Ink4a/Arf and oncogene-induced senescence prevent tumor progression during alternative colorectal tumorigenesis. - Wikidata - wikimedia - TriplyDB

Ink4a/Arf and oncogene-induced senescence prevent tumor progression during alternative colorectal tumorigenesis.

Ink4a/Arf and oncogene-induced senescence prevent tumor progression during alternative colorectal tumorigenesis.

http://www.wikidata.org/entity/Q53306400

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Mouse models of intestinal cancer Similar but different: distinct roles for KRAS and BRAF oncogenes in colorectal cancer development and therapy resistance Are KRAS/BRAF mutations potent prognostic and/or predictive biomarkers in colorectal cancers?Genetics of the serrated pathway to colorectal cancer P16INK4a overexpression and survival in osteosarcoma patients: a meta analysis A genetic progression model of Braf(V600E)-induced intestinal tumorigenesis reveals targets for therapeutic intervention.TLR4 activates the β-catenin pathway to cause intestinal neoplasia Registered report: senescence surveillance of pre-malignant hepatocytes limits liver cancer development Quantitation and Identification of Thousands of Human Proteoforms below 30 kDa Plasma homocysteine levels and genetic polymorphisms in folate metablism are associated with breast cancer risk in chinese women.Senescent tumor cells lead the collective invasion in thyroid cancer.Epithelial tissues have varying degrees of susceptibility to Kras(G12D)-initiated tumorigenesis in a mouse model.A complex secretory program orchestrated by the inflammasome controls paracrine senescence.Absence of Runx3 expression in normal gastrointestinal epithelium calls into question its tumour suppressor function The essence of senescence.Induction of histiocytic sarcoma in mouse skeletal muscle.Frequent epigenetic silencing of the folate-metabolising gene cystathionine-beta-synthase in gastrointestinal cancer.High-fat-diet-mediated dysbiosis promotes intestinal carcinogenesis independently of obesity.Human papillomavirus E7 oncoprotein induces KDM6A and KDM6B histone demethylase expression and causes epigenetic reprogramming.BRAF mutation is a powerful prognostic factor in advanced and recurrent colorectal cancer Resveratrol induces premature senescence in lung cancer cells via ROS-mediated DNA damage.Dual-specificity phosphatase 5 regulates nuclear ERK activity and suppresses skin cancer by inhibiting mutant Harvey-Ras (HRasQ61L)-driven SerpinB2 expression Prognostic significance of KRAS gene mutations in colorectal cancer--preliminary study.RAS transformation requires CUX1-dependent repair of oxidative DNA damage.Do all lung adenocarcinomas follow a stepwise progression?Long Term Exposure to Polyphenols of Artichoke (Cynara scolymus L.) Exerts Induction of Senescence Driven Growth Arrest in the MDA-MB231 Human Breast Cancer Cell Line.Distinct Transcriptional Changes and Epithelial-Stromal Interactions Are Altered in Early-Stage Colon Cancer Development p21 loss blocks senescence following Apc loss and provokes tumourigenesis in the renal but not the intestinal epithelium Oncogenic KRas suppresses inflammation-associated senescence of pancreatic ductal cells RasGRP1 opposes proliferative EGFR-SOS1-Ras signals and restricts intestinal epithelial cell growth.Pivotal Role of the Chromatin Protein Nupr1 in Kras-Induced Senescence and Transformation.Oncogenic KRAS signalling promotes the Wnt/β-catenin pathway through LRP6 in colorectal cancer.p16 Hypermethylation and KRAS Mutation Are Independent Predictors of Cetuximab Plus FOLFIRI Chemotherapy in Patients with Metastatic Colorectal Cancer Targeting aurora kinases limits tumour growth through DNA damage-mediated senescence and blockade of NF-κB impairs this drug-induced senescence.Pathology of rodent models of intestinal cancer: progress report and recommendations Up-regulation of c-MYC and SIRT1 expression correlates with malignant transformation in the serrated route to colorectal cancer.Senescence at a glance Nanoproteomic analysis of extracellular receptor kinase-1/2 post-translational activation in microdissected human hyperplastic colon lesions The FBXO4 tumor suppressor functions as a barrier to BRAFV600E-dependent metastatic melanoma.Mouse tissues that undergo neoplastic progression after K-Ras activation are distinguished by nuclear translocation of phospho-Erk1/2 and robust tumor suppressor responses.

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Ink4a/Arf and oncogene-induced senescence prevent tumor progression during alternative colorectal tumorigenesis.

http://www.wikidata.org/entity/Q53306400

description

2010 nî lūn-bûn

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name

Ink4a/Arf and oncogene-induced ...... tive colorectal tumorigenesis.

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Ink4a/Arf and oncogene-induced ...... tive colorectal tumorigenesis.

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type

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Ink4a/Arf and oncogene-induced ...... tive colorectal tumorigenesis.

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Ink4a/Arf and oncogene-induced ...... tive colorectal tumorigenesis.

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Ink4a/Arf and oncogene-induced ...... tive colorectal tumorigenesis.

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J.CCR.2010.06.013

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Ink4a/Arf and oncogene-induced ...... tive colorectal tumorigenesis.

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Andreas Jung

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Kristin Retzlaff

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Lydia Kriegl

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Melek C Arkan

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Monther Bajbouj

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Moritz Bennecke

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Sylvie Robine

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Thomas Kirchner

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135-146

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scholarly article

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10.1016/J.CCR.2010.06.013

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2

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18

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Florian R Greten

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