The NOD Idd9 genetic interval influences the pathogenicity of insulitis and contains molecular variants of Cd30, Tnfr2, and Cd137.
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Liver autoimmunity triggered by microbial activation of natural killer T cellsConstruction and analysis of tag single nucleotide polymorphism maps for six human-mouse orthologous candidate genes in type 1 diabetesIdd9.1 locus controls the suppressive activity of FoxP3+CD4+CD25+ regulatory T-cellsA wild derived quantitative trait locus on mouse chromosome 2 prevents obesity.Idd9.2 and Idd9.3 protective alleles function in CD4+ T-cells and nonlymphoid cells to prevent expansion of pathogenic islet-specific CD8+ T-cells.Use of nonobese diabetic mice to understand human type 1 diabetesDistinct genetic control of autoimmune neuropathy and diabetes in the non-obese diabetic background.A recombination hotspot leads to sequence variability within a novel gene (AK005651) and contributes to type 1 diabetes susceptibilityThe genetics of complex autoimmune diseases: non-MHC susceptibility genes.The immune system and gene expression microarrays--new answers to old questions.Blockade of the programmed death-1 (PD1) pathway undermines potent genetic protection from type 1 diabetes.Role of novel retroviruses in chronic liver disease: assessing the link of human betaretrovirus with primary biliary cirrhosisGenome-wide transcriptional analyses of islet-specific CD4+ T cells identify Idd9 genes controlling diabetogenic T cell functionResistance of the target islet tissue to autoimmune destruction contributes to genetic susceptibility in Type 1 diabetes.The regulatory role of DR4 in a spontaneous diabetes DQ8 transgenic modelModifier loci condition autoimmunity provoked by Aire deficiency.The B10 Idd9.3 locus mediates accumulation of functionally superior CD137(+) regulatory T cells in the nonobese diabetic type 1 diabetes model.Loss of T cell progenitor checkpoint control underlies leukemia initiation in Rag1-deficient nonobese diabetic mice.Dissecting genetic control of autoimmunity in NOD congenic mice.Animal models of primary biliary cirrhosisThe non-obese diabetic mouse sequence, annotation and variation resource: an aid for investigating type 1 diabetes.Co-stimulatory and Co-inhibitory Pathways in Autoimmunity.T-cell co-stimulatory pathways in autoimmunity.Idd9/11 genetic locus regulates diabetogenic activity of CD4 T-cells in nonobese diabetic (NOD) mice.NKG2D-RAE-1 receptor-ligand variation does not account for the NK cell defect in nonobese diabetic mice.Tissue- and age-specific changes in gene expression during disease induction and progression in NOD miceComparative genetics: synergizing human and NOD mouse studies for identifying genetic causation of type 1 diabetesCD8 T cells mediate direct biliary ductule damage in nonobese diabetic autoimmune biliary disease.Fine mapping of type 1 diabetes regions Idd9.1 and Idd9.2 reveals genetic complexity.γδ T cells are essential effectors of type 1 diabetes in the nonobese diabetic mouse model.Gene targeting in NOD mouse embryos using zinc-finger nucleases.Mouse models for the study of autoimmune type 1 diabetes: a NOD to similarities and differences to human disease.Advances in our understanding of the pathophysiology of Type 1 diabetes: lessons from the NOD mouse.Animal models of primary biliary cirrhosis.Genome-wide microarray expression analysis of CD4+ T Cells from nonobese diabetic congenic mice identifies Cd55 (Daf1) and Acadl as candidate genes for type 1 diabetes.Genetic predisposition for beta cell fragility underlies type 1 and type 2 diabetesFine mapping of quantitative trait loci affecting organ weights by mouse intersubspecific subcongenic strain analysis.CD137 Plays Both Pathogenic and Protective Roles in Type 1 Diabetes Development in NOD Mice.Congenic mapping identifies a novel Idd9 subregion regulating type 1 diabetes in NOD mice.The Role of NOD Mice in Type 1 Diabetes Research: Lessons from the Past and Recommendations for the Future.
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P2860
The NOD Idd9 genetic interval influences the pathogenicity of insulitis and contains molecular variants of Cd30, Tnfr2, and Cd137.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
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2000年學術文章
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2000年學術文章
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name
The NOD Idd9 genetic interval ...... nts of Cd30, Tnfr2, and Cd137.
@en
The NOD Idd9 genetic interval ...... nts of Cd30, Tnfr2, and Cd137.
@nl
type
label
The NOD Idd9 genetic interval ...... nts of Cd30, Tnfr2, and Cd137.
@en
The NOD Idd9 genetic interval ...... nts of Cd30, Tnfr2, and Cd137.
@nl
prefLabel
The NOD Idd9 genetic interval ...... nts of Cd30, Tnfr2, and Cd137.
@en
The NOD Idd9 genetic interval ...... nts of Cd30, Tnfr2, and Cd137.
@nl
P2093
P1433
P1476
The NOD Idd9 genetic interval ...... nts of Cd30, Tnfr2, and Cd137.
@en
P2093
Armitage N
Fischer PA
Hancock WW
Peterson LB
Phillips MS
P304
P356
10.1016/S1074-7613(00)00012-1
P407
P577
2000-07-01T00:00:00Z