Activation of syndecan-1 ectodomain shedding by Staphylococcus aureus alpha-toxin and beta-toxin. - Wikidata - wikimedia - TriplyDB

Activation of syndecan-1 ectodomain shedding by Staphylococcus aureus alpha-toxin and beta-toxin.

Activation of syndecan-1 ectodomain shedding by Staphylococcus aureus alpha-toxin and beta-toxin.

http://www.wikidata.org/entity/Q47428605

about

Acceleration of epithelial cell syndecan-1 shedding by anthrax hemolytic virulence factors.Bacterial Sphingomyelinases and Phospholipases as Virulence Factors Comparative genomics of the syndecans defines an ancestral genomic context associated with matrilins in vertebrates A mouse model of otitis media identifies HB-EGF as a mediator of inflammation-induced mucosal proliferation.Syndecans reside in sphingomyelin-enriched low-density fractions of the plasma membrane isolated from a parathyroid cell line Role of pore-forming toxins in bacterial infectious diseases.Syndecan-1 promotes Staphylococcus aureus corneal infection by counteracting neutrophil-mediated host defense.Syndecan-1 in the mouse parietal peritoneum microcirculation in inflammation.2-O-Sulfated Domains in Syndecan-1 Heparan Sulfate Inhibit Neutrophil Cathelicidin and Promote Staphylococcus aureus Corneal Infection Molecular and cellular mechanisms of ectodomain shedding Role of glycosaminoglycans in infectious disease Proteoglycans in host-pathogen interactions: molecular mechanisms and therapeutic implications Syndecan-1 (CD138) deficiency increases Staphylococcus aureus infection but has no effect on pathology in a mouse model of peritoneal dialysis.Syndecan-1 is an in vivo suppressor of Gram-positive toxic shock The endothelial glycocalyx in syndecan-1 deficient mice.Role of heparan sulfate in ocular diseases.Constitutive and accelerated shedding of murine syndecan-1 is mediated by cleavage of its core protein at a specific juxtamembrane site Staphylococcus aureus beta-toxin induces lung injury through syndecan-1.Glycosaminoglycans and infection.Syndecan-1 shedding facilitates the resolution of neutrophilic inflammation by removing sequestered CXC chemokines.Role of MAPK p38 in the cellular responses to pore-forming toxins.Molecular functions of syndecan-1 in disease.Shedding of cell membrane-bound proteoglycans.Heparan sulphate, its derivatives and analogues share structural characteristics that can be exploited, particularly in inhibiting microbial attachment.Syndecans as modulators and potential pharmacological targets in cancer progression.Insights into the molecular roles of heparan sulfate proteoglycans (HSPGs-syndecans) in autocrine and paracrine growth factor signaling in the pathogenesis of Hodgkin's lymphoma.Syndecan-1 ectodomain shedding is regulated by the small GTPase Rab5 Alpha-toxin interferes with integrin-mediated adhesion and internalization of Staphylococcus aureus by epithelial cells.Syndecan-1 expression in epithelial cells is induced by transforming growth factor beta through a PKA-dependent pathway.Syndecan 1 shedding contributes to Pseudomonas aeruginosa sepsis.Primary dermal fibroblasts derived from sdc-1 deficient mice migrate faster and have altered alphav integrin function.Interaction of calmodulin with L-selectin at the membrane interface: implication on the regulation of L-selectin shedding.Characterization of Virulence Factors of Staphylococcus aureus: Novel Function of Known Virulence Factors That Are Implicated in Activation of Airway Epithelial Proinflammatory Response.Mechanical strain regulates syndecan-4 expression and shedding in smooth muscle cells through differential activation of MAP kinase signaling pathways.Cell-associated hemolysis induced by Helicobacter pylori is mediated by phospholipases with mitogen-activated protein kinase-activating properties.Alpha-toxin facilitates the generation of CXC chemokine gradients and stimulates neutrophil homing in Staphylococcus aureus pneumonia.Secreted neutral metalloproteases of Bacillus anthracis as candidate pathogenic factors.Canonical Stimulation of the NLRP3 Inflammasome by Fungal Antigens Links Innate and Adaptive B-Lymphocyte Responses by Modulating IL-1β and IgM Production.Plasmin- and thrombin-accelerated shedding of syndecan-4 ectodomain generates cleavage sites at Lys(114)-Arg(115) and Lys(129)-Val(130) bonds.Porphyromonas gingivalis gingipains mediate the shedding of syndecan-1 from the surface of gingival epithelial cells.

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P2860

Activation of syndecan-1 ectodomain shedding by Staphylococcus aureus alpha-toxin and beta-toxin.

http://www.wikidata.org/entity/Q47428605

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

@zh-my

2003年学术文章

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2003年學術文章

@yue

2003年學術文章

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name

Activation of syndecan-1 ectod ...... us alpha-toxin and beta-toxin.

@en

Activation of syndecan-1 ectod ...... us alpha-toxin and beta-toxin.

@nl

type

label

Activation of syndecan-1 ectod ...... us alpha-toxin and beta-toxin.

@en

Activation of syndecan-1 ectod ...... us alpha-toxin and beta-toxin.

@nl

prefLabel

Activation of syndecan-1 ectod ...... us alpha-toxin and beta-toxin.

@en

Activation of syndecan-1 ectod ...... us alpha-toxin and beta-toxin.

@nl

P1433

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P1433

Journal of Biological Chemistry

P1476

Activation of syndecan-1 ectod ...... us alpha-toxin and beta-toxin.

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P2093

Michael Klagsbrun

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Sheila J Duncan

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Timothy J Foster

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Ye Chen

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P2860

Activation of HER4 by heparin-binding EGF-like growth factor stimulates chemotaxis but not proliferation

A metalloprotease-disintegrin, MDC9/meltrin-gamma/ADAM9 and PKCdelta are involved in TPA-induced ectodomain shedding of membrane-anchored heparin-binding EGF-like growth factor.

Ectodomain shedding of epidermal growth factor receptor ligands is required for keratinocyte migration in cutaneous wound healing

Structure of staphylococcal alpha-hemolysin, a heptameric transmembrane pore

A novel proteolytic cleavage involved in Notch signaling: the role of the disintegrin-metalloprotease TACE

gamma -Secretase cleavage and nuclear localization of ErbB-4 receptor tyrosine kinase

Platelet-activating factor receptor and ADAM10 mediate responses to Staphylococcus aureus in epithelial cells

Cortactin-Src kinase signaling pathway is involved in N-syndecan-dependent neurite outgrowth

Matrix metalloproteinase-3 releases active heparin-binding EGF-like growth factor by cleavage at a specific juxtamembrane site

Autoinduction and signal transduction in the regulation of staphylococcal virulence

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251-258

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10.1074/JBC.M308537200

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1

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279

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14573623

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