The dysbindin-containing complex (BLOC-1) in brain: developmental regulation, interaction with SNARE proteins and role in neurite outgrowth
about
The schizophrenia susceptibility factor dysbindin and its associated complex sort cargoes from cell bodies to the synapseAssembly and architecture of biogenesis of lysosome-related organelles complex-1 (BLOC-1).Nucleocytoplasmic shuttling of dysbindin-1, a schizophrenia-related protein, regulates synapsin I expressionSchizophrenia susceptibility gene dysbindin regulates glutamatergic and dopaminergic functions via distinctive mechanisms in DrosophilaHomeostatic plasticity at the Drosophila neuromuscular junctionYeast homologues of three BLOC-1 subunits highlight KxDL proteins as conserved interactors of BLOC-1.Gene × Environment Interactions in Schizophrenia: Evidence from Genetic Mouse ModelsThe Cannabinoid CB1/CB2 Agonist WIN55212.2 Promotes Oligodendrocyte Differentiation In Vitro and Neuroprotection During the Cuprizone-Induced Central Nervous System Demyelination.Dysbindin-containing complexes and their proposed functions in brain: from zero to (too) many in a decade.BLOC-1 and BLOC-3 regulate VAMP7 cycling to and from melanosomes via distinct tubular transport carriersMutations in the BLOC-1 subunits dysbindin and muted generate divergent and dosage-dependent phenotypes.Genetic modifiers of abnormal organelle biogenesis in a Drosophila model of BLOC-1 deficiency.GWA study data mining and independent replication identify cardiomyopathy-associated 5 (CMYA5) as a risk gene for schizophreniaBLOC-1 deficiency causes alterations in amino acid profile and in phospholipid and adenosine metabolism in the postnatal mouse hippocampus.Neuroplasticity signaling pathways linked to the pathophysiology of schizophrenia.Dysbindin-associated proteome in the p2 synaptosome fraction of mouse brainDysbindin-1 genotype effects on emotional working memoryRETRACTED: A BLOC-1 mutation screen reveals that PLDN is mutated in Hermansky-Pudlak Syndrome type 9.MeCP2 regulates the synaptic expression of a Dysbindin-BLOC-1 network component in mouse brain and human induced pluripotent stem cell-derived neurons.Disrupted-in-schizophrenia 1 (DISC1) regulates dysbindin function by enhancing its stabilityEarly effects of lipopolysaccharide-induced inflammation on foetal brain development in rat.The N-ethylmaleimide-sensitive factor and dysbindin interact to modulate synaptic plasticity.The Schizophrenia-Related Protein Dysbindin-1A Is Degraded and Facilitates NF-Kappa B Activity in the NucleusCell biology of the BLOC-1 complex subunit dysbindin, a schizophrenia susceptibility gene.Cardiac Dysfunction in the BACHD Mouse Model of Huntington's Disease.Mechanisms of protein delivery to melanosomes in pigment cells.Neuronal copper homeostasis susceptibility by genetic defects in dysbindin, a schizophrenia susceptibility factor.Transmission, Development, and Plasticity of Synapses.Voltage-gated Ca2+ entry promotes oligodendrocyte progenitor cell maturation and myelination in vitro.Misaligned feeding impairs memories.Loss of dysbindin-1 in mice impairs reward-based operant learning by increasing impulsive and compulsive behaviorNMDA hypofunction as a convergence point for progression and symptoms of schizophreniaEffects of sex and DTNBP1 (dysbindin) null gene mutation on the developmental GluN2B-GluN2A switch in the mouse cortex and hippocampus.Increased dysbindin-1B isoform expression in schizophrenia and its propensity in aggresome formation.Lysosome-related organelles: unusual compartments become mainstream.Regional differences in expression of β-tubulin isoforms in schizophrenia.Presynaptic membrane retrieval and endosome biology: defining molecularly heterogeneous synaptic vesiclesConditional Deletion of the L-Type Calcium Channel Cav1.2 in Oligodendrocyte Progenitor Cells Affects Postnatal Myelination in Mice.The Proteome of BLOC-1 Genetic Defects Identifies the Arp2/3 Actin Polymerization Complex to Function Downstream of the Schizophrenia Susceptibility Factor Dysbindin at the SynapseThe schizophrenia susceptibility gene DTNBP1 modulates AMPAR synaptic transmission and plasticity in the hippocampus of juvenile DBA/2J mice.
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The dysbindin-containing complex (BLOC-1) in brain: developmental regulation, interaction with SNARE proteins and role in neurite outgrowth
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The dysbindin-containing compl ...... and role in neurite outgrowth
@ast
The dysbindin-containing compl ...... and role in neurite outgrowth
@en
The dysbindin-containing compl ...... and role in neurite outgrowth
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The dysbindin-containing compl ...... and role in neurite outgrowth
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type
label
The dysbindin-containing compl ...... and role in neurite outgrowth
@ast
The dysbindin-containing compl ...... and role in neurite outgrowth
@en
The dysbindin-containing compl ...... and role in neurite outgrowth
@en-gb
The dysbindin-containing compl ...... and role in neurite outgrowth
@nl
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The dysbindin-containing compl ...... and role in neurite outgrowth
@ast
The dysbindin-containing compl ...... and role in neurite outgrowth
@en
The dysbindin-containing compl ...... and role in neurite outgrowth
@en-gb
The dysbindin-containing compl ...... and role in neurite outgrowth
@nl
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The dysbindin-containing compl ...... and role in neurite outgrowth
@en
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E C Dell'Angelica
I A Rodriguez-Fernandez
J S Malvar
J de Vellis
M Starcevic
R Nazarian
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115, 204-15
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P356
10.1038/MP.2009.58
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P577
2009-06-23T00:00:00Z
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1011106641