HTLV-I Tax self-association in optimal trans-activation function.
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The HTLV-1 Tax interactomeHepatitis C virus core protein-induced loss of LZIP function correlates with cellular transformationTORC1 and TORC2 coactivators are required for tax activation of the human T-cell leukemia virus type 1 long terminal repeatsSpecific TATAA and bZIP requirements suggest that HTLV-I Tax has transcriptional activity subsequent to the assembly of an initiation complexRole of Tax protein in human T-cell leukemia virus type-I leukemogenicityThe liver-enriched transcription factor CREB-H is a growth suppressor protein underexpressed in hepatocellular carcinomaThe zinc finger protein A20 interacts with a novel anti-apoptotic protein which is cleaved by specific caspasesActivation of IKKalpha and IKKbeta through their fusion with HTLV-I tax proteinProtein profile of tax-associated complexesCREB--a real culprit in oncogenesisTax-1 and Tax-2 similarities and differences: focus on post-translational modifications and NF-κB activation.CREB-2, a cellular CRE-dependent transcription repressor, functions in association with Tax as an activator of the human T-cell leukemia virus type 1 promoterHuman T-cell leukemia virus type 1 Tax requires direct access to DNA for recruitment of CREB binding protein to the viral promoterMolecular interactions involved in the transactivation of the human T-cell leukemia virus type 1 promoter mediated by Tax and CREB-2 (ATF-4).Human T-lymphotropic virus type 1-infected cells secrete exosomes that contain Tax protein.Stimulation of IKK-gamma oligomerization by the human T-cell leukemia virus oncoprotein Tax.The central region of human T-cell leukemia virus type 1 Tax protein contains distinct domains involved in subunit dimerizationThe complementary strand of the human T-cell leukemia virus type 1 RNA genome encodes a bZIP transcription factor that down-regulates viral transcription.Human T-cell lymphotropic virus: a model of NF-κB-associated tumorigenesisHuman T Lymphotropic Virus Type 1 (HTLV-1): Molecular Biology and Oncogenesis.Move or die: the fate of the Tax oncoprotein of HTLV-1.Molecular characterization of HTLV-1 Tax interaction with the KIX domain of CBP/p300.Cotranscriptional Chromatin Remodeling by Small RNA Species: An HTLV-1 PerspectiveHTLV tax: a fascinating multifunctional co-regulator of viral and cellular pathwaysLKB1 tumor suppressor and salt-inducible kinases negatively regulate human T-cell leukemia virus type 1 transcription.Dimerization and a novel Tax speckled structure localization signal are required for Tax nuclear localization.Intracellular localization and cellular factors interaction of HTLV-1 and HTLV-2 Tax proteins: similarities and functional differences.The MHC-II transactivator CIITA, a restriction factor against oncogenic HTLV-1 and HTLV-2 retroviruses: similarities and differences in the inhibition of Tax-1 and Tax-2 viral transactivators.Specific regions of contact between human T-cell leukemia virus type I Tax protein and DNA identified by photocross-linking.Comparison of the Genetic Organization, Expression Strategies and Oncogenic Potential of HTLV-1 and HTLV-2.Human T-cell leukemia virus type 1 Tax oncoprotein prevents DNA damage-induced chromatin egress of hyperphosphorylated Chk2.A 10-amino acid domain within human T-cell leukemia virus type 1 and type 2 tax protein sequences is responsible for their divergent subcellular distribution.Oligomerization of Ebola virus VP30 is essential for viral transcription and can be inhibited by a synthetic peptide.Cooperation of yeast peroxiredoxins Tsa1p and Tsa2p in the cellular defense against oxidative and nitrosative stress.Inhibition of LZIP-mediated transcription through direct interaction with a novel host cell factor-like protein.CpR18, a novel SAP-domain plant transcription factor, binds to a promoter region necessary for ABA mediated expression of the CDeT27-45 gene from the resurrection plant Craterostigma plantagineum Hochst.Site-specific phosphorylation differentiates active from inactive forms of the human T-cell leukemia virus type 1 Tax oncoprotein.Mammalian peroxiredoxin isoforms can reduce hydrogen peroxide generated in response to growth factors and tumor necrosis factor-alpha.Role of Adapter Function in Oncoprotein-mediated Activation of NF-κB
P2860
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P2860
HTLV-I Tax self-association in optimal trans-activation function.
description
1997 nî lūn-bûn
@nan
1997 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
name
HTLV-I Tax self-association in optimal trans-activation function.
@ast
HTLV-I Tax self-association in optimal trans-activation function.
@en
HTLV-I Tax self-association in optimal trans-activation function.
@nl
type
label
HTLV-I Tax self-association in optimal trans-activation function.
@ast
HTLV-I Tax self-association in optimal trans-activation function.
@en
HTLV-I Tax self-association in optimal trans-activation function.
@nl
prefLabel
HTLV-I Tax self-association in optimal trans-activation function.
@ast
HTLV-I Tax self-association in optimal trans-activation function.
@en
HTLV-I Tax self-association in optimal trans-activation function.
@nl
P2860
P356
P1476
HTLV-I Tax self-association in optimal trans-activation function.
@en
P2860
P304
P356
10.1093/NAR/25.2.379
P407
P577
1997-01-01T00:00:00Z