Resveratrol blocks diabetes-induced early vascular lesions and vascular endothelial growth factor induction in mouse retinas. - Wikidata - wikimedia - TriplyDB

Resveratrol blocks diabetes-induced early vascular lesions and vascular endothelial growth factor induction in mouse retinas.

Resveratrol blocks diabetes-induced early vascular lesions and vascular endothelial growth factor induction in mouse retinas.

http://www.wikidata.org/entity/Q43442634

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Resveratrol and Ophthalmic Diseases Diabetes and retinal vascular dysfunction.The sustained delivery of resveratrol or a defined grape powder inhibits new blood vessel formation in a mouse model of choroidal neovascularization.Resveratrol ameliorates high glucose and high-fat/sucrose diet-induced vascular hyperpermeability involving Cav-1/eNOS regulation Modifying Choroidal Neovascularization Development with a Nutritional Supplement in Mice Resveratrol Inhibits Hypoxia-Induced Vascular Endothelial Growth Factor Expression and Pathological Neovascularization.Resveratrol potentiates rapamycin to prevent hyperinsulinemia and obesity in male mice on high fat diet.Resveratrol ameliorates high-glucose-induced hyperpermeability mediated by caveolae via VEGF/KDR pathway Neurovascular dysfunction and neurodegeneration in dementia and Alzheimer's disease Botanical compounds: effects on major eye diseases p53 coordinates decidual sestrin 2/AMPK/mTORC1 signaling to govern parturition timing.Animal models of diabetic retinopathy: summary and comparison.Antidiabetic Effects of Resveratrol: The Way Forward in Its Clinical Utility.Resveratrol and the eye: activity and molecular mechanisms.Ocular inflammation and endoplasmic reticulum stress are attenuated by supplementation with grape polyphenols in human retinal pigmented epithelium cells and in C57BL/6 mice.Current treatment strategies and nanocarrier based approaches for the treatment and management of diabetic retinopathy.Increased O-GlcNAcylation of NF-κB Enhances Retinal Ganglion Cell Death in Streptozotocin-induced Diabetic Retinopathy.Aralia elata inhibits neurodegeneration by downregulating O-GlcNAcylation of NF-κB in diabetic mice.Effect of polyphenolic phytochemicals on ectopic oxidative phosphorylation in rod outer segments of bovine retina.Effect of Resveratrol-Based Nutritional Supplement on Choroidal Thickness: A Pilot Study.miRNA15a regulates insulin signal transduction in the retinal vasculature.Nrf2 as molecular target for polyphenols: A novel therapeutic strategy in diabetic retinopathy.[Effect of resveratrol on the fundus oculi. An overview].

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Resveratrol blocks diabetes-induced early vascular lesions and vascular endothelial growth factor induction in mouse retinas.

http://www.wikidata.org/entity/Q43442634

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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2011年學術文章

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name

Resveratrol blocks diabetes-in ...... or induction in mouse retinas.

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Resveratrol blocks diabetes-in ...... or induction in mouse retinas.

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type

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Resveratrol blocks diabetes-in ...... or induction in mouse retinas.

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Resveratrol blocks diabetes-in ...... or induction in mouse retinas.

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Resveratrol blocks diabetes-in ...... or induction in mouse retinas.

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J.1755-3768.2011.02243.X

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Acta Ophthalmologica

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Resveratrol blocks diabetes-in ...... or induction in mouse retinas.

@en

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Gu Seob Roh

http://www.w3.org/2001/XMLSchema#string

Gyeong Jae Cho

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Wan Sung Choi

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Yoon Sook Kim

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Young Hee Kim

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P2860

Small molecule activators of SIRT1 as therapeutics for the treatment of type 2 diabetes

Cancer chemopreventive activity of resveratrol, a natural product derived from grapes

Protein kinase C-delta mediates neuronal apoptosis in the retinas of diabetic rats via the Akt signaling pathway

Inducible nitric oxide synthase isoform is a key mediator of leukostasis and blood-retinal barrier breakdown in diabetic retinopathy.

Resveratrol prolongs lifespan and retards the onset of age-related markers in a short-lived vertebrate.

Resveratrol: a multitargeted agent for age-associated chronic diseases.

Microvascular basement membranes in diabetes mellitus.

Experimental diabetes causes breakdown of the blood-retina barrier by a mechanism involving tyrosine nitration and increases in expression of vascular endothelial growth factor and urokinase plasminogen activator receptor.

SIRT1 activation confers neuroprotection in experimental optic neuritis

Vascular dysfunction in aging: potential effects of resveratrol, an anti-inflammatory phytoestrogen.

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10.1111/J.1755-3768.2011.02243.X

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21914146

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