DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release - Wikidata - awgldk - TriplyDB

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

http://www.wikidata.org/entity/Q28586199

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The schizophrenia susceptibility factor dysbindin and its associated complex sort cargoes from cell bodies to the synapse Nucleocytoplasmic shuttling of dysbindin-1, a schizophrenia-related protein, regulates synapsin I expression The dysbindin-containing complex (BLOC-1) in brain: developmental regulation, interaction with SNARE proteins and role in neurite outgrowth The schizophrenia susceptibility gene dysbindin controls synaptic homeostasis The EARP Complex and Its Interactor EIPR-1 Are Required for Cargo Sorting to Dense-Core Vesicles The ameliorating effects of 5,7-dihydroxy-6-methoxy-2(4-phenoxyphenyl)-4H-chromene-4-one, an oroxylin A derivative, against memory impairment and sensorimotor gating deficit in mice Snapin facilitates the synchronization of synaptic vesicle fusion Hermansky-Pudlak protein complexes, AP-3 and BLOC-1, differentially regulate presynaptic composition in the striatum and hippocampus Dysbindin-1, WAVE2 and Abi-1 form a complex that regulates dendritic spine formation Role of dysbindin in dopamine receptor trafficking and cortical GABA function Impaired long-term memory retention and working memory in sdy mutant mice with a deletion in Dtnbp1, a susceptibility gene for schizophrenia Animal Models of Psychosis: Current State and Future Directions.Neurodevelopmental animal models of schizophrenia: role in novel drug discovery and development Behavioral characterization of mice overexpressing human dysbindin-1.Animal models of schizophrenia.Dysbindin-1 modulates prefrontal cortical activity and schizophrenia-like behaviors via dopamine/D2 pathways.Dysbindin-1 mutant mice implicate reduced fast-phasic inhibition as a final common disease mechanism in schizophrenia.Zic2 hypomorphic mutant mice as a schizophrenia model and ZIC2 mutations identified in schizophrenia patients Dysbindin-containing complexes and their proposed functions in brain: from zero to (too) many in a decade.Review of pathological hallmarks of schizophrenia: comparison of genetic models with patients and nongenetic models Modeling the positive symptoms of schizophrenia in genetically modified mice: pharmacology and methodology aspects Neurobehavioral abnormalities in the dysbindin-1 mutant, sandy, on a C57BL/6J genetic background.Age-dependent regulation of synaptic connections by dopamine D2 receptors.Dysbindin-1 loss compromises NMDAR-dependent synaptic plasticity and contextual fear conditioning.Genetic modifiers of abnormal organelle biogenesis in a Drosophila model of BLOC-1 deficiency.Components of the basal lamina and dystrophin-dystroglycan complex in the neurointermediate lobe of rat pituitary gland: different localizations of beta-dystroglycan, dystrobrevins, alpha1-syntrophin, and aquaporin-4.Synaptic dysbindin-1 reductions in schizophrenia occur in an isoform-specific manner indicating their subsynaptic location.An inhibitory effect of extracellular Ca2+ on Ca2+-dependent exocytosis.DISC1 in schizophrenia: genetic mouse models and human genomic imaging.Dysbindin-1C is required for the survival of hilar mossy cells and the maturation of adult newborn neurons in dentate gyrus.Reduced dysbindin expression mediates N-methyl-D-aspartate receptor hypofunction and impaired working memory performance.Neuroplasticity signaling pathways linked to the pathophysiology of schizophrenia.Dysbindin-1 genotype effects on emotional working memory MeCP2 regulates the synaptic expression of a Dysbindin-BLOC-1 network component in mouse brain and human induced pluripotent stem cell-derived neurons.Muscarinic inhibition of nicotinic transmission in rat sympathetic neurons and adrenal chromaffin cells.The Schizophrenia-Related Protein Dysbindin-1A Is Degraded and Facilitates NF-Kappa B Activity in the Nucleus Impaired long-term potentiation and long-term memory deficits in xCT-deficient sut mice.Cell biology of the BLOC-1 complex subunit dysbindin, a schizophrenia susceptibility gene.Regulation of synaptic activity by snapin-mediated endolysosomal transport and sorting.Convergent functional genomics of schizophrenia: from comprehensive understanding to genetic risk prediction.

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P2860

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

http://www.wikidata.org/entity/Q28586199

description

2008 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հունիսին հրատարակված գիտական հոդված

@hy

artículu científicu espublizáu en 2008

@ast

im Juni 2008 veröffentlichter wissenschaftlicher Artikel

@de

scientific journal article

@en

vedecký článok (publikovaný 2008/06/02)

@sk

vědecký článek publikovaný v roce 2008

@cs

wetenschappelijk artikel (gepubliceerd op 2008/06/02)

@nl

наукова стаття, опублікована в червні 2008

@uk

مقالة علمية (نشرت في 2-6-2008)

@ar

name

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

@ast

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

@en

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

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type

label

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

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DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

@en

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

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prefLabel

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

@ast

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

@en

DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

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Journal of Cell Biology

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DTNBP1, a schizophrenia susceptibility gene, affects kinetics of transmitter release

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P2093

Chan-Juan Hao

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Claire Xi Zhang

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Hong-Ping Huang

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Hui Zheng

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Ning Guo

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Pan-Li Zuo

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Wei Li

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Wei Xiong

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Xiao-Li Guo

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Xiao-Wei Chen

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P2860

Snapin: a SNARE-associated protein implicated in synaptic transmission

Recent advances in the genetics of schizophrenia

The synaptic vesicle cycle

Dysbindin-1 is a synaptic and microtubular protein that binds brain snapin

Identification of snapin and three novel proteins (BLOS1, BLOS2, and BLOS3/reduced pigmentation) as subunits of biogenesis of lysosome-related organelles complex-1 (BLOC-1)

Identification and characterization of Snapin as a ubiquitously expressed SNARE-binding protein that interacts with SNAP23 in non-neuronal cells

Temporally resolved catecholamine spikes correspond to single vesicle release from individual chromaffin cells

Hermansky-Pudlak syndrome type 7 (HPS-7) results from mutant dysbindin, a member of the biogenesis of lysosome-related organelles complex 1 (BLOC-1)

Dysbindin, a novel coiled-coil-containing protein that interacts with the dystrobrevins in muscle and brain

The TRPM7 ion channel functions in cholinergic synaptic vesicles and affects transmitter release

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791–801

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scholarly article

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1526084

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P356

10.1083/JCB.200711021

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P407

P433

5

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181

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2008-06-02T00:00:00Z

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5346574

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18504299

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Dystrobrevin binding protein 1

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2396815

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