Myocyte apoptosis during acute myocardial infarction in the mouse localizes to hypoxic regions but occurs independently of p53
about
Protective role of deoxyschizandrin and schisantherin A against myocardial ischemia-reperfusion injury in ratsApoptosis in myocardial ischaemia and infarctionBRCA1 is an essential regulator of heart function and survival following myocardial infarctionARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspasesThe apoptosis inhibitor ARC undergoes ubiquitin-proteasomal-mediated degradation in response to death stimuli: identification of a degradation-resistant mutantp53 independent induction of PUMA mediates intestinal apoptosis in response to ischaemia-reperfusionMicromanaging cardiac regeneration: Targeted delivery of microRNAs for cardiac repair and regenerationIntra-myocardial injection of both growth factors and heart derived Sca-1+/CD31- cells attenuates post-MI LV remodeling more than does cell transplantation alone: neither intervention enhances functionally significant cardiomyocyte regenerationHypoxia predisposes neonatal rat ventricular myocytes to apoptosis induced by activation of the Fas (CD95/Apo-1) receptor: Fas activation and apoptosis in hypoxic myocytesp53 initiates apoptosis by transcriptionally targeting the antiapoptotic protein ARCBax translocates to mitochondria of heart cells during simulated ischaemia: involvement of AMP-activated and p38 mitogen-activated protein kinasesp53 and TIGAR regulate cardiac myocyte energy homeostasis under hypoxic stress.Erythropoietin reduces myocardial infarction and left ventricular functional decline after coronary artery ligation in rats.Glucose uptake and glycolysis reduce hypoxia-induced apoptosis in cultured neonatal rat cardiac myocytes.Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart.Modulation of ceramide content and lack of apoptosis in the chronically hypoxic neonatal rat heart.E2/ER β Enhances Calcineurin Protein Degradation and PI3K/Akt/MDM2 Signal Transduction to Inhibit ISO-Induced Myocardial Cell Apoptosis.Sorafenib cardiotoxicity increases mortality after myocardial infarction.Linking endoplasmic reticulum stress to cell death in hepatocytes: roles of C/EBP homologous protein and chemical chaperones in palmitate-mediated cell death.Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53.Cytokines and their receptors in cardiovascular diseases--role of gp130 signalling pathway in cardiac myocyte growth and maintenance.Heart-targeted overexpression of caspase3 in mice increases infarct size and depresses cardiac function.Inhibition of apoptosis by progesterone in cardiomyocytes.Dexamethasone induces transcriptional activation of Bcl-xL gene and inhibits cardiac injury by myocardial ischemia.Beta(2)-adrenoceptor agonist clenbuterol reduces infarct size and myocardial apoptosis after myocardial ischaemia/reperfusion in anaesthetized ratsMicroRNA-494 targeting both proapoptotic and antiapoptotic proteins protects against ischemia/reperfusion-induced cardiac injuryThymosin-β4 prevents cardiac rupture and improves cardiac function in mice with myocardial infarction.Ligation of the left circumflex coronary artery with subsequent MRI and histopathology in rabbits.Critical role for death-receptor mediated apoptotic signaling in viral myocarditis.Targeted deletion of apoptosis signal-regulating kinase 1 attenuates left ventricular remodeling.Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy.A mechanistic role for cardiac myocyte apoptosis in heart failure.Anti-apoptotic effect of benidipine, a long-lasting vasodilating calcium antagonist, in ischaemic/reperfused myocardial cells.Apoptosis in myocardial infarction.Deficiency of ataxia telangiectasia mutated kinase modulates cardiac remodeling following myocardial infarction: involvement in fibrosis and apoptosis.Cardiac remodeling and physical training post myocardial infarctionN-acetylglucosamine conjugated to nanoparticles enhances myocyte uptake and improves delivery of a small molecule p38 inhibitor for post-infarct healing.Cyclo-oxygenase-2 (COX-2) expression at the site of recent myocardial infarction: friend or foe?Right ventricular dilatation after left ventricular acute myocardial infarction is predictive of extremely high peri-infarctual apoptosis at postmortem examination in humansPUMA is critical for neonatal cardiomyocyte apoptosis induced by endoplasmic reticulum stress
P2860
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P2860
Myocyte apoptosis during acute myocardial infarction in the mouse localizes to hypoxic regions but occurs independently of p53
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Myocyte apoptosis during acute ...... ut occurs independently of p53
@en
Myocyte apoptosis during acute ...... t occurs independently of p53.
@nl
type
label
Myocyte apoptosis during acute ...... ut occurs independently of p53
@en
Myocyte apoptosis during acute ...... t occurs independently of p53.
@nl
prefLabel
Myocyte apoptosis during acute ...... ut occurs independently of p53
@en
Myocyte apoptosis during acute ...... t occurs independently of p53.
@nl
P2093
P2860
P356
P1476
Myocyte apoptosis during acute ...... ut occurs independently of p53
@en
P2093
D L Geenen
I E Sasson
J W Horner
R N Kitsis
P2860
P304
P356
10.1172/JCI119656
P407
P577
1997-09-01T00:00:00Z