Combined therapies that induce senescence and stabilize p53 block melanoma growth and prompt antitumor immune responses.

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Connecting the Dots: Therapy-Induced Senescence and a Tumor-Suppressive Immune Microenvironment.MDM2 Antagonists Counteract Drug-Induced DNA Damage.Antitumorigenic effect of damnacanthal on melanoma cell viability through p53 and NF-κB/caspase-3 signaling pathways

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Combined therapies that induce senescence and stabilize p53 block melanoma growth and prompt antitumor immune responses.

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2015 nî lūn-bûn

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Combined therapies that induce ...... pt antitumor immune responses.

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Ann Richmond

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Anna Vilgelm

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Mdm2 and aurora kinase a inhibitors synergize to block melanoma growth by driving apoptosis and immune clearance of tumor cells

Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas

Restoration of p53 function leads to tumour regression in vivo

Four faces of cellular senescence.

Focus on TILs: prognostic significance of tumor infiltrating lymphocytes in human melanoma

Reversal of human cellular senescence: roles of the p53 and p16 pathways.

Targeting aurora kinases limits tumour growth through DNA damage-mediated senescence and blockade of NF-κB impairs this drug-induced senescence.

Senescence at a glance

Polyploidy road to therapy-induced cellular senescence and escape.

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10.1080/2162402X.2015.1009299

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Combined therapies that induce senescence and stabilize p53 block melanoma growth and prompt antitumor immune responses.

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P2860

P2860

Combined therapies that induce senescence and stabilize p53 block melanoma growth and prompt antitumor immune responses.

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