Active caspase-3 is required for osteoclast differentiation
about
Quantitative proteomics analysis of inborn errors of cholesterol synthesis: identification of altered metabolic pathways in DHCR7 and SC5D deficiencyCaspase 3 promotes surviving melanoma tumor cell growth after cytotoxic therapy.Elevated cleaved caspase-3 is associated with shortened overall survival in several cancer types.Apoptotic caspases regulate induction of iPSCs from human fibroblasts.Sinomenine induces apoptosis in RAW 264.7 cell-derived osteoclasts in vitro via caspase-3 activation.Caspase 3-mediated stimulation of tumor cell repopulation during cancer radiotherapy.Caspase-2 maintains bone homeostasis by inducing apoptosis of oxidatively-damaged osteoclasts.A miniaturized device for bioluminescence analysis of caspase-3/7 activity in a single apoptotic cell.Involvement of multiple myeloma cell-derived exosomes in osteoclast differentiation.Targeting the XIAP/caspase-7 complex selectively kills caspase-3-deficient malignancies.Bis-enoxacin inhibits bone resorption and orthodontic tooth movementThe small GTPase RhoA is crucial for MC3T3-E1 osteoblastic cell survival.Executioners of apoptosis in pancreatic {beta}-cells: not just for cell death.Reactive oxygen species are required for zoledronic acid-induced apoptosis in osteoclast precursors and mature osteoclast-like cells.Apoptotic signaling in mouse odontogenesis.Osteoporosis--a risk factor for cardiovascular disease?Caspase-9 has a nonapoptotic function in Xenopus embryonic primitive blood formation.An inhibitory role for caspase-3 at the late stage of RANKL-induced osteoclast differentiation in RAW264 cells and mouse bone marrow macrophages.NEU3 sialidase strictly modulates GM3 levels in skeletal myoblasts C2C12 thus favoring their differentiation and protecting them from apoptosis.High d(+)glucose concentration inhibits RANKL-induced osteoclastogenesisInhibition of apoptosis signal-regulating kinase 1 enhances endochondral bone formation by increasing chondrocyte survival.Decoy receptor 3 suppresses RANKL-induced osteoclastogenesis via down-regulating NFATc1 and enhancing cell apoptosis.Caspase-7 participates in differentiation of cells forming dental hard tissues.Expression profiling of microRNAs in RAW264.7 cells treated with a combination of tumor necrosis factor alpha and RANKL during osteoclast differentiation.Non-apoptotic Caspase regulation of stem cell properties.Osteogenic Potential of Caspases Related to Endochondral Ossification.Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018.Suppression of RANKL-dependent heme oxygenase-1 is required for high mobility group box 1 release and osteoclastogenesis.Activation of Pro-apoptotic Caspases in Non-apoptotic Cells During Odontogenesis and Related Osteogenesis.Effect of caspases and RANKL induced by heavy force in orthodontic root resorption.
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P2860
Active caspase-3 is required for osteoclast differentiation
description
im Januar 2006 veröffentlichter wissenschaftlicher Artikel
@de
scientific article published on 01 December 2006
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована у 2006
@uk
name
Active caspase-3 is required for osteoclast differentiation
@en
Active caspase-3 is required for osteoclast differentiation
@nl
type
label
Active caspase-3 is required for osteoclast differentiation
@en
Active caspase-3 is required for osteoclast differentiation
@nl
prefLabel
Active caspase-3 is required for osteoclast differentiation
@en
Active caspase-3 is required for osteoclast differentiation
@nl
P2093
P2860
P356
P1476
Active caspase-3 is required for osteoclast differentiation
@en
P2093
Srinivas V
Steinbeck MJ
Szymczyk KH
P2860
P304
P356
10.1002/JCP.20770
P577
2006-12-01T00:00:00Z