Roles of activated Src and Stat3 signaling in melanoma tumor cell growth. - Wikidata - wikimedia - TriplyDB

Roles of activated Src and Stat3 signaling in melanoma tumor cell growth.

Roles of activated Src and Stat3 signaling in melanoma tumor cell growth.

http://www.wikidata.org/entity/Q44172485

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Preadministration of High-Dose Salicylates, Suppressors of NF- B Activation, May Increase the Chemosensitivity of Many Cancers: An Example of Proapoptotic Signal Modulation Therapy Persistently activated Stat3 maintains constitutive NF-kappaB activity in tumors.Cucurbitacins - a promising target for cancer therapy Innate and adaptive immune cells in the tumor microenvironment STAT3 as a therapeutic target in head and neck cancer Nuclear termination of STAT3 signaling through SIPAR (STAT3-Interacting Protein As a Repressor)-dependent recruitment of T cell tyrosine phosphatase TC-PTP Brevilin A, a novel natural product, inhibits janus kinase activity and blocks STAT3 signaling in cancer cells Structurally modified curcumin analogs inhibit STAT3 phosphorylation and promote apoptosis of human renal cell carcinoma and melanoma cell lines.Preclinical characterization of signal transducer and activator of transcription 3 small molecule inhibitors for primary and metastatic brain cancer therapy The JAK2 inhibitor AZD1480 potently blocks Stat3 signaling and oncogenesis in solid tumors.Role of NADPH oxidase and Stat3 in statin-mediated protection against diabetic retinopathy.Indirubin derivatives inhibit Stat3 signaling and induce apoptosis in human cancer cells.Alteration of SHP-1/p-STAT3 Signaling: A Potential Target for Anticancer Therapy.Implications of an antiparallel dimeric structure of nonphosphorylated STAT1 for the activation-inactivation cycle.The small molecule curcumin analog FLLL32 induces apoptosis in melanoma cells via STAT3 inhibition and retains the cellular response to cytokines with anti-tumor activity Interleukin-6 in bone metastasis and cancer progression.Reciprocal regulation of c-Src and STAT3 in non-small cell lung cancer.Elevated c-Src and c-Yes expression in malignant skin cancers Glioblastoma cancer-initiating cells inhibit T-cell proliferation and effector responses by the signal transducers and activators of transcription 3 pathway Short hairpin RNA-mediated fibronectin knockdown delays tumor growth in a mouse glioma model.Critical role of STAT3 in melanoma metastasis through anoikis resistance Apoptosis-associated tyrosine kinase 1 inhibits growth and migration and promotes apoptosis in melanoma.Leelamine mediates cancer cell death through inhibition of intracellular cholesterol transport.STAT3 induces anoikis resistance, promotes cell invasion and metastatic potential in pancreatic cancer cells.(-)-Epigallocatechin-3-gallate inhibits VEGF expression induced by IL-6 via Stat3 in gastric cancer.Benzoxathiol derivative BOT-4-one suppresses L540 lymphoma cell survival and proliferation via inhibition of JAK3/STAT3 signaling.STAT3 Knockdown in B16 Melanoma by siRNA Lipopolyplexes Induces Bystander Immune Response In Vitro and In Vivo A phase 2 trial of dasatinib in advanced melanoma.2-Methoxystypandrone inhibits signal transducer and activator of transcription 3 and nuclear factor-κB signaling by inhibiting Janus kinase 2 and IκB kinase Phosphoproteomic screen identifies potential therapeutic targets in melanoma.Icaritin inhibits JAK/STAT3 signaling and growth of renal cell carcinoma.Gambogic acid inhibits STAT3 phosphorylation through activation of protein tyrosine phosphatase SHP-1: potential role in proliferation and apoptosis.Breast cancer cells condition lymphatic endothelial cells within pre-metastatic niches to promote metastasis Expression and functional role of CRIPTO-1 in cutaneous melanoma.Fad104, a positive regulator of adipocyte differentiation, suppresses invasion and metastasis of melanoma cells by inhibition of STAT3 activity.Expression of p-STAT3 in human colorectal adenocarcinoma and adenoma; correlation with clinicopathological factors Phase I clinical trial of the Src inhibitor dasatinib with dacarbazine in metastatic melanoma B7-H3 silencing increases paclitaxel sensitivity by abrogating Jak2/Stat3 phosphorylation.Down-regulation of STAT3 expression using vector-based RNA interference promotes apoptosis in Hepatocarcinoma cells.Comparisons of the efficacy of a Jak1/2 inhibitor (AZD1480) with a VEGF signaling inhibitor (cediranib) and sham treatments in mouse tumors using DCE-MRI, DW-MRI, and histology.

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Roles of activated Src and Stat3 signaling in melanoma tumor cell growth.

http://www.wikidata.org/entity/Q44172485

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name

Roles of activated Src and Stat3 signaling in melanoma tumor cell growth.

@en

Roles of activated Src and Stat3 signaling in melanoma tumor cell growth.

@nl

type

label

Roles of activated Src and Stat3 signaling in melanoma tumor cell growth.

@en

Roles of activated Src and Stat3 signaling in melanoma tumor cell growth.

@nl

prefLabel

Roles of activated Src and Stat3 signaling in melanoma tumor cell growth.

@en

Roles of activated Src and Stat3 signaling in melanoma tumor cell growth.

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Roles of activated Src and Stat3 signaling in melanoma tumor cell growth.

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Adil Daud

http://www.w3.org/2001/XMLSchema#string

Alan Kraker

http://www.w3.org/2001/XMLSchema#string

Alfred Chang

http://www.w3.org/2001/XMLSchema#string

Douglas Reintgen

http://www.w3.org/2001/XMLSchema#string

Guilian Niu

http://www.w3.org/2001/XMLSchema#string

Hua Yu

http://www.w3.org/2001/XMLSchema#string

Mei Huang

http://www.w3.org/2001/XMLSchema#string

Richard Jove

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Steve Shivers

http://www.w3.org/2001/XMLSchema#string

Tammy Bowman

http://www.w3.org/2001/XMLSchema#string

P2860

Inhibition of STAT3 signaling leads to apoptosis of leukemic large granular lymphocytes and decreased Mcl-1 expression

Phosphotyrosyl peptides block Stat3-mediated DNA binding activity, gene regulation, and cell transformation

Stat3 as an oncogene

STATs in oncogenesis

Inhibition of the epidermal growth factor receptor family of tyrosine kinases as an approach to cancer chemotherapy: progression from reversible to irreversible inhibitors.

Protein tyrosine kinase inhibitors as novel therapeutic agents.

STAT proteins as novel targets for cancer therapy. Signal transducer an activator of transcription.

Stat3 activation is required for cellular transformation by v-src.

The role of STATs in transcriptional control and their impact on cellular function.

STAT signaling in head and neck cancer.

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10.1038/SJ.ONC.1205859

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