Impaired spatial learning in the APPSwe + PSEN1DeltaE9 bigenic mouse model of Alzheimer's disease.
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Galactic cosmic radiation leads to cognitive impairment and increased aβ plaque accumulation in a mouse model of Alzheimer's diseaseSIRT1 suppresses beta-amyloid production by activating the alpha-secretase gene ADAM10.Shared Neuropathological Characteristics of Obesity, Type 2 Diabetes and Alzheimer's Disease: Impacts on Cognitive DeclineElevation of brain magnesium prevents synaptic loss and reverses cognitive deficits in Alzheimer's disease mouse model.Vitamin D2-enriched button mushroom (Agaricus bisporus) improves memory in both wild type and APPswe/PS1dE9 transgenic miceTraditional Chinese Nootropic Medicine Radix Polygalae and Its Active Constituent Onjisaponin B Reduce β-Amyloid Production and Improve Cognitive ImpairmentsProtective Effects of Dietary Supplementation with a Combination of Nutrients in a Transgenic Mouse Model of Alzheimer's DiseaseTargeting the γ-/β-secretase interaction reduces β-amyloid generation and ameliorates Alzheimer's disease-related pathogenesisMetformin attenuates Alzheimer's disease-like neuropathology in obese, leptin-resistant mice.Epidermal growth factor receptor is a preferred target for treating amyloid-β-induced memory loss.Pyrrolidine dithiocarbamate attenuates brain Aβ increase and improves long-term neurological outcome in rats after transient focal brain ischemia.APP transgenic mice for modelling behavioural and psychological symptoms of dementia (BPSD).Allele-specific RNAi mitigates phenotypic progression in a transgenic model of Alzheimer's disease.Reduced IGF-1 signaling delays age-associated proteotoxicity in miceAngiotensin-converting enzyme overexpression in myelomonocytes prevents Alzheimer's-like cognitive declineMeasuring discrimination- and reversal learning in mouse models within 4 days and without prior food deprivation.Vitamin C deficiency increases basal exploratory activity but decreases scopolamine-induced activity in APP/PSEN1 transgenic miceYXQN Reduces Alzheimer's Disease-Like Pathology and Cognitive Decline in APPswePS1dE9 Transgenic Mice.Neuroinflammation and M2 microglia: the good, the bad, and the inflamed.17α-estradiol attenuates neuron loss in ovariectomized Dtg AβPP/PS1 mice.Type 1 diabetes exaggerates features of Alzheimer's disease in APP transgenic mice.Barnes maze testing strategies with small and large rodent models.Adipose-derived mesenchymal stem cell transplantation promotes adult neurogenesis in the brains of Alzheimer's disease mice.Intracranial V. cholerae sialidase protects against excitotoxic neurodegenerationAbnormal vibrissa-related behavior and loss of barrel field inhibitory neurons in 5xFAD transgenics.Overexpression of human apolipoprotein A-I preserves cognitive function and attenuates neuroinflammation and cerebral amyloid angiopathy in a mouse model of Alzheimer disease.An anterograde rabies virus vector for high-resolution large-scale reconstruction of 3D neuron morphologyUsing mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse modelsSmart Soup, a traditional Chinese medicine formula, ameliorates amyloid pathology and related cognitive deficits.A comparative evaluation of treatments with 17β-estradiol and its brain-selective prodrug in a double-transgenic mouse model of Alzheimer's diseaseAmyloid β-induced impairments in hippocampal synaptic plasticity are rescued by decreasing mitochondrial superoxideNovel age-dependent learning deficits in a mouse model of Alzheimer's disease: implications for translational research.Aminostyrylbenzofuran directly reduces oligomeric amyloid-β and reverses cognitive deficits in Alzheimer transgenic miceDifferential proteomic and behavioral effects of long-term voluntary exercise in wild-type and APP-overexpressing transgenics.Spatial and nonspatial escape strategies in the Barnes mazeIntraneuronal APP and extracellular Aβ independently cause dendritic spine pathology in transgenic mouse models of Alzheimer's disease.Vitamin C deficiency in the brain impairs cognition, increases amyloid accumulation and deposition, and oxidative stress in APP/PSEN1 and normally aging mice.Chronic IL-1β-mediated neuroinflammation mitigates amyloid pathology in a mouse model of Alzheimer's disease without inducing overt neurodegenerationEnhanced penetration of exogenous EPCs into brains of APP/PS1 transgenic mice.Small molecule LX2343 ameliorates cognitive deficits in AD model mice by targeting both amyloid β production and clearance.
P2860
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P2860
Impaired spatial learning in the APPSwe + PSEN1DeltaE9 bigenic mouse model of Alzheimer's disease.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Impaired spatial learning in t ...... model of Alzheimer's disease.
@en
Impaired spatial learning in t ...... model of Alzheimer's disease.
@nl
type
label
Impaired spatial learning in t ...... model of Alzheimer's disease.
@en
Impaired spatial learning in t ...... model of Alzheimer's disease.
@nl
prefLabel
Impaired spatial learning in t ...... model of Alzheimer's disease.
@en
Impaired spatial learning in t ...... model of Alzheimer's disease.
@nl
P1476
Impaired spatial learning in t ...... e model of Alzheimer's disease
@en
P2093
Reiserer RS
Syverud DC
P356
10.1111/J.1601-183X.2006.00221.X
P407
P577
2007-02-01T00:00:00Z