about
P184
Insulin-like growth factors and their potential role in cardiac epigeneticsDisruption of integrin function in the murine myocardium leads to perinatal lethality, fibrosis, and abnormal cardiac performanceCardiac expression of a gain-of-function alpha(5)-integrin results in perinatal lethality.Subunit 3 of the COP9 signalosome is poised to facilitate communication between the extracellular matrix and the nucleus through the muscle-specific beta1D integrin.Cell cycle re-entry and mitochondrial defects in myc-mediated hypertrophic cardiomyopathy and heart failure.Influence of the extracellular matrix and integrins on volume-sensitive osmolyte anion channels in C2C12 myoblasts.Cardiac-specific, inducible ClC-3 gene deletion eliminates native volume-sensitive chloride channels and produces myocardial hypertrophy in adult miceMineralocorticoid accelerates transition to heart failure with preserved ejection fraction via "nongenomic effects".Expression profile and protein translation of TMEM16A in murine smooth muscle.Orai1 interacts with STIM1 and mediates capacitative Ca2+ entry in mouse pulmonary arterial smooth muscle cells.A role for nucleotides in support of breast cancer angiogenesis: heterologous receptor signalling.Knockdown of subunit 3 of the COP9 signalosome inhibits C2C12 myoblast differentiation via NF-KappaB signaling pathwayIncreased TMEM16A-encoded calcium-activated chloride channel activity is associated with pulmonary hypertension.Complex phosphatase regulation of Ca2+-activated Cl- currents in pulmonary arterial smooth muscle cells.Inhibition of Aβ42 oligomerization in yeast by a PICALM ortholog and certain FDA approved drugs.Cardiac-specific attenuation of natriuretic peptide A receptor activity accentuates adverse cardiac remodeling and mortality in response to pressure overload.Codon optimization markedly improves doxycycline regulated gene expression in the mouse heart.Doxycycline inducible expression of SERCA2a improves calcium handling and reverts cardiac dysfunction in pressure overload-induced cardiac hypertrophy.A lethal perinatal cardiac phenotype resulting from altered integrin function in cardiomyocytes.Cardiac-specific induction of the transcriptional coactivator peroxisome proliferator-activated receptor gamma coactivator-1alpha promotes mitochondrial biogenesis and reversible cardiomyopathy in a developmental stage-dependent manner.Effect of fiber orientation on propagation: electrical mapping of genetically altered mouse hearts.Integrin activation in the heart: a link between electrical and contractile dysfunction?
P50
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P50
description
moleculair bioloog
@nl
molecular biology researcher
@en
name
Maria L. Valencik
@ast
Maria L. Valencik
@en
Maria L. Valencik
@es
Maria L. Valencik
@nl
type
label
Maria L. Valencik
@ast
Maria L. Valencik
@en
Maria L. Valencik
@es
Maria L. Valencik
@nl
altLabel
Maria L Valencik
@en
Maria Valencik
@en
prefLabel
Maria L. Valencik
@ast
Maria L. Valencik
@en
Maria L. Valencik
@es
Maria L. Valencik
@nl
P1153
6602521157