Transient receptor potential vanilloid type 1 activation down-regulates voltage-gated calcium channels through calcium-dependent calcineurin in sensory neurons.
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TRPV1 channels mediate long-term depression at synapses on hippocampal interneuronsAminopyridines potentiate synaptic and neuromuscular transmission by targeting the voltage-activated calcium channel beta subunitTRPV1: A Target for Rational Drug DesignCapsaicin: Current Understanding of Its Mechanisms and Therapy of Pain and Other Pre-Clinical and Clinical UsesTRPV1 and Endocannabinoids: Emerging Molecular Signals that Modulate Mammalian VisionUse of Capsaicin to Treat Pain: Mechanistic and Therapeutic ConsiderationsMechanisms of prolonged presynaptic Ca2+ signaling and glutamate release induced by TRPV1 activation in rat sensory neuronsA current review of cypermethrin-induced neurotoxicity and nigrostriatal dopaminergic neurodegenerationAKAP150-mediated TRPV1 sensitization is disrupted by calcium/calmodulin.Casein kinase II and calcineurin modulate TRPP function and ciliary localizationCompartment-specific modulation of GABAergic synaptic transmission by TRPV1 channels in the dentate gyrus.Capsaicin in the periaqueductal gray induces analgesia via metabotropic glutamate receptor-mediated endocannabinoid retrograde disinhibitionTargeting N-methyl-D-aspartate receptors for treatment of neuropathic painNitric oxide inhibits nociceptive transmission by differentially regulating glutamate and glycine release to spinal dorsal horn neurons.Resistance to morphine analgesic tolerance in rats with deleted transient receptor potential vanilloid type 1-expressing sensory neurons.TRPV1: contribution to retinal ganglion cell apoptosis and increased intracellular Ca2+ with exposure to hydrostatic pressure.Upregulation of nuclear factor of activated T-cells by nerve injury contributes to development of neuropathic pain.Permeation and block of TRPV1 channels by the cationic lidocaine derivative QX-314Phosphoinositide signaling in somatosensory neuronsRemoving TRPV1-expressing primary afferent neurons potentiates the spinal analgesic effect of delta-opioid agonists on mechano-nociception.The glutamatergic nature of TRPV1-expressing neurons in the spinal dorsal hornA functional link between T-type calcium channels and mu-opioid receptor expression in adult primary sensory neurons.Long-term potentiation in hippocampal oriens interneurons: postsynaptic induction, presynaptic expression and evaluation of candidate retrograde factors.Roles of transient receptor potential channels in pain.Casein kinase II inhibition reverses pain hypersensitivity and potentiated spinal N-methyl-D-aspartate receptor activity caused by calcineurin inhibitorCoexpression and activation of TRPV1 suppress the activity of the KCNQ2/3 channel.Peripheral inflammation affects modulation of nociceptive synaptic transmission in the spinal cord induced by N-arachidonoylphosphatidylethanolamine.Acteoside inhibits PMA-induced matrix metalloproteinase-9 expression via CaMK/ERK- and JNK/NF-κB-dependent signaling.Tunable calcium current through TRPV1 receptor channelsModulation of CaV2.3 calcium channel currents by eugenol.Activation of TLR-4 to produce tumour necrosis factor-α in neuropathic pain caused by paclitaxel.Clustering and Functional Coupling of Diverse Ion Channels and Signaling Proteins Revealed by Super-resolution STORM Microscopy in Neurons.Homologous and heterologous desensitization of capsaicin and mustard oil responses utilize different cellular pathways in nociceptors.Antinociceptive desensitizing actions of TRPV1 receptor agonists capsaicin, resiniferatoxin and N-oleoyldopamine as measured by determination of the noxious heat and cold thresholds in the rat.Loss of TRPV1-expressing sensory neurons reduces spinal mu opioid receptors but paradoxically potentiates opioid analgesia.Calcineurin inhibitor induces pain hypersensitivity by potentiating pre- and postsynaptic NMDA receptor activity in spinal cords.Tumour necrosis factor alpha activates nuclear factor kappaB signalling to reduce N-type voltage-gated Ca2+ current in postganglionic sympathetic neurons.Capsaicin presynaptically inhibits glutamate release through the activation of TRPV1 and calcineurin in the hippocampus of rats.Modulation of intracellular calcium influences capsaicin-induced currents of TRPV-1 and voltage-activated channel currents in nociceptive neurones.Potential for therapeutic targeting of AKAP signaling complexes in nervous system disorders.
P2860
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P2860
Transient receptor potential vanilloid type 1 activation down-regulates voltage-gated calcium channels through calcium-dependent calcineurin in sensory neurons.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Transient receptor potential v ...... alcineurin in sensory neurons.
@en
Transient receptor potential v ...... alcineurin in sensory neurons.
@nl
type
label
Transient receptor potential v ...... alcineurin in sensory neurons.
@en
Transient receptor potential v ...... alcineurin in sensory neurons.
@nl
prefLabel
Transient receptor potential v ...... alcineurin in sensory neurons.
@en
Transient receptor potential v ...... alcineurin in sensory neurons.
@nl
P2093
P2860
P356
P1476
Transient receptor potential v ...... alcineurin in sensory neurons.
@en
P2093
Hui-Lin Pan
Shao-Rui Chen
Zi-Zhen Wu
P2860
P304
18142-18151
P356
10.1074/JBC.M501229200
P407
P577
2005-03-03T00:00:00Z