Abnormal axonal physiology is associated with altered expression and distribution of Kv1.1 and Kv1.2 K+channels after chronic spinal cord injury
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Myelin damage and repair in pathologic CNS: challenges and prospectsA Novel Approach for Studying the Physiology and Pathophysiology of Myelinated and Non-Myelinated Axons in the CNS White MatterAxon physiology.Alterations of action potentials and the localization of Nav1.6 sodium channels in spared axons after hemisection injury of the spinal cord in adult rats.Genome-wide gene expression profiling of stress response in a spinal cord clip compression injury modelMotoneurones "learn" and "forget" physical activity.Kv1.3 channels in postganglionic sympathetic neurons: expression, function, and modulation.Pharmacological modulation of voltage-gated potassium channels as a therapeutic strategy.Potassium channel blockers as an effective treatment to restore impulse conduction in injured axons.Ion channel blockers and spinal cord injury.Nogo receptor 1 regulates Caspr distribution at axo-glial units in the central nervous system.Neural precursor cell transplantation enhances functional recovery and reduces astrogliosis in bilateral compressive/contusive cervical spinal cord injury.Novel potassium channel blocker, 4-AP-3-MeOH, inhibits fast potassium channels and restores axonal conduction in injured guinea pig spinal cord white matter.Upregulation of Kv 1.4 protein and gene expression after chronic spinal cord injury.Functional changes in genetically dysmyelinated spinal cord axons of shiverer mice: role of juxtaparanodal Kv1 family K+ channels.Temporal and spatial patterns of Kv1.1 and Kv1.2 protein and gene expression in spinal cord white matter after acute and chronic spinal cord injury in rats: implications for axonal pathophysiology after neurotrauma.GGF2 (Nrg1-β3) treatment enhances NG2+ cell response and improves functional recovery after spinal cord injury.Alterations of juxtaparanodal domains in two rodent models of CNS demyelination.Molecular and electrophysiological evidence for the expression of BK channels in oligodendroglial precursor cells.Microenvironmental regulation of oligodendrocyte replacement and remyelination in spinal cord injury.Contribution of fast and slow conducting myelinated axons to single-peak compound action potentials in rat spinal cord white matter preparations.
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Abnormal axonal physiology is associated with altered expression and distribution of Kv1.1 and Kv1.2 K+channels after chronic spinal cord injury
description
im Februar 2000 veröffentlichter wissenschaftlicher Artikel
@de
scientific article published on 01 February 2000
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в лютому 2000
@uk
name
Abnormal axonal physiology is ...... ter chronic spinal cord injury
@en
Abnormal axonal physiology is ...... ter chronic spinal cord injury
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type
label
Abnormal axonal physiology is ...... ter chronic spinal cord injury
@en
Abnormal axonal physiology is ...... ter chronic spinal cord injury
@nl
prefLabel
Abnormal axonal physiology is ...... ter chronic spinal cord injury
@en
Abnormal axonal physiology is ...... ter chronic spinal cord injury
@nl
P2860
P1476
Abnormal axonal physiology is ...... ter chronic spinal cord injury
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P2860
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10.1046/J.1460-9568.2000.00926.X
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P577
2000-02-01T00:00:00Z