The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
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Expression in mammalian cells and electrophysiological characterization of two mutant Kv1.1 channels causing episodic ataxia type 1 (EA-1)The pore-lining region of shaker voltage-gated potassium channels: comparison of beta-barrel and alpha-helix bundle modelsPotassium channel block by a tripartite complex of two cationophilic ligands and a potassium ion.Molecular diversity and regulation of renal potassium channels.Contributions of a negatively charged residue in the hydrophobic domain of the IRK1 inwardly rectifying K+ channel to K(+)-selective permeation.Hydrophobic mutations alter the movement of Mg2+ in the pore of voltage-gated potassium channels.A permanent ion binding site located between two gates of the Shaker K+ channel.Control of ion conduction in L-type Ca2+ channels by the concerted action of S5-6 regions.Increased Kv1 channel expression may contribute to decreased sIPSC frequency following chronic inhibition of NR2B-containing NMDAR.A mutation in S6 of Shaker potassium channels decreases the K+ affinity of an ion binding site revealing ion-ion interactions in the pore.Polyunsaturated Fatty acids modify the gating of kv channelsA novel current pathway parallel to the central pore in a mutant voltage-gated potassium channel.4-Chloro-3-nitro-N-butylbenzenesulfonamide acts on KV3.1 channels by an open-channel blocker mechanism.UK-78,282, a novel piperidine compound that potently blocks the Kv1.3 voltage-gated potassium channel and inhibits human T cell activation.Metal ions and human sperm mannose receptors.The signature sequence of voltage-gated potassium channels projects into the external vestibule.Regulation by external K+ in a maize inward shaker channel targets transport activity in the high concentration range.Subunit assembly and domain analysis of electrically silent K+ channel alpha-subunits of the rat Kv9 subfamily.Properties of bipolar VIPergic interneurons and their excitation by pyramidal neurons in the rat neocortex.Abnormal axonal physiology is associated with altered expression and distribution of Kv1.1 and Kv1.2 K+channels after chronic spinal cord injury
P2860
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P2860
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
description
1994 nî lūn-bûn
@nan
1994 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
name
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
@ast
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
@en
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
@nl
type
label
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
@ast
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
@en
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
@nl
prefLabel
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
@ast
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
@en
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
@nl
P2093
P2860
P1433
P1476
The P-region and S6 of Kv3.1 contribute to the formation of the ion conduction pathway
@en
P2093
P2860
P304
P356
10.1016/S0006-3495(94)80710-6
P407
P577
1994-12-01T00:00:00Z