-independent manner.Enriched Environment Protects the Optic Nerve from Early Diabetes-Induced Damage in Adult Rats.Deconvoluting mTOR biology Astrogliosis Induced by Brain Injury Is Regulated by Sema4B Phosphorylation(123)Nuclear translocation of PKM2 modulates astrocyte proliferation via p27 and -catenin pathway after spinal cord injury The role of mTOR signaling pathway in spinal cord injury.Modeling Alexander disease with patient iPSCs reveals cellular and molecular pathology of astrocytes.Activation of mammalian target of rapamycin mediates rat pain-related responses induced by BmK I, a sodium channel-specific modulator.Involvement of p38 MAPK in reactive astrogliosis induced by ischemic stroke.LIN28 expression in rat spinal cord after injury.The stem cell potential of glia: lessons from reactive gliosis.Astrocytes in the tempest of multiple sclerosis.PI3 Kinase regulation of neural regeneration and muscle hypertrophy after spinal cord injury.mTOR kinase, a key player in the regulation of glial functions: relevance for the therapy of multiple sclerosis.Regulation and dysregulation of astrocyte activation and implications in tumor formation.The glial scar in spinal cord injury and repair.Spinal cord contusion.Identifying the role of microRNAs in spinal cord injury.Leigh syndrome: neuropathology and pathogenesis.Astrocytes reassessment - an evolving concept part one: embryology, biology, morphology and reactivity.mTOR in Brain Physiology and Pathologies.

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P2860

The Rheb-mTOR pathway is upregulated in reactive astrocytes of the injured spinal cord.

http://www.wikidata.org/entity/Q41957358

description

2009 nî lūn-bûn

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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2009年论文

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2009年论文

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name

The Rheb-mTOR pathway is upregulated in reactive astrocytes of the injured spinal cord.

@en

type

label

The Rheb-mTOR pathway is upregulated in reactive astrocytes of the injured spinal cord.

@en

prefLabel

The Rheb-mTOR pathway is upregulated in reactive astrocytes of the injured spinal cord.

@en

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JNEUROSCI.4103-08.2009

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P1476

The Rheb-mTOR pathway is upregulated in reactive astrocytes of the injured spinal cord

@en

P2093

Elena B Pasquale

http://www.w3.org/2001/XMLSchema#string

Fatima Valencia

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Martin Marsala

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Masakatsu Oshiro

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Michael P Hefferan

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Morgan D Silldorff

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P2860

PI3K induced actin filament remodeling through Akt and p70S6K1: implication of essential role in cell migration

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

Rictor, a novel binding partner of mTOR, defines a rapamycin-insensitive and raptor-independent pathway that regulates the cytoskeleton

Mammalian TOR complex 2 controls the actin cytoskeleton and is rapamycin insensitive

Promoting axon regeneration in the adult CNS by modulation of the PTEN/mTOR pathway

Glial inhibition of CNS axon regeneration

The selectivity of protein kinase inhibitors: a further update

AKT/PKB signaling: navigating downstream

Quantitative biomarker analysis of synovial gene expression by real-time PCR

TOR signaling in growth and metabolism

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Simone Codeluppi

Camilla I Svensson

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