Differential contribution of IL-4 and STAT6 vs STAT4 to the development of lupus nephritis. - Wikidata - awgldk - TriplyDB

Differential contribution of IL-4 and STAT6 vs STAT4 to the development of lupus nephritis.

Differential contribution of IL-4 and STAT6 vs STAT4 to the development of lupus nephritis.

http://www.wikidata.org/entity/Q36537018

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Lupus nephritis: current update Mechanisms of tissue injury in lupus nephritis Breaking tolerance to double stranded DNA, nucleosome, and other nuclear antigens is not required for the pathogenesis of lupus glomerulonephritis.Lupus glomerulonephritis revisited 2004: autoimmunity and end-organ damage.Autoantibody-dependent and autoantibody-independent roles for B cells in systemic lupus erythematosus: past, present, and future IL-4-STAT6 signal transduction-dependent induction of the clinical phase of Sjögren's syndrome-like disease of the nonobese diabetic mouse.B cells require "nurturing" by CD4 T cells during development in order to respond in chronic graft-versus-host model of systemic lupus erythematosus.Therapeutic strategies for SLE involving cytokines: mechanism-oriented therapies especially IFN-gamma targeting gene therapy.STAT4: genetics, mechanisms, and implications for autoimmunity.γδ T cells affect IL-4 production and B-cell tolerance Comparative transcriptional profiling of 3 murine models of SLE nephritis reveals both unique and shared regulatory networks.The histone peptide H4 71-94 alone is more effective than a cocktail of peptide epitopes in controlling lupus: immunoregulatory mechanisms.Suppression of systemic autoimmunity by the innate immune adaptor STING.The role of cytokines in the pathogenesis and treatment of systemic lupus erythematosus Targeting transcription factor Stat4 uncovers a role for interleukin-18 in the pathogenesis of severe lupus nephritis in mice.Lupus nephritis: an overview of recent findings.Signaling by IL-12 and IL-23 and the immunoregulatory roles of STAT4.Cross-species transcriptional network analysis defines shared inflammatory responses in murine and human lupus nephritis SLE: translating lessons from model systems to human disease Glomerulonephritis, Th1 and Th2: what's new?An ACE inhibitor reduces Th2 cytokines and TGF-beta1 and TGF-beta2 isoforms in murine lupus nephritis.The natural killer T cell ligand alpha-galactosylceramide prevents or promotes pristane-induced lupus in mice CD1d deficiency exacerbates inflammatory dermatitis in MRL-lpr/lpr mice.Examining the role of CD1d and natural killer T cells in the development of nephritis in a genetically susceptible lupus model A female preponderance for chemically induced lupus in SJL/J mice.A role for sex chromosome complement in the female bias in autoimmune disease.Cytokines in systemic lupus erythematosus From the Large Scale Expression Analysis of Lupus Nephritis to Targeted Molecular Medicine.Role of T cells and dendritic cells in glomerular immunopathology.Targeting of the immune system in systemic lupus erythematosus.Apigenin, a non-mutagenic dietary flavonoid, suppresses lupus by inhibiting autoantigen presentation for expansion of autoreactive Th1 and Th17 cells Interleukin-4 induces senescence in human renal carcinoma cell lines through STAT6 and p38 MAPK.Why can't we find a new treatment for SLE?High-density genotyping of STAT4 reveals multiple haplotypic associations with systemic lupus erythematosus in different racial groups.STAT4 associates with systemic lupus erythematosus through two independent effects that correlate with gene expression and act additively with IRF5 to increase risk BAFF: a local and systemic target in autoimmune diseases.The multi-faceted influences of estrogen on lymphocytes: toward novel immuno-interventions strategies for autoimmunity management.Insight into gene polymorphisms involved in toll-like receptor/interferon signalling pathways for systemic lupus erythematosus in South East Asia.Basophils, IgE, and autoantibody-mediated kidney disease Molecular studies of lupus nephritis kidneys.

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Differential contribution of IL-4 and STAT6 vs STAT4 to the development of lupus nephritis.

http://www.wikidata.org/entity/Q36537018

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Journal of Immunology

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Chaim O Jacob

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David P Witte

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Fred D Finkelman

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Lily Li

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Ram Raj Singh

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Song Zang

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Vijay Saxena

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P2860

Essential role of Stat6 in IL-4 signalling

Lack of IL-4-induced Th2 response and IgE class switching in mice with disrupted Stat6 gene

Lack of skin fibrosis in tight skin (TSK) mice with targeted mutation in the interleukin-4R alpha and transforming growth factor-beta genes.

Tumour necrosis factor and other cytokines in murine lupus.

The inhibitory effects of transforming growth factor-beta-1 (TGF-beta1) in autoimmune diseases.

Fibrosis of the lung and other tissues: new concepts in pathogenesis and treatment.

Autoimmune diseases.

Delineating the genetic basis of systemic lupus erythematosus.

Hepatic expression of mature transforming growth factor beta 1 in transgenic mice results in multiple tissue lesions.

Differential regulation of IgG1 and IgE synthesis by interleukin 4.

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