Epidermal growth factor induces the tyrosine phosphorylation and nuclear translocation of Stat 5 in mouse liver
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Identification of both positive and negative domains within the epidermal growth factor receptor COOH-terminal region for signal transducer and activator of transcription (STAT) activationHuman rhomboid family-1 gene RHBDF1 participates in GPCR-mediated transactivation of EGFR growth signals in head and neck squamous cancer cellsStat1 associates with c-kit and is activated in response to stem cell factorLIF-STAT signaling and trophoblast biologyErbB receptor-induced activation of stat transcription factors is mediated by Src tyrosine kinasesEssential functions of p21-activated kinase 1 in morphogenesis and differentiation of mammary glandsStimulation of interferon regulatory factor-1 by prolactinErbB4 signaling in the mammary gland is required for lobuloalveolar development and Stat5 activation during lactationCharacterization of Stat5a and Stat5b homodimers and heterodimers and their association with the glucocortiocoid receptor in mammary cellsShp-2 tyrosine phosphatase functions as a negative regulator of the interferon-stimulated Jak/STAT pathwaySTAT protein recruitment and activation in c-Kit deletion mutantsPlatelet-derived growth factor induces phosphorylation of multiple JAK family kinases and STAT proteinsTranscriptional activation of the suppressor of cytokine signaling-3 (SOCS-3) gene via STAT3 is increased in F9 REX1 (ZFP-42) knockout teratocarcinoma stem cells relative to wild-type cellsNovel activation of STAT5b in response to epidermal growth factor.STAT signaling in the pathogenesis and treatment of cancerA cytosolic protein-tyrosine phosphatase PTP1B specifically dephosphorylates and deactivates prolactin-activated STAT5a and STAT5b.Putative role of hepatitis B virus X protein in hepatocarcinogenesis: effects on apoptosis, DNA repair, mitogen-activated protein kinase and JAK/STAT pathways.Stat5 is a physiological substrate of the insulin receptor.The immunosuppressive fungal metabolite gliotoxin specifically inhibits transcription factor NF-kappaB.Epidermal growth factor and Ras regulate gene expression in GH4 pituitary cells by separate, antagonistic signal transduction pathways.Erythropoietin induces activation of Stat5 through association with specific tyrosines on the receptor that are not required for a mitogenic response.Deletion of the carboxyl-terminal transactivation domain of MGF-Stat5 results in sustained DNA binding and a dominant negative phenotype.Naturally occurring dominant negative variants of Stat5.Requirement of STAT5b for sexual dimorphism of body growth rates and liver gene expressionEGFR Signaling Regulates Maspin/SerpinB5 Phosphorylation and Nuclear Localization in Mammary Epithelial Cells.Origins of growth factors: NGF and EGF.Activation of Stat 5b in erythroid progenitors correlates with the ability of ErbB to induce sustained cell proliferation.Requirement of Stat3 but not Stat1 activation for epidermal growth factor receptor- mediated cell growth In vitroHBx protein of hepatitis B virus activates Jak1-STAT signaling.Growth hormone activation of Stat 1, Stat 3, and Stat 5 in rat liver. Differential kinetics of hormone desensitization and growth hormone stimulation of both tyrosine phosphorylation and serine/threonine phosphorylation.Integrin-mediated adhesion of endothelial cells induces JAK2 and STAT5A activation: role in the control of c-fos gene expression.Salt, Angiotensin II, Superoxide, and Endothelial Function.Interaction between STAT-3 and HNF-3 leads to the activation of liver-specific hepatitis B virus enhancer 1 function.Cooperative activity between HER oncogenes and the tumor suppressor IRF-1 results in apoptosis.The interleukin-4 receptor activates STAT5 by a mechanism that relies upon common gamma-chain.Transcriptional inhibition by Stat5. Differential activities at growth-related versus differentiation-specific promoters.Formation of STAT5-containing DNA binding complexes in response to colony-stimulating factor-1 and platelet-derived growth factor.STAT activation by epidermal growth factor (EGF) and amphiregulin. Requirement for the EGF receptor kinase but not for tyrosine phosphorylation sites or JAK1.Stem cell factor, the JAK-STAT pathway and signal transduction.Adaptor proteins NUMB and NUMBL promote cell cycle withdrawal by targeting ERBB2 for degradation.
P2860
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P2860
Epidermal growth factor induces the tyrosine phosphorylation and nuclear translocation of Stat 5 in mouse liver
description
1995 nî lūn-bûn
@nan
1995 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Epidermal growth factor induce ...... ation of Stat 5 in mouse liver
@ast
Epidermal growth factor induce ...... ation of Stat 5 in mouse liver
@en
Epidermal growth factor induce ...... ation of Stat 5 in mouse liver
@nl
type
label
Epidermal growth factor induce ...... ation of Stat 5 in mouse liver
@ast
Epidermal growth factor induce ...... ation of Stat 5 in mouse liver
@en
Epidermal growth factor induce ...... ation of Stat 5 in mouse liver
@nl
prefLabel
Epidermal growth factor induce ...... ation of Stat 5 in mouse liver
@ast
Epidermal growth factor induce ...... ation of Stat 5 in mouse liver
@en
Epidermal growth factor induce ...... ation of Stat 5 in mouse liver
@nl
P2093
P2860
P356
P1476
Epidermal growth factor induce ...... ation of Stat 5 in mouse liver
@en
P2093
P2860
P304
P356
10.1073/PNAS.92.10.4215
P407
P577
1995-05-01T00:00:00Z