Pleiotrophin signaling through anaplastic lymphoma kinase is rate-limiting for glioblastoma growth - sprookjes - yvandenbroek - TriplyDB

Pleiotrophin signaling through anaplastic lymphoma kinase is rate-limiting for glioblastoma growth

Pleiotrophin signaling through anaplastic lymphoma kinase is rate-limiting for glioblastoma growth

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Anaplastic lymphoma kinase: signalling in development and disease Anaplastic lymphoma kinase is activated through the pleiotrophin/receptor protein-tyrosine phosphatase beta/zeta signaling pathway: an alternative mechanism of receptor tyrosine kinase activation Emerging importance of ALK in neuroblastoma Midkine binds to anaplastic lymphoma kinase (ALK) and acts as a growth factor for different cell types Shc3 affects human high-grade astrocytomas survival Differential induction of glioblastoma migration and growth by two forms of pleiotrophin Inhibitors of the anaplastic lymphoma kinase Gene expression profiling identifies regulatory pathways involved in the late stage of rat fetal lung development The pro-angiogenic cytokine pleiotrophin potentiates cardiomyocyte apoptosis through inhibition of endogenous AKT/PKB activity Effect of single-chain antibody targeting of the ligand-binding domain in the anaplastic lymphoma kinase receptor.Suppression of lung adenocarcinoma through menin and polycomb gene-mediated repression of growth factor pleiotrophin.Activated Alk triggers prolonged neurogenesis and Ret upregulation providing a therapeutic target in ALK-mutated neuroblastoma.ALK-rearrangements and testing methods in non-small cell lung cancer: a review miR-182 aids in receptive endometrium development in dairy goats by down-regulating PTN expression The synthetic peptide P111-136 derived from the C-terminal domain of heparin affin regulatory peptide inhibits tumour growth of prostate cancer PC-3 cells.The role of pleiotrophin and beta-catenin in fetal lung development.The receptor tyrosine kinase Alk controls neurofibromin functions in Drosophila growth and learning.A Pleiotrophin C-terminus peptide induces anti-cancer effects through RPTPβ/ζ Expression analysis of macrodactyly identifies pleiotrophin upregulation.ALK-driven tumors and targeted therapy: focus on crizotinib The Drosophila midkine/pleiotrophin homologues Miple1 and Miple2 affect adult lifespan but are dispensable for alk signaling during embryonic gut formation.Pleiotrophin as a central nervous system neuromodulator, evidences from the hippocampus.Stimulation of the midkine/ALK axis renders glioma cells resistant to cannabinoid antitumoral action Pleiotrophin disrupts calcium-dependent homophilic cell-cell adhesion and initiates an epithelial-mesenchymal transition.Menin represses tumorigenesis via repressing cell proliferation.ALK a novel lymphoma-associated tumor antigen for vaccination strategies.Anaplastic lymphoma kinase is expressed in different subtypes of human breast cancer.Fusion genes with ALK as recurrent partner in ependymoma-like gliomas: a new brain tumor entity?ALK alteration is a frequent event in aggressive breast cancers.The receptor protein tyrosine phosphatase (RPTP)beta/zeta is expressed in different subtypes of human breast cancer.Anaplastic lymphoma kinase and its signalling molecules as novel targets in lymphoma therapy.Synergistic Effects of Crizotinib and Temozolomide in Experimental FIG-ROS1 Fusion-Positive Glioblastoma.Catalytic nucleic acid enzymes for the study and development of therapies in the central nervous system: Review Article.ALK receptor activation, ligands and therapeutic targeting in glioblastoma and in other cancers.Mass spectrometry-based proteomic studies of human anaplastic large cell lymphoma.Targeting pleiotropin to treat osteoarthritis.Anaplastic lymphoma kinase is dynamically expressed on subsets of motor neurons and in the peripheral nervous system.Enhanced antitumorigenic effects in glioblastoma on double targeting of pleiotrophin and its receptor ALK.Receptor-type tyrosine phosphatase ligands: looking for the needle in the haystack.Anaplastic lymphoma kinase: role in cancer pathogenesis and small-molecule inhibitor development for therapy.

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Pleiotrophin signaling through anaplastic lymphoma kinase is rate-limiting for glioblastoma growth

http://www.wikidata.org/entity/Q28216536

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2002 nî lūn-bûn

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2002 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2002 թվականի ապրիլին հրատարակված գիտական հոդված

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2002年の論文

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Pleiotrophin signaling through ...... miting for glioblastoma growth

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Pleiotrophin signaling through ...... miting for glioblastoma growth

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Pleiotrophin signaling through ...... miting for glioblastoma growth

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Pleiotrophin signaling through ...... miting for glioblastoma growth

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Journal of Biological Chemistry

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Pleiotrophin signaling through ...... miting for glioblastoma growth

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Anton Wellstein

http://www.w3.org/2001/XMLSchema#string

Ciaran Powers

http://www.w3.org/2001/XMLSchema#string

Gerald E Stoica

http://www.w3.org/2001/XMLSchema#string

Kevin McDonnell

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P2860

Identification of anaplastic lymphoma kinase as a receptor for the growth factor pleiotrophin

Fusion of a kinase gene, ALK, to a nucleolar protein gene, NPM, in non-Hodgkin's lymphoma

Molecular characterization of ALK, a receptor tyrosine kinase expressed specifically in the nervous system

PTEN/MMAC1/TEP1 suppresses the tumorigenicity and induces G1 cell cycle arrest in human glioblastoma cells

PTEN: a tumour suppressor that functions as a phospholipid phosphatase.

A secreted FGF-binding protein can serve as the angiogenic switch in human cancer.

PTEN controls tumor-induced angiogenesis

A mutant epidermal growth factor receptor common in human glioma confers enhanced tumorigenicity.

Pleiotrophin transforms NIH 3T3 cells and induces tumors in nude mice.

Trans receptor inhibition of human glioblastoma cells by erbB family ectodomains.

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10.1074/JBC.M112354200

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16

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277

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2002-04-19T00:00:00Z

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11549156

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11809760

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