Epitope map of neurofilament protein domains in cortical and peripheral nervous system Lewy bodies.
about
A new variant of Charcot-Marie-Tooth disease type 2 is probably the result of a mutation in the neurofilament-light gene.alpha-Synuclein in filamentous inclusions of Lewy bodies from Parkinson's disease and dementia with lewy bodiesOverexpression of the human NFM subunit in transgenic mice modifies the level of endogenous NFL and the phosphorylation state of NFH subunitsLoss of non-phosphorylated neurofilament immunoreactivity, with preservation of tyrosine hydroxylase, in surviving substantia nigra neurons in Parkinson's disease.The cytoskeleton in neurodegenerative diseases.Cell type specific sequestration of choline acetyltransferase and tyrosine hydroxylase within Lewy bodies.Bridging molecular genetics and biomarkers in lewy body and related disordersalpha-internexin is present in the pathological inclusions of neuronal intermediate filament inclusion disease.Pathological heterogeneity of frontotemporal lobar degeneration with ubiquitin-positive inclusions delineated by ubiquitin immunohistochemistry and novel monoclonal antibodies.Neurodegeneration with brain iron accumulation, type 1 is characterized by alpha-, beta-, and gamma-synuclein neuropathology.Aggregation of alpha-synuclein in Lewy bodies of sporadic Parkinson's disease and dementia with Lewy bodies.Purification and characterization of Lewy bodies from the brains of patients with diffuse Lewy body disease.Ubiquitination of alpha-synuclein is not required for formation of pathological inclusions in alpha-synucleinopathies.Widespread nitration of pathological inclusions in neurodegenerative synucleinopathiesClinical and neuropathologic variation in neuronal intermediate filament inclusion diseasealpha-Internexin aggregates are abundant in neuronal intermediate filament inclusion disease (NIFID) but rare in other neurodegenerative diseasesCharacterization of antibodies that selectively detect alpha-synuclein in pathological inclusions.Proteasome-dependent degradation of the human estrogen receptor.Lewy body pathology in normal elderly subjects.Biomarkers in biological fluids for dementia with Lewy bodies.Disruption of the NF-H gene increases axonal microtubule content and velocity of neurofilament transport: relief of axonopathy resulting from the toxin beta,beta'-iminodipropionitrile.Altered neurofilament expression does not contribute to Lewy body formation.Mallory body filaments become insoluble after normal assembly into intermediate filaments.Cortical and brainstem-type Lewy bodies are immunoreactive for the cyclin-dependent kinase 5Mitochondrial dysfunction in Parkinson's disease
P2860
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P2860
Epitope map of neurofilament protein domains in cortical and peripheral nervous system Lewy bodies.
description
1991 nî lūn-bûn
@nan
1991 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1991 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
name
Epitope map of neurofilament p ...... al nervous system Lewy bodies.
@ast
Epitope map of neurofilament p ...... al nervous system Lewy bodies.
@en
Epitope map of neurofilament p ...... al nervous system Lewy bodies.
@nl
type
label
Epitope map of neurofilament p ...... al nervous system Lewy bodies.
@ast
Epitope map of neurofilament p ...... al nervous system Lewy bodies.
@en
Epitope map of neurofilament p ...... al nervous system Lewy bodies.
@nl
prefLabel
Epitope map of neurofilament p ...... al nervous system Lewy bodies.
@ast
Epitope map of neurofilament p ...... al nervous system Lewy bodies.
@en
Epitope map of neurofilament p ...... al nervous system Lewy bodies.
@nl
P2093
P2860
P1476
Epitope map of neurofilament p ...... ral nervous system Lewy bodies
@en
P2093
P2860
P407
P577
1991-07-01T00:00:00Z