Inhibition of protein phosphatase 2A- and protein phosphatase 1-induced tau hyperphosphorylation and impairment of spatial memory retention in rats.
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Protein phosphatase 2A dysfunction in Alzheimer's diseasePhosphoprotein phosphatase 2A: a novel druggable target for Alzheimer's diseaseIs protein phosphatase inhibition responsible for the toxic effects of okadaic Acid in animals?The links between axin and carcinogenesisBiomaterials for the Treatment of Alzheimer's DiseaseBerberine attenuates axonal transport impairment and axonopathy induced by Calyculin A in N2a cellsSilencing PP2A inhibitor by lenti-shRNA interference ameliorates neuropathologies and memory deficits in tg2576 mice.Some recent advances in basic neuroscience research in China.Role of protein phosphatases and mitochondria in the neuroprotective effects of estrogens.Silencing [Formula: see text] Rescues Tau Pathologies and Memory Deficits through Rescuing PP2A and Inhibiting GSK-3β Signaling in Human Tau Transgenic Mice.Pituitary adenylate cyclase-activating polypeptide prevents the effects of ceramides on migration, neurite outgrowth, and cytoskeleton remodeling.A proteomic analysis of MCLR-induced neurotoxicity: implications for Alzheimer's disease.The effects of protein phosphatase inhibitors on the duration of central sensitization of rat dorsal horn neurons following injection of capsaicinUp-regulation of inhibitors of protein phosphatase-2A in Alzheimer's disease.Kinases and phosphatases and tau sites involved in Alzheimer neurofibrillary degenerationThe carboxy-terminal fragment of inhibitor-2 of protein phosphatase-2A induces Alzheimer disease pathology and cognitive impairment.Genetic programming by the proteolytic fragments of the amyloid precursor protein: somewhere between confusion and clarity.The role of protein phosphatase-1 in the modulation of synaptic and structural plasticity.Effect of treadmill exercise on PI3K/AKT/mTOR, autophagy, and Tau hyperphosphorylation in the cerebral cortex of NSE/htau23 transgenic miceThe neuropeptide pituitary adenylate cyclase-activating polypeptide exerts anti-apoptotic and differentiating effects during neurogenesis: focus on cerebellar granule neurones and embryonic stem cells.Aging analysis reveals slowed tau turnover and enhanced stress response in a mouse model of tauopathy.Decrease of protein phosphatase 2A and its association with accumulation and hyperphosphorylation of tau in Down syndrome.The role of tau in neurodegenerationTau-based treatment strategies in neurodegenerative diseasesc-Jun N-terminal Kinase (JNK) induces phosphorylation of amyloid precursor protein (APP) at Thr668, in okadaic acid-induced neurodegeneration.Screening of treatment targets for Alzheimer's disease from the molecular mechanisms of impairment by β-amyloid aggregation and tau hyperphosphorylation.Tau hyperphosphorylation induces apoptotic escape and triggers neurodegeneration in Alzheimer's disease.Molecular and cellular mechanism of okadaic acid (OKA)-induced neurotoxicity: a novel tool for Alzheimer's disease therapeutic application.Serine/threonine-protein phosphatase 1 α levels are paralleling olfactory memory formation in the CD1 mouse.Amyloid-β as a biomarker for Alzheimer's disease: quantification methods in body fluids.Polymeric alkylpyridinium salts permit intracellular delivery of human Tau in rat hippocampal neurons: requirement of Tau phosphorylation for functional deficits.Melatonin reduces the impairment of axonal transport and axonopathy induced by calyculin A.Binding of tau to heat shock protein 27 leads to decreased concentration of hyperphosphorylated tau and enhanced cell survival.Modulation of hippocampal calcium signalling and plasticity by serine/threonine protein phosphatases.Perinatal exposure to Di-(2-ethylhexyl)-Phthalate leads to cognitive dysfunction and phospho-tau level increase in aged rats.Effects of intrahippocampal administration of the phosphatase inhibitor okadaic acid: Dual effects on memory formation.Eicosanoyl-5-hydroxytryptamide (EHT) prevents Alzheimer's disease-related cognitive and electrophysiological impairments in mice exposed to elevated concentrations of oligomeric beta-amyloid.Betaine attenuates Alzheimer-like pathological changes and memory deficits induced by homocysteine.Melatonin ameliorates Alzheimer-like pathological changes and spatial memory retention impairment induced by calyculin A.Recent Status of Nanomaterial Fabrication and Their Potential Applications in Neurological Disease Management
P2860
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P2860
Inhibition of protein phosphatase 2A- and protein phosphatase 1-induced tau hyperphosphorylation and impairment of spatial memory retention in rats.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Inhibition of protein phosphat ...... tial memory retention in rats.
@en
Inhibition of protein phosphat ...... tial memory retention in rats.
@nl
type
label
Inhibition of protein phosphat ...... tial memory retention in rats.
@en
Inhibition of protein phosphat ...... tial memory retention in rats.
@nl
prefLabel
Inhibition of protein phosphat ...... tial memory retention in rats.
@en
Inhibition of protein phosphat ...... tial memory retention in rats.
@nl
P2093
P1433
P1476
Inhibition of protein phosphat ...... tial memory retention in rats.
@en
P2093
P304
P356
10.1016/S0306-4522(02)00697-8
P407
P577
2003-01-01T00:00:00Z