about
A critical review of vitamin C for the prevention of age-related cognitive decline and Alzheimer's diseaseShared Neuropathological Characteristics of Obesity, Type 2 Diabetes and Alzheimer's Disease: Impacts on Cognitive DeclineAscorbic acid and the brain: rationale for the use against cognitive decline.Vitamin C deficiency increases basal exploratory activity but decreases scopolamine-induced activity in APP/PSEN1 transgenic miceIncreased expression of SVCT2 in a new mouse model raises ascorbic acid in tissues and protects against paraquat-induced oxidative damage in lung.Behavioral and serotonergic response changes in the Dhcr7-HET mouse model of Smith-Lemli-Opitz syndrome.Low brain ascorbic acid increases susceptibility to seizures in mouse models of decreased brain ascorbic acid transport and Alzheimer's diseaseDifferential regulation of the ascorbic acid transporter SVCT2 during development and in response to ascorbic acid depletion.Differential proteomic and behavioral effects of long-term voluntary exercise in wild-type and APP-overexpressing transgenics.Vitamin C facilitates dopamine neuron differentiation in fetal midbrain through TET1- and JMJD3-dependent epigenetic control mannerVitamin C deficiency in the brain impairs cognition, increases amyloid accumulation and deposition, and oxidative stress in APP/PSEN1 and normally aging mice.Elevated oxidative stress and sensorimotor deficits but normal cognition in mice that cannot synthesize ascorbic acidVitamin C function in the brain: vital role of the ascorbate transporter SVCT2.Ascorbic acid attenuates scopolamine-induced spatial learning deficits in the water maze.Combined vitamin C and E deficiency induces motor defects in gulo(-/-)/SVCT2(+/-) mice.Role of vitamin C in the function of the vascular endothelium.Intravenous ascorbate improves spatial memory in middle-aged APP/PSEN1 and wild type mice.Oxidative stress, serotonergic changes and decreased ultrasonic vocalizations in a mouse model of Smith-Lemli-Opitz syndrome.Reversal of high fat diet-induced obesity improves glucose tolerance, inflammatory response, β-amyloid accumulation and cognitive decline in the APP/PSEN1 mouse model of Alzheimer's disease.Impaired spatial learning in the APPSwe + PSEN1DeltaE9 bigenic mouse model of Alzheimer's disease.Elimination of GD3 synthase improves memory and reduces amyloid-beta plaque load in transgenic mice.Behavioural and neurochemical effects of scurvy in gulo knockout mice
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description
hulumtuese
@sq
medical researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Fiona E Harrison
@nl
Fiona E Harrison
@sl
Fiona E. Harrison
@en
Fiona E. Harrison
@es
type
label
Fiona E Harrison
@nl
Fiona E Harrison
@sl
Fiona E. Harrison
@en
Fiona E. Harrison
@es
prefLabel
Fiona E Harrison
@nl
Fiona E Harrison
@sl
Fiona E. Harrison
@en
Fiona E. Harrison
@es
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P1153
15127207700
P21
P31
P496
0000-0003-1121-0647