Doxycycline inducible expression of SERCA2a improves calcium handling and reverts cardiac dysfunction in pressure overload-induced cardiac hypertrophy.
about
Human delta opioid receptor biogenesis is regulated via interactions with SERCA2b and calnexinTreatment of heart failure by a methanocarba derivative of adenosine monophosphate: implication for a role of cardiac purinergic P2X receptors.cAMP- and Ca²(+) /calmodulin-dependent protein kinases mediate inotropic, lusitropic and arrhythmogenic effects of urocortin 2 in mouse ventricular myocytesNeonatal gene transfer of Serca2a delays onset of hypertrophic remodeling and improves function in familial hypertrophic cardiomyopathy.Enhanced L-type calcium currents in cardiomyocytes from transgenic rats overexpressing SERCA2a.A novel steroid-like compound F90927 exerting positive-inotropic effects in cardiac muscle.Celecoxib prevents pressure overload-induced cardiac hypertrophy and dysfunction by inhibiting inflammation, apoptosis and oxidative stress.Hypoxic response elements and Tet-On advanced double-controlled systems regulate hVEGF 165 and angiopoietin-1 gene expression in vitroSERCA2a upregulation ameliorates cellular alternans induced by metabolic inhibition.Pim-1 kinase antagonizes aspects of myocardial hypertrophy and compensation to pathological pressure overload.Conditional increase in SERCA2a protein is able to reverse contractile dysfunction and abnormal calcium flux in established diabetic cardiomyopathy.Thyroid hormone receptor-beta is associated with coronary angiogenesis during pathological cardiac hypertrophy.Neuronal Na+ Channels Are Integral Components of Pro-arrhythmic Na+/Ca2+ Signaling Nanodomain That Promotes Cardiac Arrhythmias During β-adrenergic Stimulation.Pim-1 regulates cardiomyocyte survival downstream of Akt.Increased expression of SERCA in the hearts of transgenic mice results in increased oxidation of glucose.Chronic doxycycline exposure accelerates left ventricular hypertrophy and progression to heart failure in mice after thoracic aorta constriction.2-hydroxyoleic acid affects cardiomyocyte [Ca2+]i transient and contractility in a region-dependent manner.Impaired left ventricular mechanical and energetic function in mice after cardiomyocyte-specific excision of Serca2.
P2860
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P2860
Doxycycline inducible expression of SERCA2a improves calcium handling and reverts cardiac dysfunction in pressure overload-induced cardiac hypertrophy.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Doxycycline inducible expressi ...... d-induced cardiac hypertrophy.
@en
Doxycycline inducible expressi ...... d-induced cardiac hypertrophy.
@nl
type
label
Doxycycline inducible expressi ...... d-induced cardiac hypertrophy.
@en
Doxycycline inducible expressi ...... d-induced cardiac hypertrophy.
@nl
prefLabel
Doxycycline inducible expressi ...... d-induced cardiac hypertrophy.
@en
Doxycycline inducible expressi ...... d-induced cardiac hypertrophy.
@nl
P2093
P2860
P50
P1476
Doxycycline inducible expressi ...... d-induced cardiac hypertrophy.
@en
P2093
Bernd Gloss
Brian Scott
Darrell D Belke
Jorge Suarez
Wolfgang H Dillmann
Yun-Kyung Kim
P2860
P304
P356
10.1152/AJPHEART.00428.2004
P577
2004-07-15T00:00:00Z