Epsilon protein kinase C mediated ischemic tolerance requires activation of the extracellular regulated kinase pathway in the organotypic hippocampal slice. - Wikidata - wikimedia - TriplyDB

Epsilon protein kinase C mediated ischemic tolerance requires activation of the extracellular regulated kinase pathway in the organotypic hippocampal slice.

Epsilon protein kinase C mediated ischemic tolerance requires activation of the extracellular regulated kinase pathway in the organotypic hippocampal slice.

http://www.wikidata.org/entity/Q44924589

about

Novel mitochondrial targets for neuroprotection Ischemic conditioning-induced endogenous brain protection: Applications pre-, per- or post-stroke Essential role of the redox-sensitive kinase p66shc in determining energetic and oxidative status and cell fate in neuronal preconditioning Ischemic preconditioning and clinical scenarios.Preconditioning-induced ischemic tolerance: a window into endogenous gearing for cerebroprotection Ischemic preconditioning mediates cyclooxygenase-2 expression via nuclear factor-kappa B activation in mixed cortical neuronal cultures.Post-ischemic activation of protein kinase C ε protects the hippocampus from cerebral ischemic injury via alterations in cerebral blood flow.Poststroke neuronal rescue and synaptogenesis mediated in vivo by protein kinase C in adult brains Pathways for ischemic cytoprotection: role of sirtuins in caloric restriction, resveratrol, and ischemic preconditioning.Signal transducers and activators of transcription: STATs-mediated mitochondrial neuroprotection Increased BDNF protein expression after ischemic or PKC epsilon preconditioning promotes electrophysiologic changes that lead to neuroprotection.Neuroprotection: lessons from hibernators.Ischemic preconditioning via epsilon protein kinase C activation requires cyclooxygenase-2 activation in vitro Redox signaling pathways involved in neuronal ischemic preconditioning Beyond anoxia: the physiology of metabolic downregulation and recovery in the anoxia-tolerant turtle.Preconditioning mediated by sublethal oxygen-glucose deprivation-induced cyclooxygenase-2 expression via the signal transducers and activators of transcription 3 phosphorylation.Cellular and molecular neurobiology of brain preconditioning.Ischemic preconditioning targets the respiration of synaptic mitochondria via protein kinase C epsilon.Inositol 1,4,5-triphosphate receptors and NAD(P)H mediate Ca2+ signaling required for hypoxic preconditioning of hippocampal neurons.Protein kinase Cepsilon: function in neurons.GABA synapses mediate neuroprotection after ischemic and epsilonPKC preconditioning in rat hippocampal slice cultures.Ischemic tolerance as an active and intrinsic neuroprotective mechanism.Post-ischemic brain damage: effect of ischemic preconditioning and postconditioning and identification of potential candidates for stroke therapy.Protein kinase C epsilon activation delays neuronal depolarization during cardiac arrest in the euthermic arctic ground squirrel.Protein kinase C epsilon delays latency until anoxic depolarization through arc expression and GluR2 internalization.epsilonPKC confers acute tolerance to cerebral ischemic reperfusion injury.Green tea polyphenols precondition against cell death induced by oxygen-glucose deprivation via stimulation of laminin receptor, generation of reactive oxygen species, and activation of protein kinase Cε.Selective activation of protein kinase C∊ in mitochondria is neuroprotective in vitro and reduces focal ischemic brain injury in mice.Oxygen-conserving reflexes of the brain: the current molecular knowledge.epsilonPKC phosphorylates the mitochondrial K(+) (ATP) channel during induction of ischemic preconditioning in the rat hippocampus.μ-Opioid receptor attenuates Aβ oligomers-induced neurotoxicity through mTOR signaling.Protocatechualdehyde Protects Against Cerebral Ischemia-Reperfusion-Induced Oxidative Injury Via Protein Kinase Cε/Nrf2/HO-1 Pathway.Resveratrol mimics ischemic preconditioning in the brain.Extracellular signal-regulated kinase1/2-dependent changes in tight junctions after ischemic preconditioning contributes to tolerance induction after ischemic stroke.Metabolomics Based Identification of SIRT5 and Protein Kinase C Epsilon Regulated Pathways in Brain.Nicotine Alters Estrogen Receptor-Beta-Regulated Inflammasome Activity and Exacerbates Ischemic Brain Damage in Female Rats.NO-induced neuroprotection in ischemic preconditioning stimulates mitochondrial Mn-SOD activity and expression via RAS/ERK1/2 pathway

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P2860

Epsilon protein kinase C mediated ischemic tolerance requires activation of the extracellular regulated kinase pathway in the organotypic hippocampal slice.

http://www.wikidata.org/entity/Q44924589

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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name

Epsilon protein kinase C media ...... organotypic hippocampal slice.

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Epsilon protein kinase C media ...... organotypic hippocampal slice.

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type

label

Epsilon protein kinase C media ...... organotypic hippocampal slice.

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Epsilon protein kinase C media ...... organotypic hippocampal slice.

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prefLabel

Epsilon protein kinase C media ...... organotypic hippocampal slice.

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Epsilon protein kinase C media ...... organotypic hippocampal slice.

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Journal of Cerebral Blood Flow & Metabolism

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Epsilon protein kinase C media ...... organotypic hippocampal slice.

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Ami P Raval

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Christian Lange-Asschenfeldt

http://www.w3.org/2001/XMLSchema#string

Kunjan R Dave

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Miguel A Pérez-Pinzón

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Thomas J Sick

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P2860

Regulation of bad phosphorylation and association with Bcl-x(L) by the MAPK/Erk kinase

Sustained in vivo cardiac protection by a rationally designed peptide that causes epsilon protein kinase C translocation

Mitochondrial PKCepsilon and MAPK form signaling modules in the murine heart: enhanced mitochondrial PKCepsilon-MAPK interactions and differential MAPK activation in PKCepsilon-induced cardioprotection

A selective epsilon-protein kinase C antagonist inhibits protection of cardiac myocytes from hypoxia-induced cell death

Localization of protein kinases by anchoring proteins: a theme in signal transduction

A synthetic inhibitor of the mitogen-activated protein kinase cascade

Preconditioning with cortical spreading depression decreases intraischemic cerebral glutamate levels and down-regulates excitatory amino acid transporters EAAT1 and EAAT2 from rat cerebal cortex plasma membranes

Ischemic Preconditioning Induces Selective Translocation of Protein Kinase C Isoforms and in the Heart of Conscious Rabbits Without Subcellular Redistribution of Total Protein Kinase C Activity

In vivo protein transduction: delivery of a biologically active protein into the mouse

Regulation of transcription by MAP kinase cascades

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6

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Daria Mochly-Rosen

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