Capacitative calcium entry contributes to nuclear factor of activated T-cells nuclear translocation and hypertrophy in cardiomyocytes.
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Plasma membrane Ca2+-ATPase isoform 4 antagonizes cardiac hypertrophy in association with calcineurin inhibition in rodentsThe DnaJ-related factor Mrj interacts with nuclear factor of activated T cells c3 and mediates transcriptional repression through class II histone deacetylase recruitmentKaposi's sarcoma-associated herpesvirus mitochondrial K7 protein targets a cellular calcium-modulating cyclophilin ligand to modulate intracellular calcium concentration and inhibit apoptosisGlucosamine inhibits angiotensin II-induced cytoplasmic Ca2+ elevation in neonatal cardiomyocytes via protein-associated O-linked N-acetylglucosamineGlucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associated O-GlcNAcAlpha B-crystallin suppresses pressure overload cardiac hypertrophyReceptor subtype involved in alpha 1-adrenergic receptor-mediated Ca2+ signaling in cardiomyocytesGlucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signalingTRPC3 and TRPC6 are essential for angiotensin II-induced cardiac hypertrophy.Methods in cardiomyocyte isolation, culture, and gene transfer.TrpC3 regulates hypertrophy-associated gene expression without affecting myocyte beating or cell size.Alpha(1D)-adrenergic receptor insensitivity is associated with alterations in its expression and distribution in cultured vascular myocytesGlucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein O-GlcNAc and increased mitochondrial Bcl-2Gastrodin Inhibits Store-Operated Ca2+ Entry and Alleviates Cardiac Hypertrophy.Overexpression of TRPC3 increases apoptosis but not necrosis in response to ischemia-reperfusion in adult mouse cardiomyocytes.Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.Proteasome functional insufficiency activates the calcineurin-NFAT pathway in cardiomyocytes and promotes maladaptive remodelling of stressed mouse hearts.Block of TRPC5 channels by 2-aminoethoxydiphenyl borate: a differential, extracellular and voltage-dependent effect.Activation of the hexosamine biosynthesis pathway and protein O-GlcNAcylation modulate hypertrophic and cell signaling pathways in cardiomyocytes from diabetic miceProtein O-GlcNAcylation and cardiovascular (patho)physiology.STIM1-dependent store-operated Ca²⁺ entry is required for pathological cardiac hypertrophy.The Ca(2+)-activated cation channel TRPM4 is a negative regulator of angiotensin II-induced cardiac hypertrophy.STIM1 enhances SR Ca2+ content through binding phospholamban in rat ventricular myocytes.Critical role for stromal interaction molecule 1 in cardiac hypertrophyGlucose deprivation-induced increase in protein O-GlcNAcylation in cardiomyocytes is calcium-dependentModification of STIM1 by O-linked N-acetylglucosamine (O-GlcNAc) attenuates store-operated calcium entry in neonatal cardiomyocytesDichotomy of Ca2+ in the heart: contraction versus intracellular signaling.Post-translational protein modification by O-linked N-acetyl-glucosamine: its role in mediating the adverse effects of diabetes on the heart.Nuclear Ca2+ sparks and waves mediated by inositol 1,4,5-trisphosphate receptors in neonatal rat cardiomyocytes.Role of protein O-linked N-acetyl-glucosamine in mediating cell function and survival in the cardiovascular system.STIM1-dependent Ca(2+) microdomains are required for myofilament remodeling and signaling in the heart.Protein O-GlcNAcylation: a new signaling paradigm for the cardiovascular system.Effects of thapsigargin and phenylephrine on calcineurin and protein kinase C signaling functions in cardiac myocytes.Calcineurin-dependent cardiomyopathy is activated by TRPC in the adult mouse heartSTIM1/Orai1-mediated SOCE: current perspectives and potential roles in cardiac function and pathologyThe role of protein O-linked beta-N-acetylglucosamine in mediating cardiac stress responses.Sarco(endo)plasmic reticulum Ca2+ ATPase (SERCA) gene silencing and remodeling of the Ca2+ signaling mechanism in cardiac myocytes.Stromal interaction molecule 1 is essential for normal cardiac homeostasis through modulation of ER and mitochondrial function.Calcium signaling dysfunction in heart disease.Emerging roles for native Orai Ca2+ channels in cardiovascular disease
P2860
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P2860
Capacitative calcium entry contributes to nuclear factor of activated T-cells nuclear translocation and hypertrophy in cardiomyocytes.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
Capacitative calcium entry con ...... hypertrophy in cardiomyocytes.
@en
Capacitative calcium entry con ...... hypertrophy in cardiomyocytes.
@nl
type
label
Capacitative calcium entry con ...... hypertrophy in cardiomyocytes.
@en
Capacitative calcium entry con ...... hypertrophy in cardiomyocytes.
@nl
prefLabel
Capacitative calcium entry con ...... hypertrophy in cardiomyocytes.
@en
Capacitative calcium entry con ...... hypertrophy in cardiomyocytes.
@nl
P2093
P356
P1476
Capacitative calcium entry con ...... hypertrophy in cardiomyocytes.
@en
P2093
Dacia L Hunton
Louis J Dell'Italia
Pamela A Lucchesi
Richard B Marchase
Xiaogang Cheng
P304
14266-14273
P356
10.1074/JBC.M107167200
P407
P577
2002-02-04T00:00:00Z